m6A-mRNA methylation regulates cardiac gene expression and cellular growth
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Christoph Dieterich | Hugo A Katus | Janine Altmüller | Rewati Tappu | Etienne Boileau | C. Dieterich | M. Völkers | P. Most | Rewati Tappu | H. Katus | Eva Riechert | C. Hofmann | A. Górska | Etienne Boileau | Brandon Malone | Ellen Malovrh | J. Altmüller | Vivien Kmietczyk | Eva Riechert | Laura Kalinski | Ellen Malovrh | Brandon Malone | Agnieszka Gorska | Christoph Hofmann | Eshita Varma | Lonny Jürgensen | Verena Kamuf-Schenk | Martin Busch | Patrick Most | Mirko Völkers | M. Busch | Laura Kalinski | L. Jürgensen | V. Kamuf-Schenk | V. Kmietczyk | E. Varma
[1] G. Akusjärvi,et al. Gene expression, regulation of , 1995 .
[2] Thomas R. Gingeras,et al. STAR: ultrafast universal RNA-seq aligner , 2013, Bioinform..
[3] Francine E. Garrett-Bakelman,et al. The N6-methyladenosine (m6A)-forming enzyme METTL3 controls myeloid differentiation of normal and leukemia cells , 2017, Nature Medicine.
[4] Davis J. McCarthy,et al. Differential expression analysis of multifactor RNA-Seq experiments with respect to biological variation , 2012, Nucleic acids research.
[5] Erez Y. Levanon,et al. m6A mRNA methylation facilitates resolution of naïve pluripotency toward differentiation , 2015, Science.
[6] E. Graves. National Hospital Discharge Survey. , 1989, Vital and health statistics. Series 13, Data from the National Health Survey.
[7] J. Hanna,et al. The N6-Methyladenosine mRNA Methylase METTL3 Controls Cardiac Homeostasis and Hypertrophy , 2018, Circulation.
[8] M. Kupiec,et al. Topology of the human and mouse m6A RNA methylomes revealed by m6A-seq , 2012, Nature.
[9] Paul Lehner,et al. Fat mass and obesity-related (FTO) shuttles between the nucleus and cytoplasm , 2014, Bioscience reports.
[10] David A. Kass,et al. Tackling heart failure in the twenty-first century , 2008, Nature.
[11] F. Sheikh,et al. Functions of myosin light chain-2 (MYL2) in cardiac muscle and disease. , 2015, Gene.
[12] Chengqi Yi,et al. N6-Methyladenosine in Nuclear RNA is a Major Substrate of the Obesity-Associated FTO , 2011, Nature chemical biology.
[13] S. Tavazoie,et al. N6-methyladenosine marks primary microRNAs for processing , 2015, Nature.
[14] O. Elemento,et al. Comprehensive Analysis of mRNA Methylation Reveals Enrichment in 3′ UTRs and near Stop Codons , 2012, Cell.
[15] C. Dieterich,et al. Bayesian prediction of RNA translation from ribosome profiling , 2017, Nucleic acids research.
[16] M. Latronico,et al. Epigenetics: a new mechanism of regulation of heart failure? , 2013, Basic Research in Cardiology.
[17] Zhike Lu,et al. Differential m6A, m6Am, and m1A Demethylation Mediated by FTO in the Cell Nucleus and Cytoplasm. , 2018, Molecular cell.
[18] Mark A Sussman,et al. PRAS40 prevents development of diabetic cardiomyopathy and improves hepatic insulin sensitivity in obesity , 2013, EMBO molecular medicine.
[19] J. Ross,et al. Segregation of atrial-specific and inducible expression of an atrial natriuretic factor transgene in an in vivo murine model of cardiac hypertrophy , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[20] M. Boerries,et al. Cardiac adenoviral S100A1 gene delivery rescues failing myocardium. , 2004, The Journal of clinical investigation.
[21] C. Dieterich,et al. Identification of circular RNAs with host gene-independent expression in human model systems for cardiac differentiation and disease. , 2017, Journal of molecular and cellular cardiology.
[22] Carol DeFrances,et al. 2006 National Hospital Discharge Survey. , 2005, National health statistics reports.
[23] R. Palmiter,et al. Cell-type-specific isolation of ribosome-associated mRNA from complex tissues , 2009, Proceedings of the National Academy of Sciences.
[24] Gunter Meister,et al. Interactions, localization, and phosphorylation of the m6A generating METTL3–METTL14–WTAP complex , 2018, RNA.
