Ozone-induced lung injury and sterile inflammation. Role of toll-like receptor 4.

[1]  S. Garantziotis,et al.  Hyaluronan Signaling during Ozone-Induced Lung Injury Requires TLR4, MyD88, and TIRAP , 2011, PloS one.

[2]  G. Natoli,et al.  Transcriptional regulation of macrophage polarization: enabling diversity with identity , 2011, Nature Reviews Immunology.

[3]  S. Su,et al.  The essential roles of Toll-like receptor signaling pathways in sterile inflammatory diseases. , 2011, International immunopharmacology.

[4]  Frances E. Lennon,et al.  Role of hyaluronan and hyaluronan-binding proteins in lung pathobiology. , 2011, American journal of physiology. Lung cellular and molecular physiology.

[5]  S. Kleeberger,et al.  Identification of Candidate Genes Downstream of TLR4 Signaling after Ozone Exposure in Mice: A Role for Heat-Shock Protein 70 , 2011, Environmental health perspectives.

[6]  P. Kingma,et al.  Surfactant protein D deficiency increases lung injury during endotoxemia. , 2011, American journal of respiratory cell and molecular biology.

[7]  Debra L Laskin,et al.  Macrophages and tissue injury: agents of defense or destruction? , 2011, Annual review of pharmacology and toxicology.

[8]  Grace Y Chen,et al.  Sterile inflammation: sensing and reacting to damage , 2010, Nature Reviews Immunology.

[9]  S. Qureshi,et al.  The role of toll-like receptors in acute and chronic lung inflammation , 2010, Journal of Inflammation.

[10]  E. Abraham,et al.  Toll-like receptors 2 and 4: initiators of non-septic inflammation in critical care medicine? , 2010, Intensive Care Medicine.

[11]  P. Noble,et al.  Regulation of Non‐Infectious Lung Injury, Inflammation, and Repair by the Extracellular Matrix Glycosaminoglycan Hyaluronan , 2010, Anatomical record.

[12]  Y. Li,et al.  Cellular and Molecular Characterization of Ozone-Induced Pulmonary Inflammation in the Cynomolgus Monkey , 2010, Inflammation.

[13]  A. Gow,et al.  Pulmonary effects of inhaled diesel exhaust in aged mice. , 2009, Toxicology and applied pharmacology.

[14]  G. Puzo,et al.  Surfactant protein D inhibits mite‐induced alveolar macrophage and dendritic cell activations through TLR signalling and DC‐SIGN expression , 2009, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[15]  S. Bischoff,et al.  Toll‐like receptor 4 is involved in the development of fructose‐induced hepatic steatosis in mice , 2009, Hepatology.

[16]  Motoko Takahashi,et al.  Pulmonary Surfactant Protein D Inhibits Lipopolysaccharide (LPS)-induced Inflammatory Cell Responses by Altering LPS Binding to Its Receptors* , 2008, Journal of Biological Chemistry.

[17]  Motoko Takahashi,et al.  Pulmonary surfactant protein D binds MD-2 through the carbohydrate recognition domain. , 2008, Biochemistry.

[18]  A. Gow,et al.  S-Nitrosylation of Surfactant Protein-D Controls Inflammatory Function , 2008, PLoS biology.

[19]  T. Griffin,et al.  Oxidative Stress and Covalent Modification of Protein with Bioactive Aldehydes* , 2008, Journal of Biological Chemistry.

[20]  K. Rock,et al.  How dying cells alert the immune system to danger , 2008, Nature Reviews Immunology.

[21]  D. Laskin,et al.  Regulation of caveolin-1 expression, nitric oxide production and tissue injury by tumor necrosis factor-alpha following ozone inhalation. , 2008, Toxicology and applied pharmacology.

[22]  M. Rosengart,et al.  HMGB1 release induced by liver ischemia involves Toll-like receptor 4–dependent reactive oxygen species production and calcium-mediated signaling , 2007, The Journal of experimental medicine.

[23]  Debra L Laskin,et al.  Acute endotoxemia is associated with upregulation of lipocalin 24p3/Lcn2 in lung and liver. , 2007, Experimental and molecular pathology.

[24]  I. Adcock,et al.  Role of TLR2, TLR4, and MyD88 in murine ozone-induced airway hyperresponsiveness and neutrophilia. , 2007, Journal of applied physiology.

[25]  T. Miyasho,et al.  Role of Toll-like receptor 4 in hyperoxia-induced lung inflammation in mice , 2007, Inflammation Research.

[26]  D. Morgan,et al.  Signal transduction pathways of tumor necrosis factor--mediated lung injury induced by ozone in mice. , 2007, American journal of respiratory and critical care medicine.

[27]  O. Hurtado,et al.  Toll-Like Receptor 4 Is Involved in Brain Damage and Inflammation After Experimental Stroke , 2007, Circulation.

[28]  H. Schulz,et al.  Inhalation of ultrafine carbon particles triggers biphasic pro-inflammatory response in the mouse lung , 2006, European Respiratory Journal.

[29]  Tsuyoshi Saito,et al.  Human pulmonary surfactant protein D binds the extracellular domains of Toll-like receptors 2 and 4 through the carbohydrate recognition domain by a mechanism different from its binding to phosphatidylinositol and lipopolysaccharide. , 2006, Biochemistry.

[30]  M. Salmon,et al.  Susceptibility to ozone-induced airway inflammation is associated with decreased levels of surfactant protein D , 2006, Respiratory research.

