Distribution and function of human ventricular beta adrenergic receptors in congestive heart failure.
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P. Allen | W. Colucci | A. Denniss | J. Marsh
[1] W. Baumgartner,et al. Increase of the 40,000-mol wt pertussis toxin substrate (G protein) in the failing human heart. , 1988, The Journal of clinical investigation.
[2] S. Vatner,et al. Decreased stimulatory guanosine triphosphate binding protein in dogs with pressure-overload left ventricular failure. , 1988, The Journal of clinical investigation.
[3] F. Schoen,et al. Deficient production of cyclic AMP: pharmacologic evidence of an important cause of contractile dysfunction in patients with end-stage heart failure. , 1987, Circulation.
[4] M. Bristow,et al. Assessment of the beta-adrenergic receptor pathway in the intact failing human heart: progressive receptor down-regulation and subsensitivity to agonist response. , 1986, Circulation.
[5] S. Jamieson,et al. Beta 1- and beta 2-adrenergic-receptor subpopulations in nonfailing and failing human ventricular myocardium: coupling of both receptor subtypes to muscle contraction and selective beta 1-receptor down-regulation in heart failure. , 1986, Circulation research.
[6] P. Simpson,et al. Mechanisms and time course of beta 1 adrenoceptor desensitisation in mammalian cardiac myocytes. , 1985, Cardiovascular research.
[7] S. Vatner,et al. Loss of high affinity cardiac beta adrenergic receptors in dogs with heart failure. , 1985, The Journal of clinical investigation.
[8] H. Motulsky,et al. Externalization of beta-adrenergic receptors promoted by myocardial ischemia. , 1985, Science.
[9] D. Kim,et al. Mechanisms of beta-adrenergic receptor regulation in cultured chick heart cells. Role of cytoskeleton function and protein synthesis. , 1985, Circulation research.
[10] T. Smith,et al. Receptors for beta-adrenergic agonists in cultured chick ventricular cells. Relationship between agonist binding and physiologic effect. , 1985, Molecular pharmacology.
[11] T. K. Harden,et al. Relationship between an altered membrane form and a low affinity form of the beta-adrenergic receptor occurring during catecholamine-induced desensitization. Evidence for receptor internalization. , 1984, The Journal of biological chemistry.
[12] G. Stiles,et al. Translocation and uncoupling of the beta-adrenergic receptor in rat lung after catecholamine promoted desensitization in vivo. , 1984, Endocrinology.
[13] D C Harrison,et al. Decreased catecholamine sensitivity and beta-adrenergic-receptor density in failing human hearts. , 1982, The New England journal of medicine.
[14] E. Braunwald,et al. Decreased lymphocyte beta-adrenergic-receptor density in patients with heart failure and tolerance to the beta-adrenergic agonist pirbuterol. , 1981, The New England journal of medicine.
[15] D Rodbard,et al. Ligand: a versatile computerized approach for characterization of ligand-binding systems. , 1980, Analytical biochemistry.
[16] K. Paigen,et al. A simple, rapid, and sensitive DNA assay procedure. , 1980, Analytical biochemistry.
[17] R. Lefkowitz,et al. A quantitative analysis of beta-adrenergic receptor interactions: resolution of high and low affinity states of the receptor by computer modeling of ligand binding data. , 1980, Molecular pharmacology.
[18] E. Neer. The vasopressin-sensitive adenylate cyclase of the rat renal medulla. , 1973, Journal of Biological Chemistry.
[19] B B Brodie,et al. A simple, sensitive method for the assay of adenyl cyclase. , 1968, The Journal of pharmacology and experimental therapeutics.
[20] N. M. Hawkins,et al. Studies on sodium-potassium-activated adenosinetriphosphatase. IV. Correlation with cation transport sensitive to cardiac glycosides. , 1962, Archives of biochemistry and biophysics.