Molecular cause of the severe functional deficiency in osteoclasts by an arginine deletion in the basic domain of Mi transcription factor

[1]  E. Morii,et al.  mi-transcription factor as a regulator of mast cell differentiation. , 2000, International journal of hematology.

[2]  Michael C. Ostrowski,et al.  The Microphthalmia Transcription Factor Regulates Expression of the Tartrate‐Resistant Acid Phosphatase Gene During Terminal Differentiation of Osteoclasts , 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[3]  E. Morii,et al.  Involvement of mi-Transcription Factor in Expression of α-Melanocyte-Stimulating Hormone Receptor in Cultured Mast Cells of Mice1 2 , 2000, The Journal of Immunology.

[4]  E. Morii,et al.  Synergy of PEBP2/CBF with mi transcription factor (MITF) for transactivation of mouse mast cell protease 6 gene , 1999, Oncogene.

[5]  E. Morii,et al.  Inhibitory effect of the transcription factor encoded by the mi mutant allele in cultured mast cells of mice. , 1999, Blood.

[6]  T. Ochi,et al.  Microphthalmia-associated transcription factor interacts with PU.1 and c-Fos: determination of their subcellular localization. , 1999, Biochemical and biophysical research communications.

[7]  M. McKee,et al.  Mice Lacking Osteopontin Show Normal Development and Bone Structure but Display Altered Osteoclast Formation In Vitro , 1998, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[8]  S. Mckercher,et al.  Osteopetrosis in mice lacking haematopoietic transcription factor PU.1 , 1997, Nature.

[9]  K Takahashi,et al.  Functional Analysis of Microphthalmia-associated Transcription Factor in Pigment Cell-specific Transcription of the Human Tyrosinase Family Genes* , 1997, The Journal of Biological Chemistry.

[10]  E. Morii,et al.  The recessive phenotype displayed by a dominant negative microphthalmia-associated transcription factor mutant is a result of impaired nucleation potential , 1996, Molecular and cellular biology.

[11]  E. Morii,et al.  Loss of DNA binding ability of the transcription factor encoded by the mutant mi locus. , 1994, Biochemical and biophysical research communications.

[12]  James A. Vaught,et al.  microphthalmia, a critical factor in melanocyte development, defines a discrete transcription factor family. , 1994, Genes & development.

[13]  E. Wagner,et al.  c-Fos: a key regulator of osteoclast-macrophage lineage determination and bone remodeling. , 1994, Science.

[14]  B. de Crombrugghe,et al.  TFEC, a basic helix-loop-helix protein, forms heterodimers with TFE3 and inhibits TFE3-dependent transcription activation , 1993, Molecular and cellular biology.

[15]  N. Jenkins,et al.  Mutations at the mouse microphthalmia locus are associated with defects in a gene encoding a novel basic-helix-loop-helix-zipper protein , 1993, Cell.

[16]  P. Sharp,et al.  A helix-loop-helix protein related to the immunoglobulin E box-binding proteins , 1990, Molecular and cellular biology.

[17]  L. Su,et al.  TFE3: a helix-loop-helix protein that activates transcription through the immunoglobulin enhancer muE3 motif. , 1990, Genes & development.

[18]  J. Schneider,et al.  A mutant SV40 large T antigen interferes with nuclear localization of a heterologous protein , 1988, Cell.

[19]  D. G. Walker,et al.  The hematogenous origin of osteoclasts: experimental evidence from osteopetrotic (microphthalmic) mice treated with spleen cells from beige mouse donors. , 1981, The American journal of anatomy.

[20]  T. Kozel,et al.  Phagocytosis of Cryptococcus neoformans by normal and thioglycolate-activated macrophages , 1978, Infection and immunity.

[21]  N. Copeland,et al.  Mild osteopetrosis in the microphthalmia-oak ridge mouse. A model for intermediate autosomal recessive osteopetrosis in humans. , 1995, The American journal of pathology.

[22]  R. Mieremet,et al.  A morphologic study of osteoclasts isolated from osteopetrotic microphthalmic (mi/mi) mouse and human fetal long bones using an instrument permitting combination of light and scanning electron microscopy. , 1988, Bone.

[23]  M. Holtrop,et al.  The ultrastructure of osteoclasts in microphthalmic mice. , 1981, Metabolic bone disease & related research.

[24]  S. Marks,et al.  CHAPTER 6 – Mammalian Osteopetrosis—A Model for Studying Cellular and Humoral Factors in Bone Resorption , 1976 .