Excitatory Amino Acid Receptors in Brain
暂无分享,去创建一个
[1] C. Cotman,et al. Anatomical distributions of four pharmacologically distinct 3H-L-glutamate binding sites , 1983, Nature.
[2] J. Bockaert,et al. Pharmacological and functional characteristics of metabotropic excitatory amino acid receptors. , 1990, Trends in pharmacological sciences.
[3] P. Calabresi,et al. Modulatory action of metabotropic glutamate receptor (mGluR) 5 on mGluR1 function in striatal cholinergic interneurons , 2005, Neuropharmacology.
[4] W. Ong,et al. A Nuclear Microscopic Study of Elemental Changes in the Rat Hippocampus After Kainate‐Induced Neuronal Injury , 1999, Journal of neurochemistry.
[5] S. Ozawa,et al. Two types of kainate response in cultured rat hippocampal neurons. , 1991, Journal of neurophysiology.
[6] F. Pfrieger. Role of cholesterol in synapse formation and function. , 2003, Biochimica et biophysica acta.
[7] A. Rodríguez-Moreno,et al. Molecular physiology of kainate receptors. , 2001, Physiological reviews.
[8] Y. Ben-Ari,et al. Kainate, a double agent that generates seizures: two decades of progress , 2000, Trends in Neurosciences.
[9] A. Schousboe,et al. Novel Glutamate Receptor Antagonists Selectively Protect Against Kainic Acid Neurotoxicity in Cultured Cerebral Cortex Neurons , 1990, Journal of neurochemistry.
[10] Jérôme Ruel,et al. Negative allosteric modulation of AMPA-preferring receptors by the selective isomer GYKI 53784 (LY303070), a specific non-competitive AMPA antagonist. , 2006, CNS drug reviews.
[11] S. Sensi,et al. Ca2+–Zn2+ permeable AMPA or kainate receptors: possible key factors in selective neurodegeneration , 2000, Trends in Neurosciences.
[12] S. Petrou,et al. A putative fatty acid-binding domain of the NMDA receptor. , 1993, Trends in biochemical sciences.
[13] T. Endoh. Characterization of modulatory effects of postsynaptic metabotropic glutamate receptors on calcium currents in rat nucleus tractus solitarius , 2004, Brain Research.
[14] J. Hablitz,et al. Quisqualate induces an inward current via mGluR activation in neocortical pyramidal neurons , 2000, Brain Research.
[15] M. Mayer,et al. The physiology of excitatory amino acids in the vertebrate central nervous system , 1987, Progress in Neurobiology.
[16] B. Ballyk,et al. 4-Alkyl- and 4-cinnamylglutamic acid analogues are potent GluR5 kainate receptor agonists. , 2000, Journal of medicinal chemistry.
[17] G. Woodruff,et al. In vivo interaction of a polyamine with the NMDA receptor. , 1990, European journal of pharmacology.
[18] L. Horrocks,et al. A light and electron microscopic study of cytoplasmic phospholipase A2 and cyclooxygenase-2 in the hippocampus after kainate lesions , 1998, Brain Research.
[19] D. Choi,et al. Glutamate neurotoxicity and diseases of the nervous system , 1988, Neuron.
[20] M. Nieto,et al. GluR5 and GluR6 Kainate Receptor Subunits Coexist in Hippocampal Neurons and Coassemble to Form Functional Receptors , 2000, The Journal of Neuroscience.
[21] J. Crabtree,et al. A Presynaptic Kainate Receptor Is Involved in Regulating the Dynamic Properties of Thalamocortical Synapses during Development , 2002, Neuron.
[22] R. Miller,et al. A glutamate receptor regulates Ca2+ mobilization in hippocampal neurons. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[23] J. Lerma,et al. A Mosaic of Functional Kainate Receptors in Hippocampal Interneurons , 2004, The Journal of Neuroscience.
[24] Janet E. Paulsen,et al. Characterization of mGluR5R, a novel, metabotropic glutamate receptor 5-related gene. , 2002, Brain research. Molecular brain research.
[25] I. Holopainen,et al. Coupling of glutamatergic receptors to changes in intracellular Ca2+ in rat cerebellar granule cells in primary culture , 1990, Journal of neuroscience research.
