Predominant role of reduced beta-cell sensitivity to glucose over insulin resistance in impaired glucose tolerance
暂无分享,去创建一个
M. Matsuda | Y. Miyazaki | R. DeFronzo | E. Ferrannini | A. Gastaldelli | A. Natali | A. Mari | M. Pettiti | Ralph A. DeFronzo | Andrea Natali | Eleuterio Ferrannini | A. Gastaldelli | Andrea Mari
[1] P. Zimmet,et al. The relationship of insulin response to a glucose stimulus over a wide range of glucose tolerance , 1978, Diabetologia.
[2] B. Ahrén,et al. Failure to adequately adapt reduced insulin sensitivity with increased insulin secretion in women with impaired glucose tolerance , 1996, Diabetologia.
[3] M. Walker,et al. Peripheral and hepatic insulin sensitivity in subjects with impaired glucose tolerance , 1995, Diabetologia.
[4] G. Reaven,et al. Relationship between glucose tolerance, insulin secretion, and insulin action in non-obese individuals with varying degrees of glucose tolerance , 2004, Diabetologia.
[5] S. Kahn,et al. The relative contributions of insulin resistance and beta-cell dysfunction to the pathophysiology of Type 2 diabetes , 2003, Diabetologia.
[6] Andrea Mari,et al. Meal and oral glucose tests for assessment of beta -cell function: modeling analysis in normal subjects. , 2002, American journal of physiology. Endocrinology and metabolism.
[7] C. Cobelli,et al. Insulin secretory responses to rising and falling glucose concentrations are delayed in subjects with impaired glucose tolerance , 2002, Diabetologia.
[8] Andrea Tura,et al. Assessing insulin secretion by modeling in multiple-meal tests: role of potentiation. , 2002, Diabetes.
[9] T. W. Haeften,et al. Disturbances in β-Cell Function in Impaired Fasting Glycemia , 2002 .
[10] T. Jenssen,et al. Disturbances in beta-cell function in impaired fasting glycemia. , 2002, Diabetes.
[11] F Tosi,et al. Assessment of β‐cell function during the oral glucose tolerance test by a minimal model of insulin secretion , 2001, European journal of clinical investigation.
[12] K. Polonsky. Evolution of β-cell dysfunction in impaired glucose tolerance and diabetes , 2009, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.
[13] M. Wolfe,et al. Effect of GIP and GLP-1 antagonists on insulin release in the rat. , 1999, American journal of physiology. Endocrinology and metabolism.
[14] E. Ferrannini. Insulin resistance versus insulin deficiency in non-insulin-dependent diabetes mellitus: problems and prospects. , 1998, Endocrine reviews.
[15] J. Gerich,et al. The genetic basis of type 2 diabetes mellitus: impaired insulin secretion versus impaired insulin sensitivity. , 1998, Endocrine reviews.
[16] D W Neal,et al. Basal hepatic glucose production is regulated by the portal vein insulin concentration. , 1998, Diabetes.
[17] E. Ferrannini,et al. Insulin resistance and hypersecretion in obesity. European Group for the Study of Insulin Resistance (EGIR). , 1997, The Journal of clinical investigation.
[18] T. Usdin,et al. Postprandial stimulation of insulin release by glucose-dependent insulinotropic polypeptide (GIP). Effect of a specific glucose-dependent insulinotropic polypeptide receptor antagonist in the rat. , 1996, The Journal of clinical investigation.
[19] H. Yki-Järvinen,et al. Insulin Secretion and Insulin Sensitivity in People with Impaired Glucose Tolerance , 1996, Diabetic medicine : a journal of the British Diabetic Association.
[20] J. Holst,et al. Gastric emptying, glucose responses, and insulin secretion after a liquid test meal: effects of exogenous glucagon-like peptide-1 (GLP-1)-(7-36) amide in type 2 (noninsulin-dependent) diabetic patients. , 1996, The Journal of clinical endocrinology and metabolism.
