Regulation of Ion Channel Expression
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Eduardo Marbán | D. Mckinnon | G. Tomaselli | E. Marbán | B. Rosati | Gordon Tomaselli | David McKinnon | Barbara Rosati
[1] S. Counce. The Strategy of the Genes , 1958, The Yale Journal of Biology and Medicine.
[2] J. Nerbonne,et al. Distribution, Splicing and Glucocorticoid-Induced Expression of Cardiacα1Candα1DVoltage-gated Ca2+Channel mRNAs , 1997 .
[3] R. Wong,et al. Activity-dependent regulation of dendritic growth and patterning , 2002, Nature Reviews Neuroscience.
[4] D. Atar,et al. Excitation-Transcription Coupling Mediated by Zinc Influx through Voltage-dependent Calcium Channels (*) , 1995, The Journal of Biological Chemistry.
[5] W. Giles,et al. A mathematical model of action potential heterogeneity in adult rat left ventricular myocytes. , 2001, Biophysical journal.
[6] N. Spitzer,et al. Coding of neuronal differentiation by calcium transients , 2000, BioEssays : news and reviews in molecular, cellular and developmental biology.
[7] M. Ptashne,et al. Genes and Signals , 2001 .
[8] S Nattel,et al. Molecular mechanisms underlying ionic remodeling in a dog model of atrial fibrillation. , 1999, Circulation research.
[9] K. Takimoto,et al. Glucocorticoid induction of Kv1.5 K+ channel gene expression in ventricle of rat heart. , 1994, Circulation research.
[10] W. Giles,et al. A mathematical model of the electrophysiological alterations in rat ventricular myocytes in type-I diabetes. , 2003, Biophysical journal.
[11] M. Boutjdir,et al. Reexpression of T-type Ca2+ channel gene and current in post-infarction remodeled rat left ventricle. , 2000, Cardiovascular research.
[12] D. Mckinnon,et al. MinK-Related Peptide 1: A &bgr; Subunit for the HCN Ion Channel Subunit Family Enhances Expression and Speeds Activation , 2001, Circulation research.
[13] Mark Ptashne,et al. A Genetic Switch: Gene Control and Phage Lambda , 1986 .
[14] K. Rhodes,et al. Modulation of A-type potassium channels by a family of calcium sensors , 2000, Nature.
[15] A. Imbalzano,et al. Transcriptional Compensation for Loss of an Allele of the Ini1 Tumor Suppressor* , 2004, Journal of Biological Chemistry.
[16] P. Iaizzo,et al. Chloride conductance in mouse muscle is subject to post‐transcriptional compensation of the functional Cl− channel 1 gene dosage , 1997, The Journal of physiology.
[17] J. Nerbonne,et al. Concordant expression of KChIP2 mRNA, protein and transient outward current throughout the canine ventricle , 2003, The Journal of physiology.
[18] N. Klugbauer,et al. Enhanced Expression of L-type Cav1.3 Calcium Channels in Murine Embryonic Hearts from Cav1.2-deficient Mice* , 2003, Journal of Biological Chemistry.
[19] W. Dillmann. Cellular action of thyroid hormone on the heart. , 2002, Thyroid : official journal of the American Thyroid Association.
[20] A. Coulombe,et al. Expression of heart K+ channels in adrenalectomized and catecholamine-depleted reserpine-treated rats. , 2003, Journal of molecular and cellular cardiology.
[21] S. Priori,et al. Low penetrance in the long-QT syndrome: clinical impact. , 1999, Circulation.
[22] R. Harvey,et al. Molecular pathways in myocardial development: a stem cell perspective. , 2003, Cardiovascular research.
[23] C. Rose. Book Review: Na+ Signals at Central Synapses , 2002 .
[24] P. C. Viswanathan,et al. Effects of IKr and IKs heterogeneity on action potential duration and its rate dependence: a simulation study. , 1999, Circulation.
[25] A. Kobori,et al. Characterization and subcellular localization of KCNQ1 with a heterozygous mutation in the C terminus. , 2001, Journal of molecular and cellular cardiology.
[26] R. Fields. Effects of ion channel activity on development of dorsal root ganglion neurons. , 1998, Journal of neurobiology.
[27] M. Sanguinetti,et al. Functional Effects of Mutations in KvLQT1 that Cause Long QT Syndrome , 1999, Journal of cardiovascular electrophysiology.
[28] S. Nattel,et al. Canine Ventricular KCNE2 Expression Resides Predominantly in Purkinje Fibers , 2003, Circulation research.
[29] G. Gintant,et al. Tissue and species distribution of mRNA for the IKr-like K+ channel, erg. , 1997, Circulation research.
[30] M. Fishbein,et al. Sympathetic nerve sprouting, electrical remodeling and the mechanisms of sudden cardiac death. , 2001, Cardiovascular research.
[31] R. Passier,et al. Regulation of cardiac growth and development by SRF and its cofactors. , 2002, Cold Spring Harbor symposia on quantitative biology.
[32] W. Murphy,et al. Resolution of the Early Placental Mammal Radiation Using Bayesian Phylogenetics , 2001, Science.
[33] J. Nerbonne,et al. Genetic Manipulation of Cardiac K+ Channel Function in Mice: What Have We Learned, and Where Do We Go From Here? , 2001, Circulation research.
[34] C. Deutsch. The Birth of a Channel , 2003, Neuron.
