Helicobacter pylori infection triggers aberrant expression of activation-induced cytidine deaminase in gastric epithelium

[1]  L. Senelick.  It (review) , 2008 .

[2]  K. Kinoshita,et al.  Expression of activation‐induced cytidine deaminase in human hepatocytes during hepatocarcinogenesis , 2007, International journal of cancer.

[3]  M. Karin.  Nuclear factor-κB in cancer development and progression , 2006, Nature.

[4]  J. Blaydes,et al.  Immunoglobulin heavy chain locus events and expression of activation-induced cytidine deaminase in epithelial breast cancer cell lines. , 2006, Cancer research.

[5]  K. Kinoshita,et al.  The Dark Side of Activation-Induced Cytidine Deaminase: Relationship with Leukemia and Beyond , 2006, International journal of hematology.

[6]  H. Seno,et al.  Involvement of cyclooxygenase-2 in gastric mucosal hypertrophy in gastrin transgenic mice. , 2006, American journal of physiology. Gastrointestinal and liver physiology.

[7]  F. Otto,et al.  Gene deregulation in gastric cancer. , 2005, Gene.

[8]  A. Levine,et al.  Eradication of Helicobacter pylori and Resolution of Gastritis in the Gastric Mucosa of IL‐10‐Deficient Mice , 2005, Helicobacter.

[9]  H. Seno,et al.  Host factors are important in determining clinical outcomes of Helicobacter pylori infection , 2005, Journal of Gastroenterology.

[10]  L. Pasqualucci,et al.  Expression of the AID protein in normal and neoplastic B cells. , 2004, Blood.

[11]  John Bertin,et al.  Nod1 responds to peptidoglycan delivered by the Helicobacter pylori cag pathogenicity island , 2004, Nature Immunology.

[12]  Ash A. Alizadeh,et al.  AID is expressed in germinal center B-cell-like and activated B-cell-like diffuse large-cell lymphomas and is not correlated with intraclonal heterogeneity , 2004, Leukemia.

[13]  F. Alt,et al.  Induction of Activation-induced Cytidine Deaminase Gene Expression by Il-4 and Cd40 Ligation Is Dependent on Stat6 and Nfkb , 2022 .

[14]  H. Goto.  Helicobacter pylori and gastric diseases. , 2003, Nagoya journal of medical science.

[15]  N. Chiorazzi,et al.  Activation-induced cytidine deaminase in chronic lymphocytic leukemia B cells: expression as multiple forms in a dynamic, variably sized fraction of the clone. , 2003, Blood.

[16]  G. Jenkins,et al.  Detection of p53 mutations in precancerous gastric tissue , 2003, British Journal of Cancer.

[17]  A. Fischer,et al.  AID mutant analyses indicate requirement for class-switch-specific cofactors , 2003, Nature Immunology.

[18]  H. Marusawa,et al.  Role of a novel oncogenic protein, gankyrin, in hepatocyte proliferation , 2003, Journal of Gastroenterology.

[19]  Hui Zhang,et al.  The cytidine deaminase CEM15 induces hypermutation in newly synthesized HIV-1 DNA , 2003, Nature.

[20]  N. Kakazu,et al.  Constitutive Expression of AID Leads to Tumorigenesis , 2003, The Journal of experimental medicine.

[21]  M. Nussenzweig,et al.  Transcription enhances AID-mediated cytidine deamination by exposing single-stranded DNA on the nontemplate strand , 2003, Nature Immunology.

[22]  W. Klapper,et al.  Expression of activation-induced cytidine deaminase in human B-cell non-Hodgkin lymphomas. , 2003, Blood.

[23]  F. Alt,et al.  Transcription-targeted DNA deamination by the AID antibody diversification enzyme , 2003, Nature.

[24]  J. Wedekind,et al.  Messenger RNA editing in mammals: new members of the APOBEC family seeking roles in the family business. , 2003, Trends in genetics : TIG.

[25]  C. Fenoglio-Preiser,et al.  TP53 and gastric carcinoma: A review , 2003, Human mutation.

[26]  T. Honjo,et al.  De novo protein synthesis is required for the activation-induced cytidine deaminase function in class-switch recombination , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[27]  B. Bertocci,et al.  AID-dependent somatic hypermutation occurs as a DNA single-strand event in the BL2 cell line , 2002, Nature Immunology.

[28]  T. Honjo,et al.  AID Enzyme-Induced Hypermutation in an Actively Transcribed Gene in Fibroblasts , 2002, Science.

[29]  T. Honjo,et al.  Molecular mechanism of class switch recombination: linkage with somatic hypermutation. , 2002, Annual review of immunology.

[30]  M. Blaser,et al.  Helicobacter pylori and gastrointestinal tract adenocarcinomas , 2002, Nature Reviews Cancer.

[31]  K. Okazaki,et al.  Analysis of Cytokines in the Early Development of Gastric Secondary Lymphoid Follicles in Helicobacter pylori-Infected BALB/c Mice with Neonatal Thymectomy , 2001, Infection and Immunity.

[32]  K. Shimotohno,et al.  Regulation of Fas‐Mediated Apoptosis by NF‐κB Activity in Human Hepatocyte Derived Cell Lines , 2001, Microbiology and immunology.

[33]  R. Peek.  Microbes and microbial toxins : Paradigms for microbial-mucosal interactions. IV. Helicobacter pylori strain-specific activation of signal transduction cascades related to gastric inflammation , 2001 .

[34]  T. Honjo,et al.  Specific Expression of Activation-induced Cytidine Deaminase (AID), a Novel Member of the RNA-editing Deaminase Family in Germinal Center B Cells* , 1999, The Journal of Biological Chemistry.

[35]  H. Yaegashi,et al.  Application of tyramide signal amplification system to immunohistochemistry: A potent method to localize antigens that are not detectable by ordinary method , 1999, Pathology international.

[36]  M. Upton,et al.  Helicobacter pylori infection activates NF-kappa B in gastric epithelial cells. , 1997, Gastroenterology.

[37]  Mark Borodovsky,et al.  The complete genome sequence of the gastric pathogen Helicobacter pylori , 1997, Nature.

[38]  G. Friedman,et al.  Risk for gastric cancer in people with CagA positive or CagA negative Helicobacter pylori infection. , 1997, Gut.

[39]  S. Falkow,et al.  Helicobacter pylori attachment to gastric cells induces cytoskeletal rearrangements and tyrosine phosphorylation of host cell proteins. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[40]  M. Blaser,et al.  Heightened inflammatory response and cytokine expression in vivo to cagA+ Helicobacter pylori strains. , 1995, Laboratory investigation; a journal of technical methods and pathology.

[41]  M. Blaser,et al.  Infection with Helicobacter pylori strains possessing cagA is associated with an increased risk of developing adenocarcinoma of the stomach. , 1995, Cancer research.

[42]  D. Tompkins,et al.  Systemic and mucosal humoral responses to Helicobacter pylori in gastric cancer. , 1993, Gut.

[43]  B. Vogelstein,et al.  p53 mutations in human cancers. , 1991, Science.