Rolipram plus Sivelestat inhibits bone marrow-derived leukocytic lung recruitment after cardiopulmonary bypass in a primate model

[1]  N. Ageyama,et al.  Cardiopulmonary bypass induces recruitment of bone marrow-derived leukocytes to the lungs in monkeys. , 2014, The Annals of thoracic surgery.

[2]  O. Shigeta,et al.  Sivelestat reduces inflammatory mediators and preserves neutrophil deformability during simulated extracorporeal circulation. , 2005, The Annals of thoracic surgery.

[3]  Makiko Sato,et al.  Phosphodiesterase type 4 inhibitor rolipram inhibits activation of monocytes during extracorporeal circulation. , 2005, The Journal of thoracic and cardiovascular surgery.

[4]  A. Seiyama,et al.  Protective effects of a selective neutrophil elastase inhibitor (sivelestat) on lipopolysaccharide-induced acute dysfunction of the pulmonary microcirculation , 2005, Critical care medicine.

[5]  G. Bernard,et al.  Neutrophil elastase inhibition in acute lung injury: Results of the STRIVE study , 2004, Critical care medicine.

[6]  Y. Yoshimura,et al.  ONO-6818, a novel, potent neutrophil elastase inhibitor, reduces inflammatory mediators during simulated extracorporeal circulation. , 2003, The Annals of thoracic surgery.

[7]  J. Hogg,et al.  A novel method to quantify the turnover and release of monocytes from the bone marrow using the thymidine analog 5'-bromo-2'-deoxyuridine. , 2003, American journal of physiology. Cell physiology.

[8]  N. Ishikawa,et al.  The effects of a specific neutrophil elastase inhibitor (ONO‐5046) in pulmonary ischemia—reperfusion injury , 2003 .

[9]  N. Ishikawa,et al.  The effects of a specific neutrophil elastase inhibitor (ONO-5046) in pulmonary ischemia–reperfusion injury , 2003, Transplant international : official journal of the European Society for Organ Transplantation.

[10]  J. Hogg,et al.  Interleukin-6 induces demargination of intravascular neutrophils and shortens their transit in marrow. , 2000, American journal of physiology. Heart and circulatory physiology.

[11]  M. Sayeed Exuberant Ca(2+) Signaling in Neutrophils: A Cause for Concern. , 2000, News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society.

[12]  A. Usui,et al.  Protective effects of ONO-5046*Na, a specific neutrophil elastase inhibitor, on postperfusion lung injury. , 1999, The Annals of thoracic surgery.

[13]  L. Edmunds Inflammatory response to cardiopulmonary bypass. , 1998, The Annals of thoracic surgery.

[14]  T. Torphy Phosphodiesterase isozymes: molecular targets for novel antiasthma agents. , 1998, American journal of respiratory and critical care medicine.

[15]  J. Hogg,et al.  Polymorphonuclear Leukocytes Released from the Bone Marrow Preferentially Sequester in Lung Microvessels , 1997, Microcirculation.

[16]  T. Evans,et al.  Effects of inhibition of complement activation using recombinant soluble complement receptor 1 on neutrophil CD11b/CD18 and L-selectin expression and release of interleukin-8 and elastase in simulated cardiopulmonary bypass. , 1996, The Journal of thoracic and cardiovascular surgery.

[17]  F. Deist,et al.  Hypothermia during cardiopulmonary bypass delays but does not prevent neutrophil-endothelial cell adhesion. A clinical study. , 1995, Circulation.

[18]  T. Carlos,et al.  Leukocyte-endothelial adhesion molecules. , 1994, Blood.

[19]  T. Hurst,et al.  Neutrophil-mediated acute lung injury after extracorporeal perfusion. , 1994, The Journal of thoracic and cardiovascular surgery.

[20]  M. Elliott,et al.  Interleukin-8 release and neutrophil degranulation after pediatric cardiopulmonary bypass. , 1993, The Journal of thoracic and cardiovascular surgery.

[21]  C. Doerschuk The role of CD18-mediated adhesion in neutrophil sequestration induced by infusion of activated plasma in rabbits. , 1992, American journal of respiratory cell and molecular biology.

[22]  J. Mathew,et al.  Cardiopulmonary bypass induces leukocyte-platelet adhesion. , 1992, Blood.

[23]  S. Weiss Tissue destruction by neutrophils. , 1989, The New England journal of medicine.

[24]  S. Honjo The Japanese Tsukuba Primate Center for Medical Science (TPC): An Outline , 1985, Journal of medical primatology.

[25]  Y. Hiramatsu,et al.  Phosphodiesterase type 4 inhibition of activated polymorphonuclear leukocytes in a simulated extracorporeal circulation model. , 2003, The Journal of thoracic and cardiovascular surgery.

[26]  S. Honjo On Guiding Principles for Animal Experiments Using Nonhuman Primates , 1986 .