[26] E. Michaelis. Molecular biology of glutamate receptors in the central nervous system and their role in excitotoxicity, oxidative stress and aging , 1998, Progress in Neurobiology.
[27] Richard J. Miller,et al. Ca2+ entry via AMPA/KA receptors and excitotoxicity in cultured cerebellar Purkinje cells , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[28] J. Henley,et al. Kainate receptors: subunits, synaptic localization and function. , 1999, Trends in pharmacological sciences.
[29] C. Cotman,et al. The excitatory amino acid receptors: their classes, pharmacology, and distinct properties in the function of the central nervous system. , 1989, Annual review of pharmacology and toxicology.
[30] J. Olney. Excitotoxicity and N‐methyl‐D‐Aspartate receptors , 1989 .
[31] L. Horrocks,et al. Neuroprotection abilities of cytosolic phospholipase A2 inhibitors in kainic acid-induced neurodegeneration. , 2004, Current drug targets. Cardiovascular & haematological disorders.
[32] R. Ransom,et al. Cooperative Modulation of [3H]MK‐801 Binding to the N‐Methyl‐d‐Aspartate Receptor‐Ion Channel Complex by l‐Glutamate, Glycine, and Polyamines , 1988, Journal of neurochemistry.
[33] J. Garthwaite,et al. Excitatory amino acid neurotoxicity and neurodegenerative disease. , 1990, Trends in pharmacological sciences.
[34] C. Cotman,et al. [3H]CPP, a new competitive ligand for NMDA receptors. , 1986, European journal of pharmacology.
[35] G. Collingridge,et al. Kainate receptors are involved in synaptic plasticity , 1999, Nature.
[36] T. Guilarte,et al. Biochemical evidence of an interaction of lead at the zinc allosteric sites of the NMDA receptor complex: effects of neuronal development. , 1995, Neurotoxicology.
[37] T. Bliss,et al. NMDA receptors - their role in long-term potentiation , 1987, Trends in Neurosciences.
[38] E. P. Huang,et al. Metal ions and synaptic transmission: think zinc. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[39] P. Contreras,et al. A review of the in vitro and in vivo neurochemical characterization of the NMDA/PCP/Glycine/Ion channel receptor macrocomplex , 1990, Neurochemical Research.
[40] M. Mayer,et al. Micromolar concentrations of Zn2+ antagonize NMDA and GABA responses of hippocampal neurons , 1987, Nature.
[41] Stephen J. Smith,et al. NMDA-receptor activation increases cytoplasmic calcium concentration in cultured spinal cord neurones , 1986, Nature.
[42] G. Sperk,et al. Kainic acid seizures in the rat , 1994, Progress in Neurobiology.
[43] I. Holopainen,et al. Glutamate receptor agonists increase intracellular Ca2+ independently of voltage-gated Ca2+ channels in rat cerebellar granule cells , 1989, Neuroscience Letters.
[44] W. Ong,et al. Differential expression of apolipoprotein D and apolipoprotein E in the kainic acid-lesioned rat hippocampus , 1997, Neuroscience.
[45] G. Collingridge,et al. Roles of metabotropic glutamate receptors in LTP and LTD in , 1999, Current Opinion in Neurobiology.
[46] Robert Thomas. Exitatory Amino Acids in Health and Disease , 1995 .
[47] M. Kassiou,et al. Radiosynthesis and pharmacological evaluation of [11C]EMD-95885: a high affinity ligand for NR2B-containing NMDA receptors. , 2004, Bioorganic & medicinal chemistry.
[48] Á. Simonyi,et al. Kainic acid-mediated excitotoxicity as a model for neurodegeneration , 2007, Molecular Neurobiology.
[49] M. Ehlers,et al. Learning from NMDA Receptor Trafficking: Clues to the Development and Maturation of Glutamatergic Synapses , 2004, Neurosignals.
[50] J. Lerma,et al. Kainate Reveals Its Targets , 1997, Neuron.
[51] B. Halliwell,et al. Increase in Cholesterol and Cholesterol Oxidation Products, and Role of Cholesterol Oxidation Products in Kainate‐induced Neuronal Injury , 2003, Brain pathology.