[25] Olivier Elemento,et al. 5′ UTR m6A Promotes Cap-Independent Translation , 2015, Cell.
[26] Chuan He,et al. Grand challenge commentary: RNA epigenetics? , 2010, Nature chemical biology.
[27] M. Ehrenberg,et al. N6-methyladenosine in mRNA disrupts tRNA selection and translation elongation dynamics , 2016, Nature Structural &Molecular Biology.
[28] Yi Xing,et al. m6A-LAIC-seq reveals the census and complexity of the m6A epitranscriptome , 2016, Nature Methods.
[29] Yu Zhang,et al. m6A facilitates hippocampus-dependent learning and memory through Ythdf1 , 2018, Nature.
[30] N J Izzo,et al. HL-1 cells: a cardiac muscle cell line that contracts and retains phenotypic characteristics of the adult cardiomyocyte. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[31] Nicholas T. Ingolia. Ribosome Footprint Profiling of Translation throughout the Genome , 2016, Cell.
[32] Samie R. Jaffrey,et al. The dynamic epitranscriptome: N6-methyladenosine and gene expression control , 2014, Nature Reviews Molecular Cell Biology.
[33] Miao Yu,et al. A METTL3-METTL14 complex mediates mammalian nuclear RNA N6-adenosine methylation , 2013, Nature chemical biology.
[34] Mark A Sussman,et al. mTORC2 Protects the Heart from Ischemic Damage , 2013 .
[35] Gideon Rechavi,et al. Gene expression regulation mediated through reversible m6A RNA methylation , 2014, Nature Reviews Genetics.
[36] Mee-Sup Yoon,et al. XPLN is an endogenous inhibitor of mTORC2 , 2013, Proceedings of the National Academy of Sciences.
[37] E. Ashley,et al. A long non-coding RNA protects the heart from pathological hypertrophy , 2014, Nature.
[38] J. Sadoshima,et al. New Insights Into the Role of mTOR Signaling in the Cardiovascular System. , 2018, Circulation research.
[39] Alexandra King. Heart failure: Placing an EMPHASIS on the mineralocorticoid receptor—benefit of eplerenone in mild HF , 2010, Nature Reviews Cardiology.
[40] Jianbo Li,et al. The gene expression fingerprint of human heart failure , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[41] Chuan He,et al. YTHDF3 facilitates translation and decay of N6-methyladenosine-modified RNA , 2017, Cell Research.
[42] Mark A Sussman,et al. Pathological hypertrophy amelioration by PRAS40-mediated inhibition of mTORC1 , 2013, Proceedings of the National Academy of Sciences.
[43] D. Kass,et al. Reverse remodeling in heart failure—mechanisms and therapeutic opportunities , 2012, Nature Reviews Cardiology.
[44] Chuan He,et al. N 6 -methyladenosine Modulates Messenger RNA Translation Efficiency , 2015, Cell.
[45] Olivier Elemento,et al. Reversible methylation of m6Am in the 5′ cap controls mRNA stability , 2016, Nature.
[46] Philippe Froguel,et al. Loss-of-function mutation in the dioxygenase-encoding FTO gene causes severe growth retardation and multiple malformations. , 2009, American journal of human genetics.
[47] R. Hajjar,et al. FTO-Dependent N6-Methyladenosine Regulates Cardiac Function During Remodeling and Repair , 2018, Circulation.
[48] Ran Elkon,et al. Transcription Impacts the Efficiency of mRNA Translation via Co-transcriptional N6-adenosine Methylation , 2017, Cell.
[49] W. Gilbert,et al. Messenger RNA modifications: Form, distribution, and function , 2016, Science.
[50] Mark A Sussman,et al. Mechanistic Target of Rapamycin Complex 2 Protects the Heart From Ischemic Damage , 2013, Circulation.
[51] Mark A Sussman,et al. Hrd1 and ER-Associated Protein Degradation, ERAD, are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes. , 2015, Circulation research.
[52] Martin Vingron,et al. Translational regulation shapes the molecular landscape of complex disease phenotypes , 2015, Nature Communications.
[53] L. Kadaja,et al. Distinct organization of energy metabolism in HL-1 cardiac cell line and cardiomyocytes. , 2008, Biochimica et biophysica acta.
[54] Karen S. Frese,et al. Epigenome-Wide Association Study Identifies Cardiac Gene Patterning and a Novel Class of Biomarkers for Heart Failure , 2017, Circulation.