Understanding arsenic carcinogenicity by the use of animal models.
暂无分享,去创建一个
S. Fukushima | A. Kinoshita | H. Wanibuchi | K. Morimura | M. Wei | E. Salim | Jun Shen | Kaoru Yoshida | G. Endo | K. Kuroda | Anna Kinoshita
[1] M. Vahter,et al. Reduction and binding of arsenate in marmoset monkeys , 1985, Archives of Toxicology.
[2] S. Fukushima,et al. Induction of glutathione S-transferase placental form positive foci in liver and epithelial hyperplasia in urinary bladder, but no tumor development in male Fischer 344 rats treated with monomethylarsonic acid for 104 weeks. , 2003, Toxicology and applied pharmacology.
[3] S. Fukushima,et al. Liver tumorigenicity of trimethylarsine oxide in male Fischer 344 rats--association with oxidative DNA damage and enhanced cell proliferation. , 2003, Carcinogenesis.
[4] S. Okada,et al. Oxidative DNA damage following exposure to dimethylarsinous iodide: the formation of cis-thymine glycol. , 2003, Toxicology letters.
[5] S. Fukushima,et al. Carcinogenicity of dimethylarsinic acid in p53 heterozygous knockout and wild-type C57BL/6J mice. , 2003, Carcinogenesis.
[6] K. Kitchin,et al. Oxidative stress as a possible mode of action for arsenic carcinogenesis. , 2003, Toxicology letters.
[7] J. Ward,et al. Transplacental carcinogenicity of inorganic arsenic in the drinking water: induction of hepatic, ovarian, pulmonary, and adrenal tumors in mice. , 2003, Toxicology and applied pharmacology.
[8] S. Fukushima,et al. Carcinogenicity of dimethylarsinic acid in male F344 rats and genetic alterations in induced urinary bladder tumors. , 2002, Carcinogenesis.
[9] S. Fukushima,et al. Promoting effects of monomethylarsonic acid, dimethylarsinic acid and trimethylarsine oxide on induction of rat liver preneoplastic glutathione S‐transferase placental form positive foci: A possible reactive oxygen species mechanism , 2002, International journal of cancer.
[10] Michael F Hughes,et al. Arsenic toxicity and potential mechanisms of action. , 2002, Toxicology letters.
[11] W. Cullen,et al. Plasmid DNA damage caused by methylated arsenicals, ascorbic acid and human liver ferritin. , 2002, Toxicology letters.
[12] S. Fukushima,et al. Lack of promoting effect due to oral administration of dimethylarsinic acid on rat lung carcinogenesis initiated with N-bis(2-hydroxypropyl)nitrosamine. , 2002, Cancer letters.
[13] M. Vahter,et al. Role of metabolism in arsenic toxicity. , 2001, Pharmacology & toxicology.
[14] K. Kitchin. Recent advances in arsenic carcinogenesis: modes of action, animal model systems, and methylated arsenic metabolites. , 2001, Toxicology and applied pharmacology.
[15] E. Kenyon,et al. A concise review of the toxicity and carcinogenicity of dimethylarsinic acid. , 2001, Toxicology.
[16] W. Cullen,et al. Methylated trivalent arsenic species are genotoxic. , 2001, Chemical research in toxicology.
[17] S. X. Liu,et al. Induction of oxyradicals by arsenic: implication for mechanism of genotoxicity. , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[18] W. Cullen,et al. Arsenic species that cause release of iron from ferritin and generation of activated oxygen. , 2000, Archives of biochemistry and biophysics.
[19] J. Sawicki,et al. K6/ODC transgenic mice as a sensitive model for carcinogen identification. , 2000, Toxicology letters.
[20] D. Morgan,et al. Occurrence of monomethylarsonous acid in urine of humans exposed to inorganic arsenic. , 2000, Chemical research in toxicology.
[21] S. Fukushima,et al. Promotion of Skin Carcinogenesis by Dimethylarsinic Acid in Keratin (K6)/ODC Transgenic Mice , 2000, Japanese journal of cancer research : Gann.
[22] Wei Li,et al. Urinary bladder carcinogenicity of dimethylarsinic acid in male F344 rats. , 1999, Carcinogenesis.
[23] Wei Li,et al. Promotion of NCI-Black-Reiter male rat bladder carcinogenesis by dimethylarsinic acid an organic arsenic compound. , 1998, Cancer letters.
