SMPDL3b modulates insulin receptor signaling in diabetic kidney disease
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R. Inagi | A. Mendez | T. Yoo | A. Fornoni | I. Leibiger | I. D. Zelnik | A. Futerman | E. Rosenfeld-Gur | G. Burke | C. Faul | L. Barisoni | S. Mallela | Y. Zeidan | S. Merscher | Y. Ishimoto | J. Molina | A. Mitrofanova | G. Ducasa | M. Ge | A. Sloan | C. Pedigo | J. Varona Santos | J. J. Kim | J. Bryn | I. Volosenco | C. García Hernandez
[1] B. Bergman,et al. Intracellular localization of diacylglycerols and sphingolipids influences insulin sensitivity and mitochondrial function in human skeletal muscle. , 2018, JCI insight.
[2] B. Ogretmen,et al. Sphingolipid metabolism in cancer signalling and therapy , 2017, Nature Reviews Cancer.
[3] N. Hait,et al. The Role of Sphingosine-1-Phosphate and Ceramide-1-Phosphate in Inflammation and Cancer , 2017, Mediators of inflammation.
[4] Hui-Young Lee,et al. Adipocyte-Specific Deficiency of De Novo Sphingolipid Biosynthesis Leads to Lipodystrophy and Insulin Resistance , 2017, Diabetes.
[5] Liping Huang,et al. Sphingosine Kinase 2 Deficiency Attenuates Kidney Fibrosis via IFN-γ. , 2017, Journal of the American Society of Nephrology : JASN.
[6] Y. Frishberg,et al. Deficiency of the sphingosine‐1‐phosphate lyase SGPL1 is associated with congenital nephrotic syndrome and congenital adrenal calcifications , 2017, Human mutation.
[7] J. Krepinsky,et al. Caveolin-1 in the Pathogenesis of Diabetic Nephropathy: Potential Therapeutic Target? , 2017, Current Diabetes Reports.
[8] K. Schwarz,et al. Mutations in sphingosine-1-phosphate lyase cause nephrosis with ichthyosis and adrenal insufficiency , 2017, The Journal of clinical investigation.
[9] L. Metherell,et al. Sphingosine-1-phosphate lyase mutations cause primary adrenal insufficiency and steroid-resistant nephrotic syndrome , 2017, The Journal of clinical investigation.
[10] B. Marples,et al. Sphingomyelinase‐like phosphodiesterase 3b mediates radiation‐induced damage of renal podocytes , 2017, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[11] P. Meikle,et al. Sphingolipids and phospholipids in insulin resistance and related metabolic disorders , 2017, Nature Reviews Endocrinology.
[12] P. Meikle,et al. Adipocyte Ceramides Regulate Subcutaneous Adipose Browning, Inflammation, and Metabolism. , 2016, Cell metabolism.
[13] G. Superti-Furga,et al. Crystal Structure of the Acid Sphingomyelinase-like Phosphodiesterase SMPDL3B Provides Insights into Determinants of Substrate Specificity* , 2016, The Journal of Biological Chemistry.
[14] D. Maiguel,et al. Nephrin Contributes to Insulin Secretion and Affects Mammalian Target of Rapamycin Signaling Independently of Insulin Receptor. , 2016, Journal of the American Society of Nephrology : JASN.
[15] M. Idzko,et al. C1P Attenuates Lipopolysaccharide-Induced Acute Lung Injury by Preventing NF-κB Activation in Neutrophils , 2016, The Journal of Immunology.
[16] A. Gomez-Muñoz,et al. Ceramide 1-phosphate regulates cell migration and invasion of human pancreatic cancer cells. , 2016, Biochemical pharmacology.
[17] T. Kinoshita,et al. Biosynthesis of GPI-anchored proteins: special emphasis on GPI lipid remodeling , 2016, Journal of Lipid Research.
[18] W. Prinz,et al. A conserved family of proteins facilitates nascent lipid droplet budding from the ER , 2022 .
[19] Hong-Hee Kim,et al. Caveolin-1 regulates osteoclast differentiation by suppressing cFms degradation , 2015, Experimental & Molecular Medicine.
[20] G. Superti-Furga,et al. The Lipid-Modifying Enzyme SMPDL3B Negatively Regulates Innate Immunity , 2015, Cell reports.
[21] S. Dheen,et al. Sphingosine kinase 2 and sphingosine-1-phosphate promotes mitochondrial function in dopaminergic neurons of mouse model of Parkinson’s disease and in MPP+-treated MN9D cells in vitro , 2015, Neuroscience.
