A subset of neutrophils in human systemic inflammation inhibits T cell responses through Mac-1.
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Leo Koenderman | Peter Pickkers | J. Lammers | L. Koenderman | E. van Hoffen | P. Pickkers | J. Pillay | L. Leenen | Jan-Willem Lammers | Laurien H Ulfman | Els van Hoffen | Janesh Pillay | Vera M Kamp | Tjaakje Visser | Tamar Tak | Luke P Leenen | L. Ulfman | T. Visser | T. Tak | V. Kamp
[1] P. Dahm,et al. Reversal of Myeloid Cell–Mediated Immunosuppression in Patients with Metastatic Renal Cell Carcinoma , 2008, Clinical Cancer Research.
[2] Timothy A. Springer,et al. Adhesion receptors of the immune system , 1990, Nature.
[3] C. Leclerc,et al. Coactivation of Syk kinase and MyD88 adaptor protein pathways by bacteria promotes regulatory properties of neutrophils. , 2009, Immunity.
[4] Nicholas R. English,et al. Increased Production of Immature Myeloid Cells in Cancer Patients: A Mechanism of Immunosuppression in Cancer1 , 2001, The Journal of Immunology.
[5] L. Fiette,et al. Neutrophils rapidly migrate via lymphatics after Mycobacterium bovis BCG intradermal vaccination and shuttle live bacilli to the draining lymph nodes. , 2005, Blood.
[6] J. Borghans,et al. In vivo labeling with 2H2O reveals a human neutrophil lifespan of 5.4 days. , 2010, Blood.
[7] L. Koenderman,et al. Human neutrophils switch to an activated phenotype after homing to the lung irrespective of inflammatory disease , 2009, Clinical and experimental immunology.
[8] T. Mayadas,et al. Mac-1 (CD11b/CD18) is essential for Fc receptor-mediated neutrophil cytotoxicity and immunologic synapse formation. , 2001, Blood.
[9] J. Lammers,et al. Role of Ca2+/calmodulin regulated signaling pathways in chemoattractant induced neutrophil effector functions. Comparison with the role of phosphotidylinositol-3 kinase. , 2002, European journal of biochemistry.
[10] Leo Koenderman,et al. Functional heterogeneity and differential priming of circulating neutrophils in human experimental endotoxemia , 2010, Journal of leukocyte biology.
[11] M. Atkins,et al. Arginase I-producing myeloid-derived suppressor cells in renal cell carcinoma are a subpopulation of activated granulocytes. , 2009, Cancer research.
[12] M. Glogauer,et al. Timing of neutrophil tissue repopulation predicts restoration of innate immune protection in a murine bone marrow transplantation model. , 2006, Blood.
[13] N. Bhardwaj. Harnessing the immune system to treat cancer. , 2007, The Journal of clinical investigation.
[14] T. Padhya,et al. Mechanism Regulating Reactive Oxygen Species in Tumor-Induced Myeloid-Derived Suppressor Cells1 , 2009, The Journal of Immunology.
[15] A. Kumar,et al. Cytomegalovirus Reactivation in Critically Ill Immunocompetent Patients , 2009 .
[16] S. Signoretti,et al. Arginase-producing myeloid suppressor cells in renal cell carcinoma patients: a mechanism of tumor evasion. , 2005, Cancer research.
[17] J. Gallin. Human neutrophil heterogeneity exists, but is it meaningful? , 1984, Blood.
[18] A. Ho,et al. Arginase I is constitutively expressed in human granulocytes and participates in fungicidal activity. , 2005, Blood.
[19] J. G. van der Hoeven,et al. In Vivo Evidence for Nitric Oxide–Mediated Calcium-Activated Potassium-Channel Activation During Human Endotoxemia , 2006, Circulation.
[20] H. Agut,et al. Herpes simplex virus lung infection in patients undergoing prolonged mechanical ventilation. , 2007, American journal of respiratory and critical care medicine.
[21] L. Moldawer,et al. MyD88-dependent expansion of an immature GR-1+CD11b+ population induces T cell suppression and Th2 polarization in sepsis , 2007, The Journal of experimental medicine.
[22] D. Vestweber,et al. Active MAC-1 (CD11b/CD18) on DCs inhibits full T-cell activation. , 2007, Blood.
