Involvement of NK-1 and NK-2 tachykinin receptor mechanisms in jaw muscle activity reflexly evoked by inflammatory irritant application to the rat temporomandibular joint
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[1] B. Sessle,et al. Involvement of NMDA receptor mechanisms in jaw electromyographic activity and plasma extravasation induced by inflammatory irritant application to temporomandibular joint region of rats , 1996, Pain.
[2] J. Besson,et al. Physiological contributions of neurokinin 1 receptor activation, and interactions with NMDA receptors, to inflammatory-evoked spinal c-Fos expression. , 1996, Journal of neurophysiology.
[3] B. Kerdelhué,et al. Substance P and neurokinin A variations throughout the rat estrous cycle; comparison with ovariectomized and male rats: II. Trigeminal nucleus and cervical spinal cord , 1996, Journal of Neuroscience Research.
[4] H. Vernon,et al. Opioid involvement in electromyographic (EMG) responses induced by injection of inflammatory irritant into deep neck tissues. , 1996, Somatosensory & motor research.
[5] C. Maggi,et al. Peptide-Based Tachykinin NK2 Receptor Antagonists , 1995 .
[6] Q. Ma,et al. Involvement of neurokinin receptors in the induction but not the maintenance of mechanical allodynia in rat flexor motoneurones. , 1995, The Journal of physiology.
[7] James W. Hu,et al. Distribution of fos‐like immunoreactivity in the caudal brainstem of the rat following noxious chemical stimulation of the temporomandibular joint , 1995, The Journal of comparative neurology.
[8] R. Hargreaves,et al. Comparison of the effects of sumatriptan and the NK1 antagonist CP-99,994 on plasma extravasation in dura mater and c-fos mRNA expression in trigeminal nucleus caudalis of rats , 1995, Neuropharmacology.
[9] C. Maggi,et al. Peptide‐based tachykinin NK2 receptor antagonists , 1995, Medicinal research reviews.
[10] L. Luo,et al. The effects of pretreatment with tachykinin antagonists and galanin on the development of spinal cord hyperexcitability following sciatic nerve section in the rat , 1995, Neuropeptides.
[11] B. Sessle,et al. Effects of inflammatory irritant application to the rat temporomandibular joint on jaw and neck muscle activity , 1995, Pain.
[12] J. Henry,et al. Antagonism of nociceptive responses of cat spinal dorsal horn neurons in vivo by the NK-1 receptor antagonists CP-96,345 and CP-99,994, but not by CP-96,344 , 1995, Neuroscience.
[13] A. Duggan. Release of neuropeptides in the spinal cord. , 1995, Progress in brain research.
[14] L. Urbán,et al. Modulation of spinal excitability: co-operation between neurokinin and excitatory amino acid neurotransmitters , 1994, Trends in Neurosciences.
[15] J. Fauchère,et al. Receptors and antagonists for substance P and related peptides. , 1994, Pharmacological reviews.
[16] R. Morris,et al. Postnatal development of nitric oxide synthase type 1 expression in the lumbar spinal cord of the rat: a comparison with the induction of c-fos in response to peripheral application of mustard oil , 1994, Neuroscience Letters.
[17] B. Sessle,et al. Administration of opiate antagonist naloxone induces recurrence of increased jaw muscle activities related to inflammatory irritant application to rat temporomandibular joint region. , 1994, Journal of Neurophysiology.
[18] L. Urbán,et al. Injury-induced plasticity of spinal reflex activity: NK1 neurokinin receptor activation and enhanced A- and C-fiber mediated responses in the rat spinal cord in vitro , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[19] S. Fleetwood-Walker,et al. The effects of neurokinin receptor antagonists on mustard oil-evoked activation of rat dorsal horn neurons , 1993, Neuropeptides.
[20] B. Sessle,et al. Excitatory effects on neck and jaw muscle activity of inflammatory irritant applied to cervical paraspinal tissues , 1993, Pain.
[21] L. Urbán,et al. Contribution of NK1 and NK2 receptor activation to high threshold afferent fibre evoked ventral root responses in the rat spinal cord in vitro , 1993, Brain Research.
