The many masks of focal segmental glomerulosclerosis.

[1]  C. Alpers,et al.  Human mesangial cells are resistant to productive infection by multiple strains of human immunodeficiency virus types 1 and 2. , 1992, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[2]  R. Colvin,et al.  Focal and segmental glomerulosclerosis and porteinuria associated with unilateral renal agenesis. , 1982, Laboratory investigation; a journal of technical methods and pathology.

[3]  E. Lewis,et al.  The Prognosis of Focal Segmental Glomerular Sclerosis of Adulthood , 1986, Medicine.

[4]  R. Habib.  Focal glomerular sclerosis , 1973 .

[5]  C. Chantler,et al.  The long-term prognosis of patients with focal segmental glomerulosclerosis. , 1978, Clinical nephrology.

[6]  A. Fogo,et al.  Serial micropuncture analysis of glomerular function in two rat models of glomerular sclerosis. , 1988, The Journal of clinical investigation.

[7]  M. Kashgarian,et al.  Steroid-dependent nephrotic syndrome in children: histopathology and relapses after cyclophosphamide treatment. , 1981, Kidney international.

[8]  M. Schoeneman,et al.  The natural history of focal segmental glomerulosclerosis with and without mesangial hypercellularity in children. , 1978, Clinical nephrology.

[9]  M. Sporn,et al.  Transactivation of the transforming growth factor beta 1 (TGF-beta 1) gene by human T lymphotropic virus type 1 tax: a potential mechanism for the increased production of TGF-beta 1 in adult T cell leukemia , 1990, The Journal of experimental medicine.

[10]  A. Fogo,et al.  Bradykinin causes selective efferent arteriolar dilation during angiotensin I converting enzyme inhibition. , 1993, Kidney international.

[11]  V. D’Agati,et al.  Pathology of HIV-associated nephropathy: a detailed morphologic and comparative study. , 1989, Kidney international.

[12]  G. Becker,et al.  Morphometric aspects of reflux nephropathy. , 1987, Kidney international.

[13]  E. Lewis,et al.  Focal segmental glomerular sclerosis: the cellular lesion. , 1985, Kidney international.

[14]  J. Diamond,et al.  Irreversible tubulointerstitial damage associated with chronic aminonucleoside nephrosis. Amelioration by angiotensin I converting enzyme inhibition. , 1990, The American journal of pathology.

[15]  C. Alpers,et al.  Glomerular cells, extracellular matrix accumulation, and the development of glomerulosclerosis in the remnant kidney model. , 1992, Laboratory investigation; a journal of technical methods and pathology.

[16]  C. Alpers,et al.  Tubulointerstitial disease in glomerulonephritis. Potential role of osteopontin (uropontin). , 1994, The American journal of pathology.

[17]  G. Wolf,et al.  Angiotensin II stimulates the proliferation and biosynthesis of type I collagen in cultured murine mesangial cells. , 1992, The American journal of pathology.

[18]  A. Koyama,et al.  A glomerular permeability factor produced by human T cell hybridomas. , 1991, Kidney international.

[19]  A. Saifer,et al.  Conditioning factors influencing evolution of experimental glomerulonephritis in rabbits. , 1959, The American journal of physiology.

[20]  H. Gröne,et al.  Induction of glomerulosclerosis by dietary lipids. A functional and morphologic study in the rat. , 1989, Laboratory investigation; a journal of technical methods and pathology.

[21]  T. Hostetter,et al.  Adverse effects of growth in the glomerular microcirculation. , 1990, The American journal of physiology.

[22]  T. Gilbert,et al.  Long-Term Effects of Mild Oligonephronia Induced In Utero by Gentamicin in the Rat , 1991, Pediatric Research.

[23]  Caulfield Jp.  Alterations in the distribution of Alcian blue-staining fibrillar anionic sites in the glomerular basement membrane in aminonucleoside nephrosis. , 1979 .

[24]  M. Adhikari,et al.  OUTCOME OF CHILDHOOD MINIMAL-CHANGE DISEASE , 1989, The Lancet.

[25]  W. Border,et al.  From serum sickness to cytokines: advances in understanding the molecular pathogenesis of kidney disease. , 1993, Laboratory investigation; a journal of technical methods and pathology.

[26]  D. Haffner,et al.  The acute effect of growth hormone on GFR is obliterated in chronic renal failure. , 1989, Clinical nephrology.

[27]  R. Hené,et al.  The glomerular tip lesion: A distinct entity or not? , 1988, The Journal of pathology.