[24] S. X. Liu,et al. Mutagenicity of arsenic in mammalian cells: role of reactive oxygen species. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[25] S. Okada,et al. Dimethylarsinic acid, a main metabolite of inorganic arsenics, has tumorigenicity and progression effects in the pulmonary tumors of A/J mice. , 1998, Cancer letters.
[26] G. Endo,et al. Aneuploidy induced by dimethylarsinic acid in mouse bone marrow cells. , 1998, Mutation research.
[27] S. Fukushima,et al. Promotion of Rat Hepatocarcinogenesis by Dimethylarsinic Acid: Association with Elevated Ornithine Decarboxylase Activity and Formation of 8‐Hydroxydeoxyguanosine in the Liver , 1997, Japanese journal of cancer research : Gann.
[28] H. Aposhian,et al. Enzymatic methylation of arsenic compounds: IV. In vitro and in vivo deficiency of the methylation of arsenite and monomethylarsonic acid in the guinea pig. , 1997, Mutation research.
[29] Y. Hsueh,et al. Arsenic methylation capacity, body retention, and null genotypes of glutathione S-transferase M1 and T1 among current arsenic-exposed residents in Taiwan. , 1997, Mutation research.
[30] K. Harrington-Brock,et al. Relative genotoxic potency of arsenic and its methylated metabolites. , 1997, Mutation research.
[31] G. Endo,et al. Metabolites of Arsenic Induced Tetraploids and Mitotic Arrest in Cultured Cells , 1997, Archives of environmental contamination and toxicology.
[32] H. Aposhian. Enzymatic methylation of arsenic species and other new approaches to arsenic toxicity. , 1997, Annual review of pharmacology and toxicology.
[33] S. Fukushima,et al. Promoting effects of dimethylarsinic acid on N-butyl-N-(4-hydroxybutyl)nitrosamine-induced urinary bladder carcinogenesis in rats. , 1996, Carcinogenesis.
[34] T. Kaise,et al. Induction of chromosomal aberrations in cultured human fibroblasts by inorganic and organic arsenic compounds and the different roles of glutathione in such induction. , 1996, Mutation research.
[35] S. Fukushima,et al. Cancer induction by an organic arsenic compound, dimethylarsinic acid (cacodylic acid), in F344/DuCrj rats after pretreatment with five carcinogens. , 1995, Cancer research.
[36] M. Vahter. Species differences in the metabolism of arsenic compounds , 1994 .
[37] T. S. Wang,et al. Active oxygen species are involved in the induction of micronuclei by arsenite in XRS-5 cells. , 1994, Mutagenesis.
[38] U. Thorgeirsson,et al. Tumor incidence in a chemical carcinogenesis study of nonhuman primates. , 1994, Regulatory toxicology and pharmacology : RTP.
[39] S. Horiguchi,et al. Dimethylarsenic acid induces tetraploids in Chinese hamster cells , 1992, Bulletin of environmental contamination and toxicology.
[40] S. Okada,et al. Cellular response to oxidative damage in lung induced by the administration of dimethylarsinic acid, a major metabolite of inorganic arsenics, in mice. , 1991, Toxicology and applied pharmacology.
[41] S. Okada,et al. Induction of DNA damage by dimethylarsine, a metabolite of inorganic arsenics, is for the major part likely due to its peroxyl radical. , 1990, Biochemical and biophysical research communications.
[42] S. Okada,et al. Mutagenicity of dimethylated metabolites of inorganic arsenics. , 1989, Chemical & pharmaceutical bulletin.
[43] D. Bleyl,et al. IARC Monographs on the Evaluation of Carcinogenic Risks to Humans. Overall Evaluations of Carcinogenicity: An Updating of IARC Monographs vol. 1 to 42. Supplement 7. 440 Seiten. International Agency for Research on Cancer, Lyon 1987. Preis: 65, – s.Fr , 1989 .
[44] M. Vahter,et al. Biotransformation of trivalent and pentavalent inorganic arsenic in mice and rats. , 1981, Environmental research.
[45] J. Brouwer,et al. Pathologic changes in rats and dogs from two-year feeding of sodium arsenite or sodium arsenate. , 1967, Toxicology and applied pharmacology.