[21] C Cobelli,et al. Estimation of β-Cell Sensitivity From Intravenous Glucose Tolerance Test C-Peptide Data: Knowledge of the Kinetics Avoids Errors in Modeling the Secretion , 1995, Diabetes.
[22] R. Bergman,et al. Quantification of the Relationship Between Insulin Sensitivity and β-Cell Function in Human Subjects: Evidence for a Hyperbolic Function , 1993, Diabetes.
[23] J. Lancaster,et al. Total Body Fat Content and Fat Topography Are Associated Differently With In Vivo Glucose Metabolism in Nonobese and Obese Nondiabetic Women , 1992, Diabetes.
[24] Jeppe Sturis,et al. Estimation of Insulin Secretion Rates from C-Peptide Levels: Comparison of Individual and Standard Kinetic Parameters for C-Peptide Clearance , 1992, Diabetes.
[25] D. Kelley,et al. Role of reduced suppression of glucose production and diminished early insulin release in impaired glucose tolerance. , 1992, The New England journal of medicine.
[26] L. Groop,et al. Early metabolic defects in persons at increased risk for non-insulin-dependent diabetes mellitus. , 1989, The New England journal of medicine.
[27] L. Groop,et al. Glucose and free fatty acid metabolism in non-insulin-dependent diabetes mellitus. Evidence for multiple sites of insulin resistance. , 1989, The Journal of clinical investigation.
[28] R. DeFronzo,et al. Fasting hyperglycemia in non-insulin-dependent diabetes mellitus: contributions of excessive hepatic glucose production and impaired tissue glucose uptake. , 1989, Metabolism: clinical and experimental.
[29] R. DeFronzo,et al. The disposal of an oral glucose load in patients with non-insulin-dependent diabetes. , 1988, Metabolism: clinical and experimental.
[30] C Cobelli,et al. The kinetics of insulin in man. II. Role of the liver. , 1987, Diabetes/metabolism reviews.
[31] M. Carlquist,et al. Potentiation of glucose-induced insulin secretion in the perfused rat pancreas by porcine GIP (gastric inhibitory polypeptide), bovine GIP, and bovine GIP(1-39). , 1986, Acta physiologica Scandinavica.
[32] C Cobelli,et al. Effect of insulin on the distribution and disposition of glucose in man. , 1985, The Journal of clinical investigation.
[33] R. DeFronzo,et al. The Disposal of an Oral Glucose Load in Healthy Subjects: A Quantitative Study , 1985, Diabetes.
[34] K. Berntorp,et al. High and low insulin responders: relations to oral glucose tolerance, insulin secretion and physical fitness. , 2009, Acta medica Scandinavica.
[35] B V Howard,et al. Relationships between insulin secretion, insulin action, and fasting plasma glucose concentration in nondiabetic and noninsulin-dependent diabetic subjects. , 1984, The Journal of clinical investigation.
[36] L. Saccá,et al. Differential roles of splanchnic and peripheral tissues in the pathogenesis of impaired glucose tolerance. , 1984, The Journal of clinical investigation.
[37] E. Cerasi. Potentiation of insulin release by glucose in man. , 1975, Acta endocrinologica.
[38] E. Cerasi,et al. Quantitative Study on the Potentiating Effect of Arginine on Glucose-induced Insulin Response in Healthy, Prediabetic, and Diabetic Subjects , 1974, Diabetes.
[39] Vojtech Ličko. Threshold secretory mechanism: A model of derivative element in biological control , 1973 .
[40] E. Cerasi,et al. Further studies on healthy subjects with low and high insulin response to glucose infusion. , 1967, Acta endocrinologica.
[41] R. Steele,et al. INFLUENCES OF GLUCOSE LOADING AND OF INJECTED INSULIN ON HEPATIC GLUCOSE OUTPUT * , 1959, Annals of the New York Academy of Sciences.
[42] R. Steele,et al. Measurement of size and turnover rate of body glucose pool by the isotope dilution method. , 1956, The American journal of physiology.