Blunted effect of parathyroid hormone on adenosine 3′,5′-cyclic monophosphate production is derived from ATP depletion in proximal convoluted tubules of hypophosphatemic mice

[1]  R. Mason,et al.  The PEX gene: not a simple answer for X-linked hypophosphataemic rickets and oncogenic osteomalacia , 1997, Molecular and Cellular Endocrinology.

[2]  P. Rowe,et al.  The PEX gene: its role in X-linked rickets, osteomalacia, and bone mineral metabolism. , 1997, Experimental nephrology.

[3]  F. Glorieux,et al.  cDNA cloning of the murine Pex gene implicated in X-linked hypophosphatemia and evidence for expression in bone. , 1996, Genomics.

[4]  A. Poustka,et al.  A gene (PEX) with homologies to endopeptidases is mutated in patients with X–linked hypophosphatemic rickets , 1995, Nature Genetics.

[5]  A. Werner,et al.  Renal Na(+)-phosphate cotransport in murine X-linked hypophosphatemic rickets. Molecular characterization. , 1994, The Journal of clinical investigation.

[6]  S. Torikai.  Dependency of microdissected nephron segments upon oxidative phosphorylation and exogenous substrates: a relationship between tubular anatomical location in the kidney and metabolic activity. , 1989, Clinical science.

[7]  S. Schmaltz,et al.  Adenosine triphosphate turnover in humans. Decreased degradation during relative hyperphosphatemia. , 1989, The Journal of clinical investigation.

[8]  H. Endou,et al.  Substrate specificity to maintain cellular ATP along the mouse nephron. , 1988, The American journal of physiology.

[9]  H. Tenenhouse,et al.  Effect of the Hyp mutation and diet-induced hyperparathyroidism on renal parathyroid hormone- and forskolin-stimulated adenosine 3',5'-monophosphate production and brush border membrane phosphate transport. , 1986, Endocrinology.

[10]  A. Doucet,et al.  Renal sodium transport in vitamin D resistant hypophosphatemic rickets. , 1985, Canadian journal of physiology and pharmacology.

[11]  G. Quamme,et al.  Renal handling of phosphate. , 1985, Physiological reviews.

[12]  W. Guder,et al.  Compartmentation of ATP within renal proximal tubular cells. , 1984, Biochimica et biophysica acta.

[13]  H. Tenenhouse.  Investigation of the mechanism for abnormal renal 25-hydroxyvitamin D3-1-hydroxylase activity in the X-linked Hyp mouse. , 1984, Endocrinology.

[14]  S. Gore,et al.  Adenine-derived purines increase adenosine triphosphate (ATP) levels in the luteal cell: evidence that cell levels of ATP may limit the stimulation of adenosine 3',5'-monophosphate accumulation by luteinizing hormone. , 1983, Endocrinology.

[15]  E. Holmes,et al.  Reduced renal cortical ribonucleoside triphosphate pools in three different hypophosphatemic animal models. , 1982, Biochemical and biophysical research communications.

[16]  R. Meyer,et al.  X-linked hypophosphatemic mice are not hypersensitive to parathyroid hormone. , 1982, Endocrinology.

[17]  K. Kurokawa,et al.  Adenylate cyclase and cell cyclic AMP of rat cortical thick ascending limb of Henle. , 1981, Kidney international.

[18]  F. Morel,et al.  Hormone-sensitive adenylate cyclase along the nephron of genetically hypophosphatemic mice. , 1979, Kidney international.

[19]  R. Mcinnes,et al.  Renal handling of phosphate in vivo and in vitro by the X-linked hypophosphatemic male mouse: evidence for a defect in the brush border membrane. , 1978, Kidney international.

[20]  山岡 完次.  Abnormal relationship between serum phosphate concentration and renal 25-hydroxycholecalciferol-1-alpha-hydroxylase activity in X-linked hypophosphatemic mice , 1990 .

[21]  M. Imai,et al.  Intrarenal localization of degradation of atrial natriuretic peptide in isolated glomeruli and cortical nephron segments. , 1988, Life sciences.

[22]  M. Haussler,et al.  Abnormal relationship between serum phosphate concentration and renal 25-hydroxycholecalciferol-1-alpha-hydroxylase activity in X-linked hypophosphatemic mice. , 1986, Mineral and electrolyte metabolism.

[23]  S. Soltoff,et al.  ATP and the regulation of renal cell function. , 1986, Annual review of physiology.

[24]  M. Drezner,et al.  Abnormal parathyroid hormone stimulation of 25-hydroxyvitamin D-1 alpha-hydroxylase activity in the hypophosphatemic mouse. Evidence for a generalized defect of vitamin D metabolism. , 1986, The Journal of clinical investigation.

[25]  R. Meyer,et al.  Increased metabolic rate in X-linked hypophosphatemic mice. , 1986, Endocrinology.

[26]  T. Dousa,et al.  ATP and cAMP system in the in vitro response of microdissected cortical tubules to PTH. , 1985, American Journal of Physiology.

[27]  F. Morel,et al.  Methodology for Enzymatic Studies of Isolated Tubular Segments: Adenylate Cyclase , 1978 .