High glucose‐induced oxidative stress and mitochondrial dysfunction in neurons
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J. Olzmann | E. Feldman | A. Vincent | J. Russell | James A. Olzmann | Eva L. Feldman | James W. Russell | David Golovoy | Andrea M. Vincent | Pia Mahendru | James A. Olzmann | Alice Mentzer | A. Mentzer | D. Golovoy | Pia Mahendru | David Golovoy | J. Russell | Alice Mentzer
[1] T. Vanden Hoek,et al. Reactive Oxygen Species Released from Mitochondria during Brief Hypoxia Induce Preconditioning in Cardiomyocytes* , 1998, The Journal of Biological Chemistry.
[2] Hideyuki Sasaki,et al. Neuropathology and blood flow of nerve, spinal roots and dorsal root ganglia in longstanding diabetic rats , 1997, Acta Neuropathologica.
[3] Z. Lodin,et al. The metabolism of glucose of nerve cells cultivated under different conditions. , 1976, Acta histochemica.
[4] C. Cheng,et al. Does diabetes target ganglion neurones? Progressive sensory neurone involvement in long-term experimental diabetes. , 2001, Brain : a journal of neurology.
[5] I. G. Fantus,et al. Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylation. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[6] W. Tatton,et al. Mitochondrial Membrane Potential and Nuclear Changes in Apoptosis Caused by Serum and Nerve Growth Factor Withdrawal: Time Course and Modification by (؊)-deprenyl , 2022 .
[7] E. Feldman,et al. Insulin-like growth factor-I prevents apoptosis in sympathetic neurons exposed to high glucose. , 1999, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[8] A. Windebank,et al. Neurons Undergo Apoptosis in Animal and Cell Culture Models of Diabetes , 1999, Neurobiology of Disease.
[9] D. Greene,et al. Effects of DL-alpha-lipoic acid on peripheral nerve conduction, blood flow, energy metabolism, and oxidative stress in experimental diabetic neuropathy. , 2000, Diabetes.
[10] E. Feldman,et al. Glucose-induced oxidative stress and programmed cell death in diabetic neuropathy. , 1999, European journal of pharmacology.
[11] Y Li,et al. [Mitochondria and apoptosis]. , 2000, Zhonghua yu fang yi xue za zhi [Chinese journal of preventive medicine].
[12] S. Srinivasan,et al. Diabetic peripheral neuropathy: evidence for apoptosis and associated mitochondrial dysfunction. , 2000, Diabetes.
[13] P. Low,et al. Morphometry of dorsal root ganglion in chronic experimental diabetic neuropathy. , 2002, Diabetes.
[14] Eugene M. Johnson,et al. Evidence of a Novel Event during Neuronal Death Development of Competence-to-Die in Response to Cytoplasmic Cytochrome c , 1998, Neuron.
[15] A. Shahar. A Dissection and tissue culture manual of the nervous system , 1989 .
[16] Jean-Claude Martinou,et al. Bax-induced Cytochrome C Release from Mitochondria Is Independent of the Permeability Transition Pore but Highly Dependent on Mg2+ Ions , 1998, The Journal of cell biology.
[17] G. Kroemer,et al. The mitochondrial death/life regulator in apoptosis and necrosis. , 1998, Annual review of physiology.
[18] G. Kroemer,et al. Bid acts on the permeability transition pore complex to induce apoptosis , 2000, Oncogene.
[19] R. Kikkawa,et al. Apoptosis and impaired axonal regeneration of sensory neurons after nerve crush in diabetic rats , 2000, Neuroreport.
[20] Y. Kaneda,et al. Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage , 2000, Nature.
[21] V. Dixit,et al. Caspase-9, Bcl-XL, and Apaf-1 Form a Ternary Complex* , 1998, The Journal of Biological Chemistry.
[22] A. Vincent,et al. Oxidative Stress and Programmed Cell Death in Diabetic Neuropathy , 2002, Annals of the New York Academy of Sciences.
[23] M. V. Heiden,et al. Outer mitochondrial membrane permeability can regulate coupled respiration and cell survival. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[24] M. Brownlee. Biochemistry and molecular cell biology of diabetic complications , 2001, Nature.