[22] A. Ganong,et al. Pharmacology of CP-99,994; a nonpeptide antagonist of the tachykinin neurokinin-1 receptor. , 1993, The Journal of pharmacology and experimental therapeutics.
[23] B. Sessle,et al. Differential effects of cutaneous and deep application of inflammatory irritant on mechanoreceptive field properties of trigeminal brain stem nociceptive neurons. , 1993, Journal of neurophysiology.
[24] H. Schaible,et al. Afferent and spinal mechanisms of joint pain , 1993, Pain.
[25] S. Fleetwood-Walker,et al. Evidence for a role of tachykinin NK2 receptors in mediating brief nociceptive inputs to rat dorsal horn (laminae III-V) neurons. , 1993, European journal of pharmacology.
[26] U. Otten,et al. Nerve growth factor (NGF) regulates tachykinin gene expression and biosynthesis in rat sensory neurons during early postnatal development , 1993, Neuropeptides.
[27] K. Yoshioka,et al. Neurotransmitter functions of mammalian tachykinins. , 1993, Physiological reviews.
[28] J. Howbert,et al. Use of non-peptide tachykinin receptor antagonists to substantiate the involvement of NK1 and NK2 receptors in a spinal nociceptive reflex in the rat. , 1993, European journal of pharmacology.
[29] T. Tanaka,et al. Distribution of Substance P and Calcitonin Gene-related Peptide-like lmmunoreactive Nerve Fibers in the Rat Temporomandibular Joint , 1993, Journal of dental research.
[30] C. Maggi,et al. Tachykinin receptors and tachykinin receptor antagonists. , 1993, Journal of autonomic pharmacology.
[31] W. Maixner,et al. Activation of kappa opioid receptors by U50488H and morphine enhances the release of substance P from rat trigeminal nucleus slices. , 1993, The Journal of pharmacology and experimental therapeutics.
[32] J. Henry. Substance P and inflammatory pain: potential of substance P antagonists as analgesics. , 1993, Agents and actions. Supplements.
[33] R. Minshall,et al. Lack of potent antinociceptive activity by substance P antagonist CP-96,345 in the rat spinal cord. , 1993, Life sciences.
[34] Z. Wiesenfeld‐Hallin,et al. Intrathecal CP-96,345 blocks reflex facilitation induced in rats by substance P and C-fiber-conditioning stimulation. , 1992, European journal of pharmacology.
[35] D. Haas,et al. Development of an orofacial model of acute inflammation in the rat. , 1992, Archives of oral biology.
[36] M. Bakke,et al. Craniomandibular disorders and masticatory muscle function. , 1992, Scandinavian journal of dental research.
[37] C. Maggi,et al. On the role of NK-2 tachykinin receptors in the mediation of spinal reflex excitability in the rat , 1991, Neuroscience.
[38] P. Hansson,et al. Concentrations of neuropeptides substance P, neurokinin A, calcitonin gene-related peptide, neuropeptide Y and vasoactive intestinal polypeptide in synovial fluid of the human temporomandibular joint. A correlation with symptoms, signs and arthroscopic findings. , 1991, International journal of oral and maxillofacial surgery.
[39] H. Handwerker,et al. Discharge patterns of human C-fibers induced by itching and burning stimuli. , 1991, Journal of neurophysiology.
[40] C. Maggi,et al. In vivo evidence for tachykininergic transmission using a new NK-2 receptor-selective antagonist, MEN 10,376. , 1991, The Journal of pharmacology and experimental therapeutics.
[41] B. Sessle,et al. Mechanisms of pain arising from articular tissues. , 1991, Canadian journal of physiology and pharmacology.
[42] Clifford J. Woolf,et al. The induction and maintenance of central sensitization is dependent on N-methyl-d-aspartic acid receptor activation; implications for the treatment of post-injury pain hypersensitivity states , 1991, Pain.
[43] P. Wall,et al. Relative effectiveness of C primary afferent fibers of different origins in evoking a prolonged facilitation of the flexor reflex in the rat , 1986, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[44] A. Calin. Pain and inflammation , 1981, PAIN.
[45] L. Iversen,et al. Opiate analgesics inhibit substance P release from rat trigeminal nucleus , 1977, Nature.