[28]  N. Yoshikawa,et al.  Focal segmental glomerulosclerosis with and without nephrotic syndrome in children. , 1986, The Journal of pediatrics.

[29]  S. Feriozzi,et al.  Glomerular hypertrophy and chronic renal failure in focal segmental glomerulosclerosis. , 1994, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[30]  A. Eddy.  Interstitial nephritis induced by protein-overload proteinuria. , 1989, The American journal of pathology.

[31]  V. Pardo,et al.  The Epidemiology of Human Immunodeficiency Virus Associated Nephropathy , 1991 .

[32]  E. Kohaut,et al.  TREATMENT OF GROWTH FAILURE IN CHILDREN AFTER RENAL TRANSPLANTATION1 , 1993, Transplantation.

[33]  A. Howie,et al.  The glomerular tip lesion: A previously undescribed type of segmental glomerular abnormality , 1984, The Journal of pathology.

[34]  R. Shalhoub.  Pathogenesis of lipoid nephrosis: a disorder of T-cell function. , 1974, Lancet.

[35]  Jennifer K. Adams,et al.  A relationship between proteinuria and acute tubulointerstitial disease in rats with experimental nephrotic syndrome. , 1991, The American journal of pathology.

[36]  M. Gubler,et al.  The significance of pure diffuse mesangial proliferation in idiopathic nephrotic syndrome. , 1978, Clinical nephrology.

[37]  R. Hogg,et al.  Focal segmental glomerulosclerosis in children with idiopathic nephrotic syndrome. A report of the Southwest Pediatric Nephrology Study Group1 , 1985 .

[38]  A. Remuzzi,et al.  Three-dimensional morphometric analysis of segmental glomerulosclerosis in the rat. , 1990, Kidney international.

[39]  V. Savin,et al.  Mechanisms of proteinuria in noninflammatory glomerular diseases. , 1993, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[40]  P. Kimmel,et al.  Effect of race on expression of acquired immunodeficiency syndrome-associated nephropathy. , 1991, Archives of internal medicine.

[41]  B. Julian,et al.  Focal sclerosis of hypertrophied glomeruli in solitary functioning kidneys of humans. , 1985, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[42]  W. Hsueh.  The role of the extrarenal renin‐angiotensin system , 1992 .

[43]  Rich Ar.  A hitherto undescribed vulnerability of the juxtamedullary glomeruli in lipoid nephrosis. , 1957 .

[44]  B. Brenner,et al.  Control of glomerular hypertension limits glomerular injury in rats with reduced renal mass. , 1985, The Journal of clinical investigation.

[45]  B. Kasiske,et al.  Treatment of hyperlipidemia reduces glomerular injury in obese Zucker rats. , 1988, Kidney international.

[46]  M. Kretzler,et al.  The role of podocytes in the development of glomerular sclerosis. , 1994, Kidney international. Supplement.

[47]  A. Fogo,et al.  Glomerular hypertrophy in minimal change disease predicts subsequent progression to focal glomerular sclerosis. , 1990, Kidney international.

[48]  R. Zatz,et al.  Glomerular hypertrophy and progressive glomerulopathy. Is there a definite pathogenetic correlation? , 1994, Kidney international. Supplement.

[49]  B. Chavers,et al.  20 years or more of follow-up of living kidney donors , 1992, The Lancet.

[50]  A. Fogo,et al.  Glomerular hemodynamic changes vs. hypertrophy in experimental glomerular sclerosis. , 1989, Kidney international.

[51]  Y. Kanwar,et al.  Unaltered anionic sites of glomerular basement membrane in aminonucleoside nephrosis. , 1984, Kidney international.

[52]  M. Kitamura,et al.  The effect of selective growth hormone defect in the progression of glomerulosclerosis. , 1994, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[53]  A. Fogo,et al.  Effects of antihypertensive drugs on glomerular morphology. , 1989, Kidney international.

[54]  D. Senitzer,et al.  Possible genetic predisposition to idiopathic focal segmental glomerulosclerosis. , 1988, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[55]  T. Meyer,et al.  Glomerular hypertrophy and epithelial cell injury modulate progressive glomerulosclerosis in the rat. , 1989, Laboratory investigation; a journal of technical methods and pathology.

[56]  N. Yoshikawa,et al.  Twenty-seven children with focal segmental glomerulosclerosis: correlation between the segmental location of the glomerular lesions and prognosis. , 1984, Clinical nephrology.

