Muscle atrophy and preferential loss of myosin in prolonged critically ill patients*
暂无分享,去创建一个
Sarah Derde | Ilse Vanhorebeek | G. Van den Berghe | F. Güiza | L. Larsson | I. Vanhorebeek | G. Hermans | F. Bruyninckx | Greet Van den Berghe | Greet Hermans | Inge Derese | Fabian Güiza | Yvette Hedström | Pieter J Wouters | Frans Bruyninckx | André D'Hoore | Lars Larsson | I. Derese | P. Wouters | A. D'hoore | Y. Hedström | S. Derde | Sarah Derde
[1] D. Ferrington,et al. Myosin and actin expression and oxidation in aging muscle , 2006, Experimental Gerontology.
[2] G. Van den Berghe,et al. Intensive insulin therapy in the medical ICU. , 2006, The New England journal of medicine.
[3] L. Larsson,et al. Acute quadriplegia and loss of muscle myosin in patients treated with nondepolarizing neuromuscular blocking agents and corticosteroids: Mechanisms at the cellular and molecular levels , 2000, Critical care medicine.
[4] L. Larsson,et al. Transcription Factors in Muscle Atrophy Caused by Blocked Neuromuscular Transmission and Muscle Unloading In Rats , 2007, Molecular medicine.
[5] L. Eriksson,et al. A porcine model of acute quadriplegic myopathy: a feasibility study , 2006, Acta anaesthesiologica Scandinavica.
[6] G. Biolo,et al. Treating hyperglycemia improves skeletal muscle protein metabolism in cancer patients after major surgery , 2008, Critical care medicine.
[7] P. Hasselgren. Ubiquitination, phosphorylation, and acetylation—triple threat in muscle wasting , 2007, Journal of cellular physiology.
[8] G. Van den Berghe,et al. Increasing intravenous glucose load in the presence of normoglycemia: Effect on outcome and metabolism in critically ill rabbits , 2010, Critical care medicine.
[9] G. Van den Berghe,et al. Impact of intensive insulin therapy on neuromuscular complications and ventilator dependency in the medical intensive care unit. , 2007, American journal of respiratory and critical care medicine.
[10] C. Lang,et al. Regulation of Myostatin by Glucocorticoids After Thermal Injury , 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[11] Isabelle Durand-Zaleski,et al. Paresis acquired in the intensive care unit: a prospective multicenter study. , 2002, JAMA.
[12] J. Vecchiet,et al. Human muscle aging: ROS-mediated alterations in rectus abdominis and vastus lateralis muscles , 2005, Experimental Gerontology.
[13] L. Visser,et al. Risk factors for the development of polyneuropathy and myopathy in critically ill patients , 2001, Critical care medicine.
[14] Marco Sandri,et al. Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophy , 2004, Cell.
[15] L. Edström,et al. Electrophoretic determination of the myosin/actin ratio in the diagnosis of critical illness myopathy , 2003, Intensive Care Medicine.
[16] D. Recupero,et al. Critical illness myopathy and neuropathy , 1996, The Lancet.
[17] J. Wang,et al. Energy-ubiquitin-dependent muscle proteolysis during sepsis in rats is regulated by glucocorticoids. , 1996, The Journal of clinical investigation.
[18] G. Biolo,et al. Insulin action on protein metabolism. , 1993, Bailliere's clinical endocrinology and metabolism.
[19] Arthur S Slutsky,et al. One-year outcomes in survivors of the acute respiratory distress syndrome. , 2003, The New England journal of medicine.
[20] J. Garnacho-Montero,et al. Critical illness polyneuropathy: risk factors and clinical consequences. A cohort study in septic patients , 2001, Intensive Care Medicine.
[21] G. D’Antona,et al. Myosin and actin content of human skeletal muscle fibers following 35 days bed rest , 2010, Scandinavian journal of medicine & science in sports.
[22] P. Vondracek,et al. Critical illness polyneuromyopathy: the electrophysiological components of a complex entity , 2003, Intensive Care Medicine.
[23] D. Attaix,et al. Myostatin Gene Deletion Prevents Glucocorticoid- Induced Muscle Atrophy , 2022 .
[24] R. Wolfe,et al. Extremity hyperinsulinemia stimulates muscle protein synthesis in severely injured patients. , 2004, American journal of physiology. Endocrinology and metabolism.
[25] B. Ahlman,et al. Proteasome proteolytic activity in skeletal muscle is increased in patients with sepsis. , 2007, Clinical science.
[26] van Mook Wn,et al. Critical illness polyneuropathy , 2002, Current opinion in critical care.
[27] Jan Gunst,et al. Insufficient activation of autophagy allows cellular damage to accumulate in critically ill patients. , 2011, The Journal of clinical endocrinology and metabolism.
[28] A. Mebazaa,et al. Muscular contractile failure in septic patients: role of the inducible nitric oxide synthase pathway. , 2000, American journal of respiratory and critical care medicine.
