Response to Chemotherapy and Prognosis in Metastatic Colorectal Cancer With DNA Deficient Mismatch Repair
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R. Riechelmann | P. Hoff | S. Siqueira | R. Coudry | Juliana Santos | A. Alex | M. Alves
[1] J. Meyerhardt,et al. The relationship between primary tumor sidedness and prognosis in colorectal cancer. , 2016 .
[2] Preet Paul Singh,et al. Immune checkpoints and immunotherapy for colorectal cancer , 2015, Gastroenterology report.
[3] Jeffrey S. Morris,et al. The Consensus Molecular Subtypes of Colorectal Cancer , 2015, Nature Medicine.
[4] E. Lau. Mismatch repair deficiency predicts benefit of anti-PD-1 therapy. , 2015, The Lancet. Oncology.
[5] T. Aparicio,et al. PD-1 blockade in tumors with mismatch-repair deficiency , 2015 .
[6] D. Chang,et al. Lynch‐like syndrome: Characterization and comparison with EPCAM deletion carriers , 2015, International journal of cancer.
[7] R. Riechelmann,et al. The effects of palliative chemotherapy in metastatic colorectal cancer patients with an ECOG performance status of 3 and 4. , 2015, Clinical colorectal cancer.
[8] H. Lynch,et al. 570PTHYMIDYLATE SYNTHASE OVER-EXPRESSION UNDERLIES THE OBSERVED LACK OF 5-FU THERAPY BENEFIT FOR MSI-H COLORECTAL CANCERS. , 2014, Annals of oncology : official journal of the European Society for Medical Oncology.
[9] E. Mello,et al. Prognostic implication of mucinous histology in resected colorectal cancer liver metastases. , 2014, Surgery.
[10] S. Friend,et al. Colorectal Cancer Subtyping Consortium (CRCSC) identification of a consensus of molecular subtypes. , 2014 .
[11] P. Gibbs,et al. Multicenter retrospective analysis of metastatic colorectal cancer (CRC) with high-level microsatellite instability (MSI-H). , 2014, Annals of oncology : official journal of the European Society for Medical Oncology.
[12] Hyuk Hur,et al. Prognostic Value of Mucinous Histology Depends on Microsatellite Instability Status in Patients with Stage III Colon Cancer Treated with Adjuvant FOLFOX Chemotherapy: A Retrospective Cohort Study , 2013, Annals of Surgical Oncology.
[13] Á. Carracedo,et al. Risk of cancer in cases of suspected lynch syndrome without germline mutation. , 2013, Gastroenterology.
[14] P. Demetter,et al. Mucinous subtype as prognostic factor in colorectal cancer: a systematic review and meta-analysis , 2012, Journal of Clinical Pathology.
[15] P. Gibbs,et al. Impact of BRAF mutation and microsatellite instability on the pattern of metastatic spread and prognosis in metastatic colorectal cancer , 2011, Cancer.
[16] Yoon-Koo Kang,et al. Association between deficient mismatch repair system and efficacy to irinotecan‐containing chemotherapy in metastatic colon cancer , 2011, Cancer science.
[17] N. Lindor,et al. Lynch syndrome and MYH-associated polyposis: review and testing strategy. , 2011, Journal of clinical gastroenterology.
[18] Joon-Oh Park,et al. The effect of DNA mismatch repair (MMR) status on oxaliplatin-based first-line chemotherapy as in recurrent or metastatic colon cancer , 2010, Medical oncology.
[19] K. Öhrling,et al. Mismatch repair protein expression is an independent prognostic factor in sporadic colorectal cancer , 2010, Acta oncologica.
[20] R. Labianca,et al. Defective mismatch repair as a predictive marker for lack of efficacy of fluorouracil-based adjuvant therapy in colon cancer. , 2010, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[21] D. Sargent,et al. Clinical implications of microsatellite instability in sporadic colon cancers , 2009, Current opinion in oncology.