[57]  R. Srinivasan,et al.  Predicting the response to cytotoxic therapy for childhood nephrotic syndrome: superiority of response to corticosteroid therapy over histopathologic patterns. , 1988, The Journal of pediatrics.

[58]  A. Fogo,et al.  Evidence for the central role of glomerular growth promoters in the development of sclerosis. , 1989, Seminars in nephrology.

[59]  S. Mauer,et al.  Recurrence of focal segmental sclerosis in children following renal transplantation. , 1986, Kidney international. Supplement.

[60]  A. Tejani,et al.  Recurrence of focal segmental glomerulosclerosis posttransplantation: a special report of the North American Pediatric Renal Transplant Cooperative Study. , 1992, Journal of the American Society of Nephrology : JASN.

[61]  W. Hsueh,et al.  Angiotensin II causes mesangial cell hypertrophy. , 1993, Hypertension.

[62]  M. Karnovsky,et al.  Effects of the aminonucleoside of puromycin on glomerular epithelial cells in vitro. , 1985, The American journal of pathology.

[63]  R. Falk,et al.  Collapsing glomerulopathy: a clinically and pathologically distinct variant of focal segmental glomerulosclerosis. , 1994, Kidney international.

[64]  T. Meyer,et al.  Glomerular hypertrophy aggravates epithelial cell injury in nephrotic rats. , 1990, The Journal of clinical investigation.

[65]  B. Kasiske,et al.  Renal disease in patients with massive obesity. , 1986, Archives of internal medicine.

[66]  J. Jennette,et al.  Glomerulomegaly and focal segmental glomerulosclerosis associated with obesity and sleep-apnea syndrome. , 1987, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[67]  W. Keane.  Lipids and the kidney. , 1994, Kidney international.

[68]  R. Verani.  Obesity-associated focal segmental glomerulosclerosis: pathological features of the lesion and relationship with cardiomegaly and hyperlipidemia. , 1992, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[69]  S. Tomlanovich,et al.  Recurrent focal glomerulosclerosis: natural history and response to therapy. , 1992, The American journal of medicine.

[70]  H. D. de Wardener,et al.  Relationship between renal function and histological changes found in renal-biopsy specimens from patients with persistent glomerular nephritis. , 1968, Lancet.

[71]  L. Klareskog,et al.  Transmission of infectious agents by inadequately sterilized ENT instruments. , 1989 .

[72]  C. Ponticelli,et al.  The impact of prolonged immunosuppression on the outcome of idiopathic focal-segmental glomerulosclerosis with nephrotic syndrome in adults. A collaborative retrospective study. , 1991, Clinical nephrology.

[73]  G. Maschio,et al.  The solitary kidney: a risky situation for progressive renal damage? , 1991, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[74]  P. Kimmel,et al.  Viral DNA in microdissected renal biopsy tissue from HIV infected patients with nephrotic syndrome. , 1993, Kidney international.

[75]  G. Zauli,et al.  tat protein stimulates production of transforming growth factor-beta 1 by marrow macrophages: a potential mechanism for human immunodeficiency virus-1-induced hematopoietic suppression. , 1992, Blood.

[76]  J. Velosa,et al.  Significance of proteinuria on the outcome of renal function in patients with focal segmental glomerulosclerosis. , 1983, Mayo Clinic proceedings.

[77]  B. Brenner,et al.  Therapeutic advantage of converting enzyme inhibitors in arresting progressive renal disease associated with systemic hypertension in the rat. , 1986, The Journal of clinical investigation.

[78]  F. Raafat,et al.  The clinical significance of the glomerular location of segmental lesions in focal segmental glomerulosclerosis. , 1990, Clinical nephrology.

[79]  D. de Zeeuw,et al.  Additive antiproteinuric effect of angiotensin-converting enzyme inhibition and non-steroidal anti-inflammatory drug therapy: a clue to the mechanism of action. , 1991, Clinical science.

[80]  K. Kitamoto,et al.  Long-term antiproteinuric effect of nipradilol , a beta-blocker with ONO-2, in patients with mesangial proliferative GN A 1-year-follow-up , 1993 .

[81]  L. Resnick,et al.  HIV infects glomerular endothelial and mesangial but not epithelial cells in vitro. , 1992, Kidney international.

[82]  A. Novick,et al.  Long-term follow-up after partial removal of a solitary kidney. , 1991, The New England journal of medicine.

[83]  E. Orringer,et al.  Prevalence and pathologic features of sickle cell nephropathy and response to inhibition of angiotensin-converting enzyme. , 1992, The New England journal of medicine.

