Sp1 promotes dental pulp stem cell osteoblastic differentiation through regulating noggin.

[1]  M. A. d’Ávila,et al.  Osteogenic potential of human dental pulp stem cells cultured onto poly-ε-caprolactone/poly (rotaxane) scaffolds. , 2019, Dental materials : official publication of the Academy of Dental Materials.

[2]  Yong Xia,et al.  MKK7 transcription positively or negatively regulated by SP1 and KLF5 depends on HDAC4 activity in glioma , 2019, International journal of cancer.

[3]  Huan Liu,et al.  SP1 regulates KLF4 via SP1 binding motif governed by DNA methylation during odontoblastic differentiation of human dental pulp cells , 2019, Journal of cellular biochemistry.

[4]  S. Hashimi.  Exogenous noggin binds the BMP‐2 receptor and induces alkaline phosphatase activity in osteoblasts , 2019, Journal of cellular biochemistry.

[5]  D. Yoon,et al.  Specific protein 1(SP1) regulates the epithelial-mesenchymal transition via lysyl oxidase-like 2(LOXL2) in pancreatic ductal adenocarcinoma , 2019, Scientific Reports.

[6]  R. Serra,et al.  Antagonism of BMP signaling is insufficient to induce fibrous differentiation in primary sclerotome , 2019, Experimental cell research.

[7]  M. Peppa,et al.  Denosumab effects on serum levels of the bone morphogenetic proteins antagonist noggin in patients with transfusion-dependent thalassemia and osteoporosis , 2019, Hematology.

[8]  R. Hortigüela,et al.  Noggin rescues age-related stem cell loss in the brain of senescent mice with neurodegenerative pathology , 2018, Proceedings of the National Academy of Sciences.

[9]  A. Burny,et al.  MiR302c, Sp1, and NFATc2 regulate interleukin‐21 expression in human CD4+CD45RO+ T lymphocytes , 2018, Journal of cellular physiology.

[10]  Ş. Çetinkalp,et al.  The low levels of bone morphogenic protein-4 and its antagonist noggin in type 2 diabetes , 2018, Hormones.

[11]  H. Ryoo,et al.  Fibrous Topography-Potentiated Canonical Wnt Signaling Directs the Odontoblastic Differentiation of Dental Pulp-Derived Stem Cells. , 2018, ACS applied materials & interfaces.

[12]  B. Lee,et al.  Potential Roles of Dental Pulp Stem Cells in Neural Regeneration and Repair , 2018, Stem cells international.

[13]  G. Pizzo,et al.  Dental pulp stem cells for bone tissue engineering: a review of the current literature and a look to the future. , 2018, Regenerative medicine.

[14]  M. Tabrizian,et al.  Efficient in vitro delivery of Noggin siRNA enhances osteoblastogenesis , 2017, Heliyon.

[15]  S. Pyo,et al.  Pneumolysin-induced autophagy contributes to inhibition of osteoblast differentiation through downregulation of Sp1 in human osteosarcoma cells. , 2017, Biochimica et biophysica acta. General subjects.

[16]  Y. Kumazawa,et al.  Human Dental Pulp Cells Differentiate toward Neuronal Cells and Promote Neuroregeneration in Adult Organotypic Hippocampal Slices In Vitro , 2017, International journal of molecular sciences.

[17]  V. Machado,et al.  Dental stem cells: recent progresses in tissue engineering and regenerative medicine , 2017, Annals of medicine.

[18]  Y. Liu,et al.  Pro-inflammatory cytokine-driven PI3K/Akt/Sp1 signalling and H2S production facilitates the pathogenesis of severe acute pancreatitis , 2017, Bioscience reports.

[19]  Zhidai Liu,et al.  The cellular senescence of leukemia-initiating cells from acute lymphoblastic leukemia is postponed by β-Arrestin1 binding with P300-Sp1 to regulate hTERT transcription , 2017, Cell Death and Disease.