[22] J. Lee,et al. Methyl-β-cyclodextrin up-regulates collagen I expression in chronologically-aged skin via its anti-caveolin-1 activity , 2014, Oncotarget.
[23] C. Forsblom,et al. Sphingomyelinase-like phosphodiesterase 3b expression levels determine podocyte injury phenotypes in glomerular disease. , 2015, Journal of the American Society of Nephrology : JASN.
[24] E. Parati,et al. Autocrine/paracrine sphingosine‐1‐phosphate fuels proliferative and stemness qualities of glioblastoma stem cells , 2014, Glia.
[25] A. Castro,et al. Partial inhibition of Cdk1 in G2 phase overrides the SAC and decouples mitotic events , 2014, Cell cycle.
[26] D. Maiguel,et al. Podocyte-Specific GLUT4-Deficient Mice Have Fewer and Larger Podocytes and Are Protected From Diabetic Nephropathy , 2014, Diabetes.
[27] Anup M Oommen,et al. Sphingolipid Metabolic Pathway: An Overview of Major Roles Played in Human Diseases , 2013, Journal of lipids.
[28] R. Weichselbaum,et al. Sphingosine-1-phosphate, FTY720, and sphingosine-1-phosphate receptors in the pathobiology of acute lung injury. , 2013, American journal of respiratory cell and molecular biology.
[29] Thomas R. Gingeras,et al. STAR: ultrafast universal RNA-seq aligner , 2013, Bioinform..
[30] M. Ratajczak,et al. CONDITIONING FOR HEMATOPOIETIC TRANSPLANTATION ACTIVATES THE COMPLEMENT CASCADE AND INDUCES A PROTEOLYTIC ENVIRONMENT IN BONE MARROW – A NOVEL ROLE FOR BIOACTIVE LIPIDS AND SOLUBLE C5b-C9 AS HOMING FACTORS , 2011, Leukemia.
[31] I. Novak,et al. Cell Volume Regulation and Signaling in 3T3-L1 Pre-adipocytes and Adipocytes: On the Possible Roles of Caveolae, Insulin Receptors, FAK and ERK1/2 , 2011, Cellular Physiology and Biochemistry.
[32] E. Salido,et al. Circulating urokinase receptor as a cause of focal segmental glomerulosclerosis , 2011, Nature Medicine.
[33] M. Rastaldi,et al. Rituximab Targets Podocytes in Recurrent Focal Segmental Glomerulosclerosis , 2011, Science Translational Medicine.
[34] G. King,et al. Glomerular-specific protein kinase C-β-induced insulin receptor substrate-1 dysfunction and insulin resistance in rat models of diabetes and obesity. , 2011, Kidney international.
[35] S. Summers,et al. Sphingolipids and insulin resistance: the five Ws , 2010, Current opinion in lipidology.
[36] C. Gong,et al. Deregulation of sphingolipid metabolism in Alzheimer's disease , 2010, Neurobiology of Aging.
[37] Reiko Kurotani,et al. Caveolin gene transfer improves glucose metabolism in diabetic mice. , 2010, American journal of physiology. Cell physiology.
[38] Mark D. Robinson,et al. edgeR: a Bioconductor package for differential expression analysis of digital gene expression data , 2009, Bioinform..
[39] A. Gomez-Muñoz,et al. Ceramide-1-phosphate in cell survival and inflammatory signaling. , 2010, Advances in experimental medicine and biology.
[40] D. Pisetsky,et al. Glomerular type 1 angiotensin receptors augment kidney injury and inflammation in murine autoimmune nephritis. , 2009, The Journal of clinical investigation.
[41] A. Gomez-Muñoz,et al. Ceramide 1-phosphate (C1P) promotes cell migration Involvement of a specific C1P receptor. , 2009, Cellular signalling.
[42] A. Fornoni,et al. Failure to phosphorylate AKT in podocytes from mice with early diabetic nephropathy promotes cell death. , 2008, Kidney international.
[43] A. Gomez-Muñoz,et al. Ceramide 1-phosphate stimulates macrophage proliferation through activation of the PI3-kinase/PKB, JNK and ERK1/2 pathways. , 2008, Cellular signalling.
[44] P. V. Van Veldhoven,et al. Neutropenia with Impaired Immune Response to Streptococcus pneumoniae in Ceramide Kinase-Deficient Mice , 2008, The Journal of Immunology.
[45] Yusuf A. Hannun,et al. Principles of bioactive lipid signalling: lessons from sphingolipids , 2008, Nature Reviews Molecular Cell Biology.