[22] B. Uzzan,et al. Microsatellite instability does not predict the efficacy of chemotherapy in metastatic colorectal cancer. A systematic review and meta-analysis. , 2009, Anticancer research.
[23] C. Compton,et al. Microsatellite instability predicts improved response to adjuvant therapy with irinotecan, fluorouracil, and leucovorin in stage III colon cancer: Cancer and Leukemia Group B Protocol 89803. , 2009, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[24] M. Ligtenberg,et al. Deficient mismatch repair system in patients with sporadic advanced colorectal cancer , 2009, British Journal of Cancer.
[25] Peter W. Laird,et al. Molecular Characterization of MSI-H Colorectal Cancer by MLHI Promoter Methylation, Immunohistochemistry, and Mismatch Repair Germline Mutation Screening , 2008, Cancer Epidemiology Biomarkers & Prevention.
[26] K. Schulmann,et al. Predictive and prognostic value of microsatellite instability in patients with advanced colorectal cancer treated with a fluoropyrimidine and oxaliplatin containing first-line chemotherapy. A report of the AIO Colorectal Study Group , 2008, International Journal of Colorectal Disease.
[27] W. Scheithauer,et al. Bevacizumab in Combination With Oxaliplatin-Based Chemotherapy As First-Line Therapy in Metastatic Colorectal Cancer: A Randomized Phase III Study , 2023, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[28] J. Cucherousset,et al. Microsatellite instability and sensitivitiy to FOLFOX treatment in metastatic colorectal cancer. , 2007, Anticancer research.
[29] J. Warusavitarne,et al. The role of chemotherapy in microsatellite unstable (MSI-H) colorectal cancer , 2007, International Journal of Colorectal Disease.
[30] J. Jass. Classification of colorectal cancer based on correlation of clinical, morphological and molecular features , 2007, Histopathology.
[31] M. Ducreux,et al. Microsatellite instability is a predictive factor of the tumor response to irinotecan in patients with advanced colorectal cancer. , 2003, Cancer research.
[32] Daniel J Sargent,et al. Tumor microsatellite-instability status as a predictor of benefit from fluorouracil-based adjuvant chemotherapy for colon cancer. , 2003, The New England journal of medicine.
[33] King-Jen Chang,et al. High‐frequency microsatellite instability predicts better chemosensitivity to high‐dose 5‐fluorouracil plus leucovorin chemotherapy for stage IV sporadic colorectal cancer after palliative bowel resection , 2002, International journal of cancer.
[34] Robert Brown,et al. Primary ovarian carcinomas display multiple methylator phenotypes involving known tumor suppressor genes. , 2001, The American journal of pathology.
[35] G. Strathdee,et al. A role for methylation of the hMLH1 promoter in loss of hMLH1 expression and drug resistance in ovarian cancer , 1999, Oncogene.
[36] T. Kunkel,et al. The role of hMLH1, hMSH3, and hMSH6 defects in cisplatin and oxaliplatin resistance: correlation with replicative bypass of platinum-DNA adducts. , 1998, Cancer research.
[37] A. V. D. Van Der Zee,et al. hMLH1 expression and cellular responses of ovarian tumour cells to treatment with cytotoxic anticancer agents , 1997, Oncogene.
[38] C. Macleod,et al. In vitro and in vivo resistance to cisplatin in cells that have lost DNA mismatch repair. , 1997, Cancer research.
[39] S. Aebi,et al. The role of DNA mismatch repair in platinum drug resistance. , 1996, Cancer research.
[40] P. Hoff,et al. Modified FLOX as first-line chemotherapy for metastatic colorectal cancer patients in the public health system in Brazil: Effectiveness and cost-utility analysis. , 2013, Molecular and clinical oncology.
[41] E. Chiorean. DNA Mismatch Repair Status and Colon Cancer Recurrence and Survival in Clinical Trials of 5-Fluorouracil-Based Adjuvant Therapy , 2011 .
[42] P. Loehrer. High Thymidylate Synthase Expression in Colorectal Cancer With Microsatellite Instability: Implications for Chemotherapeutic Strategies , 2006 .