Somatostatin release, electrical activity, membrane currents and exocytosis in human pancreatic delta cells

[1]  G. Rutter Regulating Glucagon Secretion: Somatostatin in the Spotlight , 2009, Diabetes.

[2]  C. C. Richardson,et al.  Somatostatin Secreted by Islet δ-Cells Fulfills Multiple Roles as a Paracrine Regulator of Islet Function , 2009, Diabetes.

[3]  Patrik Rorsman,et al.  Voltage-Gated Ion Channels in Human Pancreatic β-Cells: Electrophysiological Characterization and Role in Insulin Secretion , 2008, Diabetes.

[4]  M. Strowski,et al.  Function and expression of somatostatin receptors of the endocrine pancreas , 2008, Molecular and Cellular Endocrinology.

[5]  F. Ashcroft,et al.  R-type Ca2+-channel-evoked CICR regulates glucose-induced somatostatin secretion , 2007, Nature Cell Biology.

[6]  Paloma Alonso-Magdalena,et al.  Glucose Induces Opposite Intracellular Ca2+ Concentration Oscillatory Patterns in Identified α- and β-Cells Within Intact Human Islets of Langerhans , 2006, Diabetes.

[7]  Camillo Ricordi,et al.  The unique cytoarchitecture of human pancreatic islets has implications for islet cell function , 2006, Proceedings of the National Academy of Sciences of the United States of America.

[8]  E. Carneiro,et al.  Glucose induces opposite intracellular Ca2+ concentration oscillatory patterns in identified alpha- and beta-cells within intact human islets of Langerhans. , 2006, Diabetes.

[9]  Min Zhang,et al.  Pharmacological Properties and Functional Role of Kslow Current in Mouse Pancreatic β-Cells , 2005, The Journal of General Physiology.

[10]  Alvin C. Powers,et al.  Assessment of Human Pancreatic Islet Architecture and Composition by Laser Scanning Confocal Microscopy , 2005, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[11]  D. Roden,et al.  Pharmacological properties and functional role of Kslow current in mouse pancreatic beta-cells: SK channels contribute to Kslow tail current and modulate insulin secretion. , 2005, The Journal of general physiology.

[12]  C. Austin,et al.  Expression of voltage-gated potassium channels in human and rhesus pancreatic islets. , 2004, Diabetes.

[13]  M. Pipeleers-Marichal,et al.  A method for the purification of single A, B and D cells and for the isolation of coupled cells from isolated rat islets , 1981, Diabetologia.

[14]  H. Meissner,et al.  Membrane potential of beta-cells in pancreatic islets , 2004, Pflügers Archiv.

[15]  J. Adelman,et al.  Small-conductance calcium-activated K+ channels are expressed in pancreatic islets and regulate glucose responses. , 2003, Diabetes.

[16]  P. Stanfield,et al.  Constitutively active and G-protein coupled inward rectifier K+ channels: Kir2.0 and Kir3.0. , 2002, Reviews of physiology, biochemistry and pharmacology.

[17]  J. Walsh,et al.  Immunoneutralization of Somatostatin, Insulin, and Glucagon Causes Alterations in Islet Cell Secretion in the Isolated Perfused Human Pancreas , 2001, Pancreas.

[18]  A. Koschak,et al.  α1D (Cav1.3) Subunits Can Form L-type Ca2+ Channels Activating at Negative Voltages* , 2001, The Journal of Biological Chemistry.

[19]  P. Rorsman,et al.  Patch‐clamp characterisation of somatostatin‐secreting δ‐cells in intact mouse pancreatic islets , 2000, The Journal of physiology.

[20]  J. Henquin,et al.  Triggering and amplifying pathways of regulation of insulin secretion by glucose. , 2000, Diabetes.

[21]  L. Eliasson,et al.  Tight coupling between electrical activity and exocytosis in mouse glucagon-secreting alpha-cells. , 2000, Diabetes.

[22]  B. Soria,et al.  Different effects of tolbutamide and diazoxide in alpha, beta-, and delta-cells within intact islets of Langerhans. , 1999, Diabetes.

[23]  L. Eliasson,et al.  Activation of Ca(2+)-dependent K(+) channels contributes to rhythmic firing of action potentials in mouse pancreatic beta cells , 1999 .

[24]  Angel Nadal,et al.  Homologous and heterologous asynchronicity between identified α‐, β‐ and δ‐cells within intact islets of Langerhans in the mouse , 1999 .

[25]  B. Soria,et al.  Homologous and heterologous asynchronicity between identified alpha-, beta- and delta-cells within intact islets of Langerhans in the mouse. , 1999, The Journal of physiology.

[26]  A. Berts,et al.  Oscillatory Ca2+ signaling in somatostatin-producing cells from the human pancreas. , 1997, Metabolism: clinical and experimental.

[27]  Y. Kurachi,et al.  Inwardly rectifying potassium channels: their molecular heterogeneity and function. , 1997, The Japanese journal of physiology.

[28]  A. Berts,et al.  Glucose stimulation of somatostatin-producing islet cells involves oscillatory Ca2+ signaling. , 1996, Endocrinology.

[29]  J. Ruppersberg Ion Channels in Excitable Membranes , 1996 .

[30]  P. Smith,et al.  Block of ATP‐regulated and Ca2(+)‐activated K+ channels in mouse pancreatic beta‐cells by external tetraethylammonium and quinine. , 1990, The Journal of physiology.

[31]  Y. Michotte,et al.  Basal and Tolbutamide‐induced Plasma Somatostatin in Healthy Subjects and in Patients with Diabetes and Impaired Glucose Tolerance , 1989, Diabetic medicine : a journal of the British Diabetic Association.

[32]  R. Holman,et al.  Islet amyloid, increased A-cells, reduced B-cells and exocrine fibrosis: quantitative changes in the pancreas in type 2 diabetes. , 1988, Diabetes research.

[33]  J. Rinzel,et al.  Emergence of organized bursting in clusters of pancreatic beta-cells by channel sharing. , 1988, Biophysical journal.

[34]  P. Rorsman,et al.  Calcium and delayed potassium currents in mouse pancreatic beta‐cells under voltage‐clamp conditions. , 1986, The Journal of physiology.

[35]  B. Hille,et al.  Ionic channels of excitable membranes , 2001 .

[36]  V. Grill,et al.  A stimulating effect of glucose on somatostatin release is impaired in noninsulin-dependent diabetes mellitus. , 1984, The Journal of clinical endocrinology and metabolism.

[37]  S. Efendić,et al.  Effect of glucose/sulfonylurea interaction on release of insulin, glucagon, and somatostatin from isolated perfused rat pancreas. , 1979, Proceedings of the National Academy of Sciences of the United States of America.