[84]  P. Russo,et al.  Focal segmental glomerulosclerosis (FSG) 20 years later. From toddler to grown up. , 1993, Clinical nephrology.

[85]  M. Karnovsky,et al.  An ultrastructural study of the mechanisms of proteinuria in aminonucleoside nephrosis. , 1975, Kidney international.

[86]  K. Nath,et al.  Tubulointerstitial changes as a major determinant in the progression of renal damage. , 1992, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[87]  Douglas C. Folts,et al.  A lipid chemotactic factor associated with proteinuria and interstitial nephritis induced by protein overload , 1991 .

[88]  R. Ettenger,et al.  Treatment of recurrent focal segmental glomerulosclerosis with high-dose cyclosporine A and plasmapheresis. , 1993, Transplantation proceedings.

[89]  J. Silver,et al.  HIV-associated nephropathy in transgenic mice expressing HIV-1 genes. , 1991, Virology.

[90]  C. Merlet-Benichou,et al.  Retard de croissance intra-utérin et déficit en néphrons , 1993 .

[91]  D. Senitzer,et al.  HLA associations in heroin-associated nephropathy. , 1988, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[92]  A. Howie.  Changes at the glomerular tip: A feature of membranous nephropathy and other disorders associated with proteinuria , 1986, The Journal of pathology.

[93]  V. Pollak,et al.  Nephrotic syndrome, progressive irreversible renal failure, and glomerular "collapse": a new clinicopathologic entity? , 1986, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[94]  A. Fogo,et al.  Internephron heterogeneity of growth factors and sclerosis. , 1994, Experimental nephrology.

[95]  Michael Af,et al.  Experimental model of focal sclerosis. I. Relationship to protein excretion in aminonucleoside nephrosis. , 1977 .

[96]  T. Meyer,et al.  Forty-five year follow-up after uninephrectomy. , 1993, Kidney international.

[97]  Y. Pei,et al.  Evidence suggesting under-treatment in adults with idiopathic focal segmental glomerulosclerosis. Regional Glomerulonephritis Registry Study. , 1987, The American journal of medicine.

[98]  P. Bennett,et al.  Large glomerular size in Pima Indians: lack of change with diabetic nephropathy. , 1992, Journal of the American Society of Nephrology : JASN.

[99]  T. Meyer,et al.  Increased single-nephron protein excretion after renal ablation in nephrotic rats. , 1988, The American journal of physiology.

[100]  A. Fogo,et al.  Angiotensin converting enzyme inhibitor modulates glomerular function and structure by distinct mechanisms. , 1994, Kidney international.

[101]  E. Lewis,et al.  Recurrent nephrotic syndrome in renal allografts. , 1988, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[102]  A. Fogo,et al.  Serial micropuncture analysis of single nephron function in subtotal renal ablation. , 1988, Kidney international.

[103]  H. Rennke,et al.  Pathogenesis and significance of nonprimary focal and segmental glomerulosclerosis. , 1989, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[104]  K. Yoshioka,et al.  Transforming growth factor-beta protein and mRNA in glomeruli in normal and diseased human kidneys. , 1993, Laboratory investigation; a journal of technical methods and pathology.

[105]  P. Klotman,et al.  Progressive glomerulosclerosis and enhanced renal accumulation of basement membrane components in mice transgenic for human immunodeficiency virus type 1 genes. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[106]  L. Ruilope,et al.  Nephrotic proteinuria without hypoalbuminemia: clinical characteristics and response to angiotensin-converting enzyme inhibition. , 1991, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[107]  D. Adu,et al.  The glomerular tip lesion: a steroid responsive nephrotic syndrome. , 1987, Clinical nephrology.

[108]  B. Brenner,et al.  Hyperfiltration in remnant nephrons: a potentially adverse response to renal ablation. , 1981, The American journal of physiology.

[109]  G. Striker,et al.  Glomerular lesions in mice transgenic for growth hormone and insulinlike growth factor-I. I. Relationship between increased glomerular size and mesangial sclerosis. , 1990, The American journal of pathology.

[110]  P. Shapshak,et al.  Demonstration of human immunodeficiency virus in renal epithelium in HIV-associated nephropathy. , 1989, Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc.

[111]  G. Remuzzi,et al.  Adriamycin-induced glomerulosclerosis in the rat. , 1986, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[112]  L. Jarett,et al.  Chemical Pathology , 1907, Buffalo Medical Journal.