[23] I. Müller,et al. Polymorphonuclear neutrophils and T lymphocytes: strange bedfellows or brothers in arms? , 2009, Trends in immunology.
[24] Michelle Collazo,et al. Subsets of Myeloid-Derived Suppressor Cells in Tumor-Bearing Mice1 , 2008, The Journal of Immunology.
[25] G. Findlay,et al. IMMATURE CIRCULATING NEUTROPHILS IN SEPSIS HAVE IMPAIRED PHAGOCYTOSIS AND CALCIUM SIGNALING , 2008, Shock.
[26] Y. Samstag,et al. Oxidation of cofilin mediates T cell hyporesponsiveness under oxidative stress conditions. , 2008, Immunity.
[27] J. Piette,et al. Crucial Role of the Amino-Terminal Tyrosine Residue 42 and the Carboxyl-Terminal PEST Domain of IκBα in NF-κB Activation by an Oxidative Stress1 , 2000, The Journal of Immunology.
[28] D. Quiceno,et al. L-arginine availability regulates T-lymphocyte cell-cycle progression. , 2007, Blood.
[29] J. Hogg,et al. Glucocorticoid-induced granulocytosis: contribution of marrow release and demargination of intravascular granulocytes. , 1998, Circulation.
[30] S. Slavin,et al. CpG-induced myeloid CD11b+Gr-1+ cells efficiently suppress T cell-mediated immunoreactivity and graft-versus-host disease in a murine model of allogeneic cell therapy. , 2008, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.
[31] C. Winterbourn,et al. Lethal Weapons , 2002, Science.
[32] R. Beelen,et al. Immature Neutrophils Mediate Tumor Cell Killing via IgA but Not IgG Fc Receptors1 , 2005, The Journal of Immunology.
[33] L. Moldawer,et al. MyD 88-dependent expansion of an immature GR-1 + CD 11 b + population induces T cell suppression and Th 2 polarization in sepsis , 2007 .
[34] S. Bloom,et al. Phagocyte dysfunction and inflammatory bowel disease. , 2008, Inflammatory bowel diseases.
[35] D. Gabrilovich,et al. Myeloid-Derived Suppressor Cells in Human Cancer , 2010, Cancer journal.
[36] P. De Baetselier,et al. Identification of discrete tumor-induced myeloid-derived suppressor cell subpopulations with distinct T cell-suppressive activity. , 2008, Blood.
[37] R. Holmdahl,et al. T cell surface redox levels determine T cell reactivity and arthritis susceptibility , 2006, Proceedings of the National Academy of Sciences.
[38] C. Coopersmith,et al. The sepsis seesaw: tilting toward immunosuppression , 2009, Nature Medicine.
[39] Miriam V. Liscovsky,et al. Presence of neutrophil-bearing antigen in lymphoid organs of immune mice. , 2006, Blood.
[40] Shu-Hsia Chen,et al. Gr-1+ Myeloid Cells Derived from Tumor-Bearing Mice Inhibit Primary T Cell Activation Induced Through CD3/CD28 Costimulation1 , 2000, The Journal of Immunology.
[41] R. Holmdahl,et al. Macrophages suppress T cell responses and arthritis development in mice by producing reactive oxygen species. , 2007, The Journal of clinical investigation.
[42] Srinivas Nagaraj,et al. Myeloid-derived suppressor cells as regulators of the immune system , 2009, Nature Reviews Immunology.
[43] M. Manns,et al. Myeloid-derived suppressor cells in inflammatory bowel disease: a new immunoregulatory pathway. , 2008, Gastroenterology.
[44] A. Sharpe,et al. CD80+Gr-1+ Myeloid Cells Inhibit Development of Antifungal Th1 Immunity in Mice with Candidiasis1 , 2002, The Journal of Immunology.
[45] E. Christensen,et al. Arginase 1 is expressed in myelocytes/metamyelocytes and localized in gelatinase granules of human neutrophils. , 2007, Blood.
[46] J. Ochoa,et al. CD11b+/Gr-1+ Myeloid Suppressor Cells Cause T Cell Dysfunction after Traumatic Stress1 , 2006, The Journal of Immunology.