Antiphospholipid antibodies (APLA) are immunoglobulins of the lgG and lgM groups with activity against negatively charged phospholipids. Common manifestations of APLA in clude the presence of lupus anticoagulant and anticardiolipin antibodies, and false-positive Venereal Disease Research Laboratory (VORL) and rapid plasma reagin (RPR) tests [1 , 2]. APLA has been associated with an increased frequency of venous and arterial thromboses, recurrent fetal loss, throm bocytopenia, cardiac valve lesions, livedo reticularis and other skin lesions, and a variety of neurologic syndromes [1 - 9). The disease has been reported most frequently in patients with systemic lupus erythematosus (SLE) and lupuslike dis ease , but also in other clinical entities , including infections, neoplasms, hematologic disorders, AIDS , primary immuno deficiency, collagenosis, and following treatment with certain drugs (especially in those associated with drug-induced SLE) [1 ' 2). Recently, a new subgroup of patients who have idiopathic APLA has been described. The diagnostic criteria of this primary antiphospholipid syndrome (PAPS) include a history of venous and{or arterial thrombotic occlusions, recurrent fetal loss or thrombocytopenia, and the presence of lupus anticoagulant, anticardiolipin antibodies, or both, in the ab sence of SLE or any other cause of APLA [1 , 2). To make the diagnosis, at least one clinical feature and one positive laboratory test should be present on two different occasions separated by a 2-month interval [2]. We report the MR findings in a case of PAPS clinically resembling multiple scle rosis .
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