The relationship between extracellular calcium concentrations and the induction of hippocampal long-term potentiation

[1]  G. Lynch,et al.  Long‐term potentiation and depression of synaptic responses in the rat hippocampus: localization and frequency dependency. , 1978, The Journal of physiology.

[2]  G. Lynch,et al.  Heterosynaptic depression: a postsynaptic correlate of long-term potentiation , 1977, Nature.

[3]  P. Andersen,et al.  Specific long-lasting potentiation of synaptic transmission in hippocampal slices , 1977, Nature.

[4]  T. Teyler,et al.  Reversible loss of hippocampal long term potentiation following electroconvulsive seizures , 1976, Nature.

[5]  T. Teyler,et al.  Long-term and short-term plasticity in the CA1, CA3, and dentate regions of the rat hippocampal slice , 1976, Brain Research.

[6]  P. Schwartzkroin,et al.  Long-lasting facilitation of a synaptic potential following tetanization in thein vitro hippocampal slice , 1975, Brain Research.

[7]  R. Douglas,et al.  Long-term potentiation of the perforant path-granule cell synapse in the rat hippocampus , 1975, Brain Research.

[8]  T. Bliss,et al.  Long‐lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant path , 1973, The Journal of physiology.

[9]  C. D. Richards,et al.  Calcium, magnesium and the electrical activity of guinea‐pig olfactory cortex in vitro , 1970, The Journal of physiology.

[10]  J. Rosenthal.  Post‐tetanic potentiation at the neuromuscular junction of the frog , 1969, The Journal of physiology.

[11]  P. F. Baker.  Transport and metabolism of calcium ions in nerve. , 1972, Progress in biophysics and molecular biology.

[12]  D. Weinreich.  Ionic mechanism of post‐tetanic potentiation at the neuromuscular junction of the frog , 1971, The Journal of physiology.