[20]  A. Gaharwar,et al.  Nanoengineered Osteoinductive and Elastomeric Scaffolds for Bone Tissue Engineering. , 2017, ACS biomaterials science & engineering.

[21]  K. Shakesheff,et al.  Decellularized bone extracellular matrix and human dental pulp stem cells as a construct for bone regeneration , 2017, Journal of biomaterials science. Polymer edition.

[22]  Rohit K. Sharma,et al.  Self-assembly of aromatic α-amino acids into amyloid inspired nano/micro scaled architects. , 2017, Materials science & engineering. C, Materials for biological applications.

[23]  P. Tylzanowski,et al.  Noggin inactivation affects the number and differentiation potential of muscle progenitor cells in vivo , 2016, Scientific Reports.

[24]  Zhen-bo Feng,et al.  Sp1 cooperates with Sp3 to upregulate MALAT1 expression in human hepatocellular carcinoma. , 2015, Oncology reports.

[25]  S. Imaoka,et al.  Down-regulation of EPHX2 gene transcription by Sp1 under high-glucose conditions. , 2015, The Biochemical journal.

[26]  Nu Zhang,et al.  Transcriptional factor specificity protein 1 (SP1) promotes the proliferation of glioma cells by up-regulating midkine (MDK) , 2015, Molecular biology of the cell.

[27]  Zhixiang Wang,et al.  Noggin suppression decreases BMP‐2‐induced osteogenesis of human bone marrow‐derived mesenchymal stem cells In Vitro , 2012, Journal of cellular biochemistry.

[28]  Ramon Kranaster,et al.  A non-canonical DNA structure is a binding motif for the transcription factor SP1 in vitro , 2011, Nucleic acids research.

[29]  L. Laino,et al.  Human mandible bone defect repair by the grafting of dental pulp stem/progenitor cells and collagen sponge biocomplexes. , 2009, European cells & materials.

[30]  S. Gronthos,et al.  Mesenchymal Stem Cells Derived from Dental Tissues vs. Those from Other Sources: Their Biology and Role in Regenerative Medicine , 2009, Journal of dental research.

[31]  C. Bunker,et al.  Identification and association analysis of single nucleotide polymorphisms in the human noggin (NOG) gene and osteoporosis phenotypes. , 2009, Bone.

[32]  V. Castronovo,et al.  HDAC4 represses p21WAF1/Cip1 expression in human cancer cells through a Sp1-dependent, p53-independent mechanism , 2009, Oncogene.

[33]  E. Coto,et al.  A functional Sp1/Egr1‐tandem repeat polymorphism in the 5‐lipoxygenase gene is not associated with myocardial infarction , 2007, International journal of immunogenetics.

[34]  P H Krebsbach,et al.  Craniofacial Tissue Engineering by Stem Cells , 2006, Journal of dental research.

[35]  X. Xing,et al.  The role of Sp1 in BMP2-up-regulated Erk2 gene expression. , 2002, Biochemical and biophysical research communications.

[36]  S. Gronthos,et al.  Postnatal human dental pulp stem cells (DPSCs) in vitro and in vivo. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[37]  S. Ju,et al.  FasL promoter activation by IL‐2 through SP1 and NFAT but not Egr‐2 and Egr‐3 , 1999, European journal of immunology.

[38]  Frank Grosveld,et al.  Transcription Factor Sp1 Is Essential for Early Embryonic Development but Dispensable for Cell Growth and Differentiation , 1997, Cell.

[39]  Z. Meng,et al.  Acetylated Sp1 inhibits PTEN expression through binding to PTEN core promoter and recruitment of HDAC1 and promotes cancer cell migration and invasion. , 2013, Carcinogenesis.

[40]  G. Schmalz,et al.  Transcription factors TP53 and SP1 and the osteogenic differentiation of dental stem cells. , 2012, Differentiation; research in biological diversity.