MicroRNA-155 Deficiency Promotes Nephrin Acetylation and Attenuates Renal Damage in Hyperglycemia-Induced Nephropathy

[1]  Yu-Ting Huang,et al.  MicroRNA-29a promotion of nephrin acetylation ameliorates hyperglycemia-induced podocyte dysfunction. , 2014, Journal of the American Society of Nephrology : JASN.

[2]  Feng Tian,et al.  Rheumatoid Arthritis-Associated MicroRNA-155 Targets SOCS1 and Upregulates TNF-α and IL-1β in PBMCs , 2013, International journal of molecular sciences.

[3]  Haiping Yang,et al.  The Role of Th17/IL-17 in the Pathogenesis of Primary Nephrotic Syndrome in Children , 2013, Kidney and Blood Pressure Research.

[4]  C. Alpers,et al.  Reversibility of structural and functional damage in a model of advanced diabetic nephropathy. , 2013, Journal of the American Society of Nephrology : JASN.

[5]  K. Connelly,et al.  eNOS deficiency predisposes podocytes to injury in diabetes. , 2012, Journal of the American Society of Nephrology : JASN.

[6]  Huanhuan Li,et al.  MicroRNA-155 Modulates Treg and Th17 Cells Differentiation and Th17 Cell Function by Targeting SOCS1 , 2012, PloS one.

[7]  S. Hattori,et al.  SIRPα interacts with nephrin at the podocyte slit diaphragm , 2012, The FEBS journal.

[8]  T. Curran,et al.  Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease. , 2012, The Journal of clinical investigation.

[9]  H. Weiner,et al.  Silencing MicroRNA-155 Ameliorates Experimental Autoimmune Encephalomyelitis , 2011, The Journal of Immunology.

[10]  G. Levy-Clarke,et al.  Suppressor of cytokine signaling-1 (SOCS1) inhibits lymphocyte recruitment into the retina and protects SOCS1 transgenic rats and mice from ocular inflammation. , 2011, Investigative ophthalmology & visual science.

[11]  Xia Li,et al.  Th17/Treg imbalance in adult patients with minimal change nephrotic syndrome. , 2011, Clinical immunology.

[12]  M. Hall,et al.  mTORC1 activation in podocytes is a critical step in the development of diabetic nephropathy in mice. , 2011, The Journal of clinical investigation.

[13]  Lilian W. Waiboci,et al.  The kinase inhibitory region of SOCS-1 is sufficient to inhibit T-helper 17 and other immune functions in experimental allergic encephalomyelitis , 2011, Journal of Neuroimmunology.

[14]  Chunmei Wang,et al.  Inducible microRNA-155 Feedback Promotes Type I IFN Signaling in Antiviral Innate Immunity by Targeting Suppressor of Cytokine Signaling 1 , 2010, The Journal of Immunology.

[15]  Ryan M. O’Connell,et al.  MicroRNA-155 promotes autoimmune inflammation by enhancing inflammatory T cell development. , 2010, Immunity.

[16]  Shuai Jiang,et al.  MicroRNA-155 functions as an OncomiR in breast cancer by targeting the suppressor of cytokine signaling 1 gene. , 2010, Cancer research.

[17]  M. Saleem,et al.  Nephrin—signature molecule of the glomerular podocyte? , 2009, The Journal of pathology.

[18]  Michael T. McManus,et al.  LPS induces KH‐type splicing regulatory protein‐dependent processing of microRNA‐155 precursors in macrophages , 2009, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[19]  D. Stolz,et al.  Wnt/beta-catenin signaling promotes podocyte dysfunction and albuminuria. , 2009, Journal of the American Society of Nephrology : JASN.

[20]  P. Scambler,et al.  Nephrin deficiency activates NF-kappaB and promotes glomerular injury. , 2009, Journal of the American Society of Nephrology : JASN.

[21]  Xiaodong Zhao,et al.  The prevalence of Th17 cells and FOXP3 regulate T cells (Treg) in children with primary nephrotic syndrome , 2009, Pediatric Nephrology.

[22]  Hana Lee,et al.  Foxp3-dependent microRNA155 confers competitive fitness to regulatory T cells by targeting SOCS1 protein. , 2009, Immunity.

[23]  N. Rajewsky,et al.  Regulation of the Germinal Center Response by MicroRNA-155 , 2007, Science.

[24]  Anton J. Enright,et al.  Requirement of bic/microRNA-155 for Normal Immune Function , 2007, Science.

[25]  M. Kubo,et al.  Overexpression of Suppressor of Cytokine Signaling-3 in T Cells Exacerbates Acetaminophen-Induced Hepatotoxicity1 , 2007, The Journal of Immunology.

[26]  David Baltimore,et al.  MicroRNA-155 is induced during the macrophage inflammatory response , 2007, Proceedings of the National Academy of Sciences.

[27]  Jianping Jin,et al.  Characterization of susceptibility of inbred mouse strains to diabetic nephropathy. , 2005, Diabetes.

[28]  L. Flowers,et al.  A SOCS-1 peptide mimetic inhibits both constitutive and IL-6 induced activation of STAT3 in prostate cancer cells , 2005, Oncogene.

[29]  Yu-Ting Huang,et al.  Dickkopf-1 promotes hyperglycemia-induced accumulation of mesangial matrix and renal dysfunction. , 2010, Journal of the American Society of Nephrology : JASN.