[29] C. Maganaris,et al. The temporal responses of protein synthesis, gene expression and cell signalling in human quadriceps muscle and patellar tendon to disuse , 2007, The Journal of physiology.
[30] M Schetz,et al. Intensive insulin therapy in critically ill patients. , 2001, The New England journal of medicine.
[31] Griffiths,et al. Muscle fibre atrophy in critically ill patients is associated with the loss of myosin filaments and the presence of lysosomal enzymes and ubiquitin , 1998, Neuropathology and applied neurobiology.
[32] T. Meyer,et al. Sepsis is associated with increased mRNAs of the ubiquitin-proteasome proteolytic pathway in human skeletal muscle. , 1997, The Journal of clinical investigation.
[33] F. Leijten,et al. The role of polyneuropathy in motor convalescence after prolonged mechanical ventilation. , 1996, JAMA.
[34] David D. Thomas,et al. Age-related decline in actomyosin structure and function , 2007, Experimental Gerontology.
[35] N. Matsumoto,et al. Analysis of muscle proteins in acute quadriplegic myopathy , 2000, Muscle & nerve.
[36] C. Bolton,et al. Critical illness polyneuropathy. A complication of sepsis and multiple organ failure. , 1987, Brain : a journal of neurology.
[37] Guoping Feng,et al. The Histone Deacetylase HDAC4 Connects Neural Activity to Muscle Transcriptional Reprogramming* , 2007, Journal of Biological Chemistry.
[38] L. Larsson,et al. Muscle paralysis and myosin loss in a patient with cancer cachexia. , 2007, Acta myologica : myopathies and cardiomyopathies : official journal of the Mediterranean Society of Myology.
[39] B. Bistrian,et al. Intensive insulin therapy in critically ill patients. , 2002, The New England journal of medicine.
[40] L. Larsson,et al. Effects of a preferential myosin loss on Ca2+ activation of force generation in single human skeletal muscle fibres , 2008, Experimental physiology.
[41] R. Stevens,et al. Hormonal status and ICU-acquired paresis in critically ill patients , 2010, Intensive Care Medicine.
[42] E. Zomer,et al. [Critical illness polyneuropathy]. , 1990, Nederlandsch tijdschrift voor geneeskunde.
[43] B. de Jonghe,et al. Intensive care unit-acquired weakness: Risk factors and prevention , 2009, Critical care medicine.
[44] M. Rich,et al. Direct muscle stimulation in acute quadriplegic myopathy , 1997, Muscle & nerve.
[45] B. Beaufrère,et al. Increased mRNA levels for components of the lysosomal, Ca2+-activated, and ATP-ubiquitin-dependent proteolytic pathways in skeletal muscle from head trauma patients. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[46] F Bruyninckx,et al. Insulin therapy protects the central and peripheral nervous system of intensive care patients , 2005, Neurology.
[47] R. Wolfe,et al. The effect of prolonged euglycemic hyperinsulinemia on lean body mass after severe burn. , 2002, Surgery.
[48] C. Ibebunjo,et al. Disparate dysfunction of skeletal muscles located near and distant from burn site in the rat , 2001, Muscle and Nerve.
[49] M. A. Chambers,et al. Physical inactivity and muscle weakness in the critically ill , 2009, Critical Care Medicine.
[50] L. Larsson,et al. Maximum velocity of shortening in relation to myosin isoform composition in single fibres from human skeletal muscles. , 1993, The Journal of physiology.
[51] E. Hoffman,et al. Gene expression and muscle fiber function in a porcine ICU model. , 2009, Physiological Genomics.
[52] P. Hasselgren,et al. Sepsis downregulates myostatin mRNA levels without altering myostatin protein levels in skeletal muscle , 2010, Journal of cellular biochemistry.
[53] P. Hasselgren,et al. Sepsis increases the expression and activity of the transcription factor Forkhead Box O 1 (FOXO1) in skeletal muscle by a glucocorticoid-dependent mechanism. , 2010, The international journal of biochemistry & cell biology.
[54] C. Lang,et al. Burn-induced increase in atrogin-1 and MuRF-1 in skeletal muscle is glucocorticoid independent but downregulated by IGF-I. , 2007, American journal of physiology. Regulatory, integrative and comparative physiology.
[55] J. Thissen,et al. Mechanisms of glucocorticoid-induced myopathy. , 2008, The Journal of endocrinology.
[56] L. Larsson. Acute quadriplegic myopathy: an acquired "myosinopathy". , 2008, Advances in experimental medicine and biology.
[57] L. Dušek,et al. Risk factors for critical illness polyneuromyopathy , 2005, Journal of Neurology.
[58] W. Sibbald,et al. Critically ill polyneuropathy: electrophysiological studies and differentiation from Guillain-Barré syndrome. , 1986, Journal of neurology, neurosurgery, and psychiatry.