CpG island methylator phenotype in colorectal cancer.
暂无分享,去创建一个
J. Herman | S. Baylin | N. Ahuja | M. Toyota | M. Ohe-Toyota | J. Issa | S B Baylin | J G Herman | J P Issa | M Toyota | N Ahuja | M Ohe-Toyota | J. Issa | Mutsumi Ohe-Toyota
[1] S. Baylin,et al. Identification of differentially methylated sequences in colorectal cancer by methylated CpG island amplification. , 1999, Cancer research.
[2] S. Baylin,et al. Aging and DNA methylation in colorectal mucosa and cancer. , 1998, Cancer research.
[3] S N Thibodeau,et al. Hypermethylation of the hMLH1 promoter in colon cancer with microsatellite instability. , 1998, Cancer research.
[4] S. Baylin,et al. Concordant methylation of the ER and N33 genes in glioblastoma multiforme , 1998, Oncogene.
[5] J. Herman,et al. Incidence and functional consequences of hMLH1 promoter hypermethylation in colorectal carcinoma. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[6] R. White,et al. Tumor Suppressing Pathways , 1998, Cell.
[7] J. Herman,et al. Alterations in DNA methylation: a fundamental aspect of neoplasia. , 1998, Advances in cancer research.
[8] J. Herman,et al. Association between CpG island methylation and microsatellite instability in colorectal cancer. , 1997, Cancer research.
[9] P. Laird,et al. COBRA: a sensitive and quantitative DNA methylation assay. , 1997, Nucleic acids research.
[10] M. Slattery,et al. Microsatellite instability in colorectal adenomas. , 1997, Gastroenterology.
[11] L. Harris,et al. Cytoplasmic sequestration of an O6-methylguanine-DNA methyltransferase enhancer binding protein in DNA repair-deficient human cells. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[12] T. Bestor,et al. Formation of methylation patterns in the mammalian genome. , 1997, Mutation research.
[13] M. Loda,et al. Methylation of the hMLH1 promoter correlates with lack of expression of hMLH1 in sporadic colon tumors and mismatch repair-defective human tumor cell lines. , 1997, Cancer research.
[14] J C Reed,et al. Somatic Frameshift Mutations in the BAX Gene in Colon Cancers of the Microsatellite Mutator Phenotype , 1997, Science.
[15] A. de la Chapelle,et al. Mutations predisposing to hereditary nonpolyposis colorectal cancer. , 1997, Advances in cancer research.
[16] K. Kinzler,et al. Lessons from Hereditary Colorectal Cancer , 1996, Cell.
[17] S. Baylin,et al. Methylation of the estrogen receptor CpG island in lung tumors is related to the specific type of carcinogen exposure. , 1996, Cancer research.
[18] T. Iwama,et al. Molecular nature of colon tumors in hereditary nonpolyposis colon cancer, familial polyposis, and sporadic colon cancer. , 1996, Gastroenterology.
[19] S. Baylin,et al. De novo methylation of CpG island sequences in human fibroblasts overexpressing DNA (cytosine-5-)-methyltransferase , 1996, Molecular and cellular biology.
[20] B. Andrews,et al. Binding to the yeast SwI4,6-dependent cell cycle box, CACGAAA, is cell cycle regulated in vivo. , 1996, Nucleic acids research.
[21] J. Herman,et al. Inactivation of the CDKN2/p16/MTS1 gene is frequently associated with aberrant DNA methylation in all common human cancers. , 1995, Cancer research.
[22] K. Kinzler,et al. Inactivation of the type II TGF-beta receptor in colon cancer cells with microsatellite instability. , 1995, Science.
[23] R. Weinberg,et al. Suppression of intestinal neoplasia by DNA hypomethylation , 1995, Cell.
[24] A. Kamb,et al. Cell-cycle regulators and cancer. , 1995, Trends in genetics : TIG.
[25] K. A. Walker,et al. Epigenetic Gene Inactivation Induced by a Cis-acting Methylation Center (*) , 1995, The Journal of Biological Chemistry.
[26] A. Bird,et al. Sp1 sites in the mouse aprt gene promoter are required to prevent methylation of the CpG island. , 1994, Genes & development.
[27] Z. Siegfried,et al. Spl elements protect a CpG island from de novo methylation , 1994, Nature.
[28] K. Dameron,et al. Control of angiogenesis in fibroblasts by p53 regulation of thrombospondin-1. , 1994, Science.
[29] P. Jones,et al. Ubiquitous and tenacious methylation of the CpG site in codon 248 of the p53 gene may explain its frequent appearance as a mutational hot spot in human cancer , 1994, Molecular and cellular biology.
[30] S. Baylin,et al. Methylation of the oestrogen receptor CpG island links ageing and neoplasia in human colon , 1994, Nature Genetics.
[31] M. Turker,et al. A cis-acting element accounts for a conserved methylation pattern upstream of the mouse adenine phosphoribosyltransferase gene. , 1993, The Journal of biological chemistry.
[32] A. Bird,et al. High levels of De Novo methylation and altered chromatin structure at CpG islands in cell lines , 1990, Cell.
[33] P. Holt,et al. Colonic proliferation is increased in senescent rats. , 1988, Gastroenterology.
[34] L. Roncucci,et al. The influence of age on colonic epithelial cell proliferation , 1988, Cancer.
[35] M. Lipkin. Biomarkers of increased susceptibility to gastrointestinal cancer: new application to studies of cancer prevention in human subjects. , 1988, Cancer research.
[36] M. Frommer,et al. CpG islands in vertebrate genomes. , 1987, Journal of molecular biology.
[37] A. Bird. CpG-rich islands and the function of DNA methylation , 1986, Nature.