Ovarian function is effectively inhibited by a low-dose triphasic oral contraceptive containing ethinylestradiol and levonorgestrel.
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[1] P. Franchimont,et al. Plasma hormone levels in women receiving new oral contraceptives containing ethinyl estradiol plus levonorgestrel or desogestrel. , 1983, Contraception.
[2] A. Vermeulen,et al. Metabolic effects of the triphasic oral contraceptive Trigynon. , 1982, Contraception.
[3] M. Raj,et al. Normalization of Testosterone Levels Using a Low Estrogen-Containing Oral Contraceptive in Women with Polycystic Ovary Syndrome , 1982, Obstetrics and gynecology.
[4] R. Wild,et al. Adrenal function in hirsutism. II. Effect of an oral contraceptive. , 1982, The Journal of clinical endocrinology and metabolism.
[5] W. Spellacy. Carbohydrate metabolism during treatment with estrogen, progestogen, and low-dose oral contraceptives. , 1982, American journal of obstetrics and gynecology.
[6] M. Briggs. Randomized Prospective Studies on Metabolic Effects of Oral Contraceptives , 1982, Acta obstetricia et gynecologica Scandinavica. Supplement.
[7] P. Wahl,et al. Oral contraceptive and postmenopausal estrogen effects on lipoprotein triglyceride and cholesterol in an adult female population: relationships to estrogen and progestin potency. , 1981, The Journal of clinical endocrinology and metabolism.
[8] B. Cohen,et al. Further studies on pituitary and ovarian function in women receiving hormonal contraception. , 1981, Contraception.
[9] L. Wallentin,et al. Lipoprotein changes may be minimized by proper composition of a combined oral contraceptive. , 1981, Fertility and sterility.
[10] P. Kalra,et al. Pituitary and ovarian responsiveness to a graded gonadotropin releasing factor stimulation test in women using a low-estrogen or a regular type of oral contraceptive. , 1980, American journal of obstetrics and gynecology.
[11] J. Duvivier,et al. Chromatographie des stéroïdes gonadiques sur Sephadex LH-20 , 1976 .
[12] K. Aktories,et al. Dose-dependent inhibition by oral contraceptives of the pituitary to release LH and FSH in response to stimulation with LH-RH+. , 1976, Contraception.
[13] K. Aktories,et al. [The effect of different types of oral contraceptives upon ovarian function (author's transl)]. , 1976, Geburtshilfe und Frauenheilkunde.
[14] W. Wiser,et al. The effectiveness of two oral contraceptives in suppressing plasma androstenedione, testosterone, LH, and FSH, and in stimulating plasma testosterone-binding capacity in hirsute women. , 1976, American journal of obstetrics and gynecology.
[15] E. Johansson. Plasma Levels of Progesterone and Oestrogens in Women Treated with an Oral Contraceptive of Low Oestrogen Content (Ovostat 1375) , 1975, Acta obstetricia et gynecologica Scandinavica.
[16] H. Meiers,et al. Wirkungen von Antikonzeptiva auf Alopecia androgenetica, Seborrhoea oleosa, Akne vulgaris und Hirsutismus , 1974 .
[17] A. Vermeulen,et al. The apparent free testosterone concentration, an index of androgenicity. , 1971, The Journal of clinical endocrinology and metabolism.
[18] R. Greenblatt,et al. The Development of a New Triphasic Oral Contraceptive , 1984, Springer Netherlands.
[19] J. Colau,et al. [Residual ovarian function after minidoses of estroprogestative agents]. , 1983, Journal de gynecologie, obstetrique et biologie de la reproduction.
[20] A. Haspels,et al. Benefits and Risks of Hormonal Contraception , 1982, Springer Netherlands.
[21] P. Keller. Hirsutism and Virilism , 1981 .
[22] K. Aktories. Die Beeinflussung des Ovarialzyklus durch verschiedene Typen hormonaler Kontrazeptiva , 1977 .
[23] P. Franchimont,et al. Homologous radioimmunoassay for human prolactin. , 1976, International journal of nuclear medicine and biology.
[24] J. Spona,et al. Inhibition of ovulation by means of a combined preparation with reduced amounts of active substance , 1974 .