How one TSH receptor antibody induces thyrocyte proliferation while another induces apoptosis.

[1]  G. McKhann,et al.  Cyclophilin D deficiency rescues Aβ-impaired PKA/CREB signaling and alleviates synaptic degeneration. , 2014, Biochimica et biophysica acta.

[2]  T. Jørgensen,et al.  Serum selenium is low in newly diagnosed Graves’ disease: a population‐based study , 2013, Clinical endocrinology.

[3]  N. Chandel,et al.  Physiological roles of mitochondrial reactive oxygen species. , 2012, Molecular cell.

[4]  R. Latif,et al.  Delineating the autoimmune mechanisms in Graves’ disease , 2012, Immunologic research.

[5]  M. Žarković.  The Role of Oxidative Stress on the Pathogenesis of Graves' Disease , 2011, Journal of thyroid research.

[6]  P. Pinton,et al.  Mitochondria-Ros Crosstalk in the Control of Cell Death and Aging , 2011, Journal of signal transduction.

[7]  C. Steegborn,et al.  A Phosphodiesterase 2A Isoform Localized to Mitochondria Regulates Respiration* , 2011, The Journal of Biological Chemistry.

[8]  M. Weissel,et al.  Selenium and the course of mild Graves' orbitopathy. , 2011, The New England journal of medicine.

[9]  C. Gourlay,et al.  cAMP/PKA signaling balances respiratory activity with mitochondria dependent apoptosis via transcriptional regulation , 2010, BMC Cell Biology.

[10]  T. Ando,et al.  Neutral antibodies to the TSH receptor are present in Graves' disease and regulate selective signaling cascades. , 2010, Endocrinology.

[11]  R. Paus,et al.  Thyrotropin powers human mitochondria , 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[12]  R. Acín-Pérez,et al.  Cyclic AMP produced inside mitochondria regulates oxidative phosphorylation. , 2009, Cell metabolism.

[13]  R. Latif,et al.  Characterization of thyrotropin receptor antibody-induced signaling cascades. , 2009, Endocrinology.

[14]  A. Bossowski,et al.  Identification of apoptotic proteins in thyroid gland from patients with Graves' disease and Hashimoto's thyroiditis , 2008, Autoimmunity.

[15]  M. Madesh,et al.  Simultaneous detection of apoptosis and mitochondrial superoxide production in live cells by flow cytometry and confocal microscopy , 2007, Nature Protocols.

[16]  J. Dumont,et al.  Duox expression and related H2O2 measurement in mouse thyroid: onset in embryonic development and regulation by TSH in adult. , 2007, The Journal of endocrinology.

[17]  O. Konuk,et al.  Apoptosis in Orbital Fibroadipose Tissue and its Association with Clinical Features in Graves’ Ophthalmopathy , 2007, Ocular immunology and inflammation.

[18]  Barry Halliwell,et al.  Oxidative stress and cancer: have we moved forward? , 2007, The Biochemical journal.

[19]  P. Venditti,et al.  Thyroid hormone-induced oxidative stress , 2006, Cellular and Molecular Life Sciences CMLS.

[20]  D. Ginty,et al.  Mitochondrial Cyclic AMP Response Element-binding Protein (CREB) Mediates Mitochondrial Gene Expression and Neuronal Survival* , 2005, Journal of Biological Chemistry.

[21]  E. Avvedimento,et al.  cAMP-PKA signaling to the mitochondria: protein scaffolds, mRNA and phosphatases. , 2005, Cellular signalling.

[22]  J. Blendy,et al.  Activating Transcription Factor 1 and CREB Are Important for Cell Survival during Early Mouse Development , 2002, Molecular and Cellular Biology.

[23]  G. Stassi,et al.  Autoimmune thyroid disease: new models of cell death in autoimmunity , 2002, Nature Reviews Immunology.

[24]  Terry J. Smith,et al.  Igs from Patients with Graves’ Disease Induce the Expression of T Cell Chemoattractants in Their Fibroblasts1 , 2002, The Journal of Immunology.

[25]  M. Sallese,et al.  Thyrotropin activates mitogen-activated protein kinase pathway in FRTL-5 by a cAMP-dependent protein kinase A-independent mechanism. , 2001, Molecular pharmacology.

[26]  P. Kopp,et al.  A dominant negative CREB (cAMP response element-binding protein) isoform inhibits thyrocyte growth, thyroid-specific gene expression, differentiation, and function. , 2000, Molecular endocrinology.

[27]  A. Nagasaka,et al.  Changes in Free Radical Scavengers and Lipid Peroxide in Thyroid Glands of Various Thyroid Disorders , 1997, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.

[28]  T. Davies,et al.  Human autoantibodies to the thyrotropin receptor: recognition of linear, folded, and glycosylated recombinant extracellular domain. , 1995, The Journal of clinical endocrinology and metabolism.

[29]  B. Prabhakar,et al.  Heterogeneity in cellular and antibody responses against thyrotropin receptor in patients with Graves' disease detected using synthetic peptides. , 1993, Journal of autoimmunity.

[30]  K. Burman,et al.  Graves' IgG recognizes linear epitopes in the human thyrotropin receptor. , 1992, Biochemical and biophysical research communications.

[31]  M. Ohmori,et al.  Heterogeneous responses of recombinant human thyrotropin receptor to immunoglobulins from patients with Graves' disease. , 1992, Biochemical and biophysical research communications.

[32]  B. Prabhakar,et al.  Prokaryotic expression of the thyrotropin receptor and identification of an immunogenic region of the protein using synthetic peptides. , 1991, Biochemical and biophysical research communications.

[33]  H. Imura,et al.  A synthetic oligopeptide derived from human thyrotropin receptor sequence binds to Graves' immunoglobulin and inhibits thyroid stimulating antibody activity but lacks interactions with TSH. , 1991, Biochemical and biophysical research communications.

[34]  H. Coon,et al.  Culture of hormone-dependent functional epithelial cells from rat thyroids. , 1980, Proceedings of the National Academy of Sciences of the United States of America.