Functional chromatin features are associated with structural mutations in cancer
暂无分享,去创建一个
[1] Jeffrey H. Chuang,et al. Functional chromatin features are associated with structural mutations in cancer , 2014, BMC Genomics.
[2] Niranjan Nagarajan,et al. Systems consequences of amplicon formation in human breast cancer , 2014, Genome research.
[3] Cheng-Zhong Zhang,et al. Chromothripsis and beyond: rapid genome evolution from complex chromosomal rearrangements , 2013, Genes & development.
[4] Yan Li,et al. A high-resolution map of three-dimensional chromatin interactome in human cells , 2013, Nature.
[5] M. Shen. Chromoplexy: a new category of complex rearrangements in the cancer genome. , 2013, Cancer cell.
[6] Lovelace J. Luquette,et al. Diverse Mechanisms of Somatic Structural Variations in Human Cancer Genomes , 2013, Cell.
[7] Ryan M. Layer,et al. Breakpoint profiling of 64 cancer genomes reveals numerous complex rearrangements spawned by homology-independent mechanisms , 2013, Genome research.
[8] A. Sivachenko,et al. Punctuated Evolution of Prostate Cancer Genomes , 2013, Cell.
[9] S. Gabriel,et al. Somatic rearrangements across cancer reveal classes of samples with distinct patterns of DNA breakage and rearrangement-induced hypermutability , 2012, Genome research.
[10] Edison T Liu,et al. Structural mutations in cancer: mechanistic and functional insights. , 2012, Trends in genetics : TIG.
[11] Data production leads,et al. An integrated encyclopedia of DNA elements in the human genome , 2012 .
[12] Vishwanath R Iyer,et al. Genome-wide Studies of CCCTC-binding Factor (CTCF) and Cohesin Provide Insight into Chromatin Structure and Regulation* , 2012, The Journal of Biological Chemistry.
[13] Marc D. Perry,et al. ChIP-seq guidelines and practices of the ENCODE and modENCODE consortia , 2012, Genome research.
[14] J. Lindberg,et al. Identification of novel CHD1-associated collaborative alterations of genomic structure and functional assessment of CHD1 in prostate cancer , 2012, Oncogene.
[15] S. Boulton,et al. Playing the end game: DNA double-strand break repair pathway choice. , 2012, Molecular cell.
[16] M. Stratton,et al. Tandem duplication of chromosomal segments is common in ovarian and breast cancer genomes , 2012, The Journal of pathology.
[17] ENCODEConsortium,et al. An Integrated Encyclopedia of DNA Elements in the Human Genome , 2012, Nature.
[18] Francesca Chiaromonte,et al. A genome-wide analysis of common fragile sites: What features determine chromosomal instability in the human genome? , 2012, Genome research.
[19] A. Børresen-Dale,et al. The Life History of 21 Breast Cancers , 2012, Cell.
[20] Michael S. Becker,et al. Spatial Organization of the Mouse Genome and Its Role in Recurrent Chromosomal Translocations , 2012, Cell.
[21] M. Nussenzweig,et al. Translocation capture sequencing: a method for high throughput mapping of chromosomal rearrangements. , 2012, Journal of immunological methods.
[22] David A. Orlando,et al. Master Transcription Factors Determine Cell-Type-Specific Responses to TGF-β Signaling , 2011, Cell.
[23] Stefano Monti,et al. Genome-wide Translocation Sequencing Reveals Mechanisms of Chromosome Breaks and Rearrangements in B Cells , 2011, Cell.
[24] Kristian Cibulskis,et al. Genomic sequencing of colorectal adenocarcinomas identifies a recurrent VTI1A-TCF7L2 fusion , 2011, Nature Genetics.
[25] A. McKenna,et al. The Mutational Landscape of Head and Neck Squamous Cell Carcinoma , 2011, Science.
[26] Chee Seng Chan,et al. Comprehensive long-span paired-end-tag mapping reveals characteristic patterns of structural variations in epithelial cancer genomes. , 2011, Genome research.
[27] Timothy J. Durham,et al. "Systematic" , 1966, Comput. J..
[28] Timothy J. Durham,et al. Systematic analysis of chromatin state dynamics in nine human cell types , 2011, Nature.
[29] Eric S. Lander,et al. The genomic complexity of primary human prostate cancer , 2010, Nature.
[30] P. Cook,et al. The DNA moves, not the polymerase , 2011 .
[31] A. Børresen-Dale,et al. COMPLEX LANDSCAPES OF SOMATIC REARRANGEMENT IN HUMAN BREAST CANCER GENOMES , 2009, Nature.
[32] Jie Zhang,et al. Nuclear Receptor-Induced Chromosomal Proximity and DNA Breaks Underlie Specific Translocations in Cancer , 2009, Cell.
[33] I. Amit,et al. Comprehensive mapping of long range interactions reveals folding principles of the human genome , 2011 .
[34] Dustin E. Schones,et al. Global analysis of the insulator binding protein CTCF in chromatin barrier regions reveals demarcation of active and repressive domains. , 2008, Genome research.
[35] Peter R. Cook,et al. Similar active genes cluster in specialized transcription factories , 2008, The Journal of cell biology.
[36] Dustin E. Schones,et al. High-Resolution Profiling of Histone Methylations in the Human Genome , 2007, Cell.
[37] Juliet A. Ellis,et al. The spatial organization of human chromosomes within the nuclei of normal and emerin-mutant cells. , 2001, Human molecular genetics.
[38] P. Seeburg,et al. Structural mechanism for STI-571 inhibition of abelson tyrosine kinase. , 2000, Science.
[39] J. Spivak,et al. Commentary on and reprint of Nowell PC, Hungerford DA, A minute chromosome in human chronic granulocytic leukemia, in Science (1960) 132:1497 , 2000 .
[40] P. Nowell,et al. A minute chromosome in human chronic granulocytic leukemia , 1960 .