[31]  Toby Lawrence,et al.  IKKα limits macrophage NF-κB activation and contributes to the resolution of inflammation , 2005, Nature.

[32]  Hitomi Sano,et al.  The lung collectins, SP-A and SP-D, modulate pulmonary innate immunity. , 2005, Molecular immunology.

[33]  Olaf Holz,et al.  Association of Tumor Necrosis Factor-α Polymorphisms and Ozone-induced Change in Lung Function , 2005 .

[34]  W. M. Foster,et al.  The role of Toll-like receptor 4 in environmental airway injury in mice. , 2004, American journal of respiratory and critical care medicine.

[35]  Shizuo Akira,et al.  Toll-like receptor signalling , 2004, Nature Reviews Immunology.

[36]  Ian S Mudway,et al.  An investigation of inhaled ozone dose and the magnitude of airway inflammation in healthy adults. , 2004, American journal of respiratory and critical care medicine.

[37]  D. Laskin,et al.  Ozone-induced production of nitric oxide and TNF-α and tissue injury are dependent on NF-κB p50 , 2004 .

[38]  W. M. Foster,et al.  Ozone-induced acute pulmonary injury in inbred mouse strains. , 2004, American journal of respiratory cell and molecular biology.

[39]  J. Oyama,et al.  Reduced Myocardial Ischemia-Reperfusion Injury in Toll-Like Receptor 4-Deficient Mice , 2003, Circulation.

[40]  P. Matzinger.  The Danger Model: A Renewed Sense of Self , 2002, Science.

[41]  J. Whitsett,et al.  The pulmonary collectins, SP-A and SP-D, orchestrate innate immunity in the lung. , 2002, The Journal of clinical investigation.

[42]  C. Irvin,et al.  Strain dependence of airway hyperresponsiveness reflects differences in eosinophil localization in the lung. , 2001, American journal of physiology. Lung cellular and molecular physiology.

[43]  Hye-Youn Cho,et al.  Ozone-induced lung inflammation and hyperreactivity are mediated via tumor necrosis factor-alpha receptors. , 2001, American journal of physiology. Lung cellular and molecular physiology.

[44]  Hye-Youn Cho,et al.  Toll-like receptor 4 mediates ozone-induced murine lung hyperpermeability via inducible nitric oxide synthase. , 2001, American journal of physiology. Lung cellular and molecular physiology.

[45]  Heinz Fehrenbach,et al.  Alveolar epithelial type II cell: defender of the alveolus revisited , 2001, Respiratory research.

[46]  S. Reddy,et al.  Genetic susceptibility to ozone-induced lung hyperpermeability: role of toll-like receptor 4. , 2000, American journal of respiratory cell and molecular biology.

[47]  F. Kelly,et al.  Ozone and the lung: a sensitive issue. , 2000, Molecular aspects of medicine.

[48]  P. Barnes,et al.  Late response to allergen is associated with increased concentrations of tumor necrosis factor-alpha and IL-5 in induced sputum. , 1997, The Journal of allergy and clinical immunology.

[49]  M. Morandi,et al.  4-hydroxy-2-nonenal-protein adducts and apoptosis in murine lung cells after acute ozone exposure. , 1996, Toxicology and applied pharmacology.

[50]  M. Burdick,et al.  Hyaluronan (HA) fragments induce chemokine gene expression in alveolar macrophages. The role of HA size and CD44. , 1996, The Journal of clinical investigation.

[51]  R. Hamilton,et al.  4-Hydroxynonenal mimics ozone-induced modulation of macrophage function ex vivo. , 1996, American journal of respiratory cell and molecular biology.

[52]  R. Hamilton,et al.  4-Hydroxynonenal-induced cell death in murine alveolar macrophages. , 1996, Toxicology and applied pharmacology.

[53]  K. Chung,et al.  Expression of inducible nitric oxide synthase mRNA in Brown Norway rats exposed to ozone: effect of dexamethasone. , 1995, European journal of pharmacology.

[54]  D. Laskin,et al.  Inhibition of macrophages with gadolinium chloride abrogates ozone-induced pulmonary injury and inflammatory mediator production. , 1995, American journal of respiratory cell and molecular biology.

[55]  S. Giri,et al.  Effects of Pretreatment with Anti-Inflammatory Drugs on Ozone-Induced Lung Damage in Rats , 1975, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[56]  Y. Kuwahara,et al.  Oxidative stress induced lipocalin 2 gene expression: addressing its expression under the harmful conditions. , 2007, Journal of radiation research.

[57]  T. Weaver,et al.  Function of surfactant proteins B and C. , 2001, Annual review of physiology.

[58]  E. Schadt,et al.  Quantitative trait loci controlling allergen-induced airway hyperresponsiveness in inbred mice. , 2000, American journal of respiratory cell and molecular biology.

[59]  D. Bhalla.  Ozone-induced lung inflammation and mucosal barrier disruption: toxicology, mechanisms, and implications. , 1999, Journal of toxicology and environmental health. Part B, Critical reviews.

[60]  R. Hamilton,et al.  Potential involvement of 4-hydroxynonenal in the response of human lung cells to ozone. , 1998, The American journal of physiology.

[61]  W. Pryor,et al.  Aldehydes, hydrogen peroxide, and organic radicals as mediators of ozone toxicity. , 1991, Free radical biology & medicine.