HSV-1-encoded microRNA miR-H1 targets Ubr1 to promote accumulation of neurodegeneration-associated protein
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Yifei Wang | K. Kitazato | Depo Yang | Qiu-ying Liu | K. Zheng | Zhe Ren | Shaoxiang Wang
[1] A. Varshavsky,et al. The N-Terminal Methionine of Cellular Proteins as a Degradation Signal , 2014, Cell.
[2] C. S. Brower,et al. Neurodegeneration-associated protein fragments as short-lived substrates of the N-end rule pathway. , 2013, Molecular cell.
[3] M. Ball,et al. Intracerebral propagation of Alzheimer’s disease: Strengthening evidence of a herpes simplex virus etiology , 2013, Alzheimer's & Dementia.
[4] Rodney P Kincaid,et al. Virus-Encoded microRNAs: An Overview and a Look to the Future , 2012, PLoS pathogens.
[5] D. Coen,et al. Herpes Simplex Virus Is Equipped with RNA- and Protein-Based Mechanisms To Repress Expression of ATRX, an Effector of Intrinsic Immunity , 2012, Journal of Virology.
[6] A. Burch,et al. A novel miRNA produced during lytic HSV-1 infection is important for efficient replication in tissue culture , 2012, Archives of Virology.
[7] M. Bullido,et al. Herpes simplex virus type I induces the accumulation of intracellular β-amyloid in autophagic compartments and the inhibition of the non-amyloidogenic pathway in human neuroblastoma cells , 2012, Neurobiology of Aging.
[8] A. Palamara,et al. HSV-1 promotes Ca2+-mediated APP phosphorylation and Aβ accumulation in rat cortical neurons , 2011, Neurobiology of Aging.
[9] C. M. Preston,et al. Antivirals Reduce the Formation of Key Alzheimer's Disease Molecules in Cell Cultures Acutely Infected with Herpes Simplex Virus Type 1 , 2011, PloS one.
[10] B. Cullen,et al. Viruses and microRNAs: RISCy interactions with serious consequences. , 2011, Genes & development.
[11] D. Coen,et al. Herpes simplex virus 1 microRNAs expressed abundantly during latent infection are not essential for latency in mouse trigeminal ganglia. , 2011, Virology.
[12] A. Varshavsky. The N‐end rule pathway and regulation by proteolysis , 2011, Protein science : a publication of the Protein Society.
[13] M. Concha,et al. [Herpes simplex virus type 1 as risk factor associated to Alzheimer disease]. , 2011, Revista medica de Chile.
[14] A. Palamara,et al. APP Processing Induced by Herpes Simplex Virus Type 1 (HSV-1) Yields Several APP Fragments in Human and Rat Neuronal Cells , 2010, PloS one.
[15] B. Cullen,et al. Viruses, microRNAs, and host interactions. , 2010, Annual review of microbiology.
[16] O. Mandelboim,et al. The human cytomegalovirus microRNA miR-UL112 acts synergistically with a cellular microRNA to escape immune elimination , 2010, Nature Immunology.
[17] J. Mellor,et al. Numerous Conserved and Divergent MicroRNAs Expressed by Herpes Simplex Viruses 1 and 2 , 2010, Journal of Virology.
[18] B. Cullen,et al. Analysis of Human Alphaherpesvirus MicroRNA Expression in Latently Infected Human Trigeminal Ganglia , 2009, Journal of Virology.
[19] W. Fu,et al. Autophagy protects neuron from Aβ-induced cytotoxicity , 2009, Autophagy.
[20] Noam Stern-Ginossar,et al. Diverse herpesvirus microRNAs target the stress-induced immune ligand MICB to escape recognition by natural killer cells. , 2009, Cell host & microbe.
[21] V. Kim,et al. Biogenesis of small RNAs in animals , 2009, Nature Reviews Molecular Cell Biology.
[22] D. Bartel. MicroRNAs: Target Recognition and Regulatory Functions , 2009, Cell.
[23] P. Krause,et al. Novel Less-Abundant Viral MicroRNAs Encoded by Herpes Simplex Virus 2 Latency-Associated Transcript and Their Roles in Regulating ICP34.5 and ICP0 mRNAs , 2008, Journal of Virology.
[24] J. Cohen,et al. An acutely and latently expressed herpes simplex virus 2 viral microRNA inhibits expression of ICP34.5, a viral neurovirulence factor , 2008, Proceedings of the National Academy of Sciences.
[25] B. Cullen,et al. MicroRNAs expressed by herpes simplex virus 1 during latent infection regulate viral mRNAs , 2008, Nature.
[26] Wenbin Ye,et al. The Effect of Central Loops in miRNA:MRE Duplexes on the Efficiency of miRNA-Mediated Gene Regulation , 2008, PloS one.
[27] M. Wozniak,et al. Herpes simplex virus infection causes cellular β-amyloid accumulation and secretase upregulation , 2007, Neuroscience Letters.
[28] Hanah Margalit,et al. Host Immune System Gene Targeting by a Viral miRNA , 2007, Science.
[29] A. Hatzigeorgiou,et al. A guide through present computational approaches for the identification of mammalian microRNA targets , 2006, Nature Methods.
[30] D. Gerlier,et al. Viral hijacking of cellular ubiquitination pathways as an anti-innate immunity strategy. , 2006, Viral immunology.
[31] A. Hatzigeorgiou,et al. Anti-apoptotic function of a microRNA encoded by the HSV-1 latency-associated transcript , 2006, Nature.
[32] Guanglin Li,et al. Prediction and Identification of Herpes Simplex Virus 1-Encoded MicroRNAs , 2006, Journal of Virology.
[33] N. Rajewsky,et al. Silencing of microRNAs in vivo with ‘antagomirs’ , 2005, Nature.
[34] K. Livak,et al. Real-time quantification of microRNAs by stem–loop RT–PCR , 2005, Nucleic acids research.
[35] Min Jae Lee,et al. A Family of Mammalian E3 Ubiquitin Ligases That Contain the UBR Box Motif and Recognize N-Degrons , 2005, Molecular and Cellular Biology.
[36] R. Russell,et al. Principles of MicroRNA–Target Recognition , 2005, PLoS biology.
[37] R. Giegerich,et al. Fast and effective prediction of microRNA/target duplexes. , 2004, RNA.
[38] G. Higson. Overview and look to the future , 2001 .
[39] R. L. Thompson,et al. The herpes simplex virus type 1 latency-associated transcript gene regulates the establishment of latency , 1997, Journal of virology.
[40] G. Lyon,et al. The N-Terminal Methionine of Cellular Proteins as a Degradation Signal , 2014 .
[41] O. Bagasra,et al. A herpes simplex virus type 1 mutant with a deletion immediately upstream of the LAT locus establishes latency and reactivates from latently infected mice with normal kinetics. , 1996, Journal of neurovirology.