[46] Y. Igarashi,et al. Dissociation of the insulin receptor and caveolin-1 complex by ganglioside GM3 in the state of insulin resistance , 2007, Proceedings of the National Academy of Sciences.
[47] M. Birnbaum,et al. Inhibition of ceramide synthesis ameliorates glucocorticoid-, saturated-fat-, and obesity-induced insulin resistance. , 2007, Cell metabolism.
[48] G. Boysen,et al. Tandem mass spectrometry measurements of creatinine in mouse plasma and urine for determining glomerular filtration rate. , 2007, Kidney international.
[49] A. Gomez-Muñoz. Ceramide 1-phosphate/ceramide, a switch between life and death. , 2006, Biochimica et biophysica acta.
[50] B. Ogretmen. Sphingolipids in cancer: Regulation of pathogenesis and therapy , 2006, FEBS letters.
[51] A. Bielawska,et al. Simultaneous quantitative analysis of bioactive sphingolipids by high-performance liquid chromatography-tandem mass spectrometry. , 2006, Methods.
[52] S. Spiegel,et al. Sphingosine 1-phosphate and ceramide 1-phosphate: expanding roles in cell signaling , 2005, Journal of Cell Science.
[53] S. Summers,et al. Acid Ceramidase Overexpression Prevents the Inhibitory Effects of Saturated Fatty Acids on Insulin Signaling* , 2005, Journal of Biological Chemistry.
[54] K. Asanuma,et al. Synaptopodin regulates the actin-bundling activity of α-actinin in an isoform-specific manner , 2005 .
[55] O. Eickelberg,et al. Caveolin-1 Facilitates Mechanosensitive Protein Kinase B (Akt) Signaling In Vitro and In Vivo , 2005, Circulation research.
[56] K. Asanuma,et al. Synaptopodin regulates the actin-bundling activity of alpha-actinin in an isoform-specific manner. , 2005, The Journal of clinical investigation.
[57] J. G. van den Berg,et al. Podocyte foot process effacement is not correlated with the level of proteinuria in human glomerulopathies. , 2004, Kidney international.
[58] F. Liu,et al. Regulation of Insulin Action by Ceramide , 2004, Journal of Biological Chemistry.
[59] Y. Igarashi,et al. Ceramide Kinase Is a Mediator of Calcium-dependent Degranulation in Mast Cells* , 2004, Journal of Biological Chemistry.
[60] S. Stratford. Regulation of insulin action by ceramide : dual mechanisms linking ceramide accumulation to the inhibition of Akt/protein kinase , 2004 .
[61] R. DeFronzo,et al. Ceramide content is increased in skeletal muscle from obese insulin-resistant humans. , 2004, Diabetes.
[62] M. Lisanti,et al. Role of caveolin and caveolae in insulin signaling and diabetes. , 2003, American journal of physiology. Endocrinology and metabolism.
[63] R. Dobrowsky,et al. A Role for Ceramide, but Not Diacylglycerol, in the Antagonism of Insulin Signal Transduction by Saturated Fatty Acids* , 2003, The Journal of Biological Chemistry.
[64] L. Holzman,et al. Podocyte‐specific expression of cre recombinase in transgenic mice , 2003, Genesis.
[65] David S. Park,et al. Caveolin-1-deficient Mice Are Lean, Resistant to Diet-induced Obesity, and Show Hypertriglyceridemia with Adipocyte Abnormalities* , 2002, The Journal of Biological Chemistry.
[66] M. O'hare,et al. A conditionally immortalized human podocyte cell line demonstrating nephrin and podocin expression. , 2002, Journal of the American Society of Nephrology : JASN.
[67] C. Kahn,et al. Selective insulin signaling through A and B insulin receptors regulates transcription of insulin and glucokinase genes in pancreatic beta cells. , 2001, Molecular cell.
[68] Philippe Soriano,et al. Widespread recombinase expression using FLPeR (Flipper) mice , 2000, Genesis.
[69] Y. Toya,et al. Caveolin Is an Activator of Insulin Receptor Signaling* , 1998, The Journal of Biological Chemistry.
[70] G. S. Montes,et al. STRUCTURAL BIOLOGY OF THE FIBRES OF THE COLLAGENOUS AND ELASTIC SYSTEMS , 1996, Cell biology international.
[71] O. Boudker,et al. Detection and characterization of ceramide-1-phosphate phosphatase activity in rat liver plasma membrane. , 1993, The Journal of biological chemistry.
[72] D. O'sullivan,et al. 1,2-Diacylglycerol and ceramide levels in insulin-resistant tissues of the rat in vivo. , 1990, The Journal of biological chemistry.