[35] D. Mckinnon,et al. Regulation of KChIP2 potassium channel β subunit gene expression underlies the gradient of transient outward current in canine and human ventricle , 2001, The Journal of physiology.
[36] M. Freeman. Feedback control of intercellular signalling in development , 2000, Nature.
[37] Ira S. Cohen,et al. MinK-Related Peptide 1 , 2001 .
[38] W. Catterall,et al. Class I and IV antiarrhythmic drugs and cytosolic calcium regulate mRNA encoding the sodium channel alpha subunit in rat cardiac muscle. , 1992, Molecular pharmacology.
[39] L. Leinwand. Heart Development , 1999, Nature Medicine.
[40] Stanley Nattel,et al. Differential Distribution of Cardiac Ion Channel Expression as a Basis for Regional Specialization in Electrical Function , 2002, Circulation research.
[41] M. Stern,et al. Calcium in close quarters: microdomain feedback in excitation-contraction coupling and other cell biological phenomena. , 1997, Annual review of biophysics and biomolecular structure.
[42] M. Rosen,et al. Cardiac Memory Is Associated With Decreased Levels of the Transcriptional Factor CREB Modulated by Angiotensin II and Calcium , 2003, Circulation research.
[43] R. Veitia,et al. Exploring the etiology of haploinsufficiency. , 2002, BioEssays : news and reviews in molecular, cellular and developmental biology.
[44] B. Garvik,et al. Principles for the Buffering of Genetic Variation , 2001, Science.
[45] J. Nerbonne,et al. Expression of Distinct ERG Proteins in Rat, Mouse, and Human Heart , 2000, The Journal of Biological Chemistry.
[46] A. Paulussen,et al. A Novel Mutation (T65P) in the PAS Domain of the Human Potassium Channel HERG Results in the Long QT Syndrome by Trafficking Deficiency* , 2002, The Journal of Biological Chemistry.
[47] P. Backx,et al. Effects of Development and Thyroid Hormone on K+ Currents and K+ Channel Gene Expression in Rat Ventricle , 1997, The Journal of physiology.
[48] J. Sadoshima,et al. The cellular and molecular response of cardiac myocytes to mechanical stress. , 1997, Annual review of physiology.
[49] H. Strauss,et al. Heterogeneous expression of KChIP2 isoforms in the ferret heart , 2002, The Journal of physiology.
[50] R. Cripps,et al. Control of cardiac development by an evolutionarily conserved transcriptional network. , 2002, Developmental biology.
[51] Eric C. Griffith,et al. Regulation of transcription factors by neuronal activity , 2002, Nature Reviews Neuroscience.
[52] Stanley Nattel,et al. Molecular basis of species-specific expression of repolarizing K+ currents in the heart. , 2003, American journal of physiology. Heart and circulatory physiology.
[53] W. Giles,et al. Heterogeneity of action potential waveforms and potassium currents in rat ventricle. , 1993, Cardiovascular research.
[54] C. Antzelevitch,et al. Characteristics of the delayed rectifier current (IKr and IKs) in canine ventricular epicardial, midmyocardial, and endocardial myocytes. A weaker IKs contributes to the longer action potential of the M cell. , 1995, Circulation research.
[55] Mark E. Anderson,et al. Cardiac ion channels. , 2002, Annual review of physiology.
[56] D. Bers. Cardiac excitation–contraction coupling , 2002, Nature.
[57] C. Antzelevitch. The Brugada Syndrome: Ionic Basis and Arrhythmia Mechanisms , 2001, Journal of cardiovascular electrophysiology.
[58] J. Nerbonne. Molecular basis of functional voltage‐gated K+ channel diversity in the mammalian myocardium , 2000, The Journal of physiology.
[59] J. Nerbonne,et al. Microarray Analysis Reveals Complex Remodeling of Cardiac Ion Channel Expression With Altered Thyroid Status: Relation to Cellular and Integrated Electrophysiology , 2003, Circulation research.
[60] E. Olson,et al. MEF2: a calcium-dependent regulator of cell division, differentiation and death. , 2002, Trends in biochemical sciences.
[61] T. Lømo,et al. Control of ACh sensitivity in rat muscle fibers. , 1976, Cold Spring Harbor symposia on quantitative biology.
[62] M. Sanguinetti,et al. Long QT Syndrome-associated Mutations in the S4-S5 Linker of KvLQT1 Potassium Channels Modify Gating and Interaction with minK Subunits* , 1999, The Journal of Biological Chemistry.
[63] J. Nerbonne,et al. Molecular basis of transient outward K+ current diversity in mouse ventricular myocytes , 1999, The Journal of physiology.
[64] Ronald W. Davis,et al. Role of duplicate genes in genetic robustness against null mutations , 2003, Nature.
[65] E. Neher,et al. Linearized Buffered Ca2+ Diffusion in Microdomains and Its Implications for Calculation of [Ca2+] at the Mouth of a Calcium Channel , 1997, The Journal of Neuroscience.
[66] J. Nerbonne,et al. Functional consequences of elimination of i(to,f) and i(to,s): early afterdepolarizations, atrioventricular block, and ventricular arrhythmias in mice lacking Kv1.4 and expressing a dominant-negative Kv4 alpha subunit. , 2000, Circulation research.
[67] E. Marbán,et al. Molecular dissection of cardiac repolarization by in vivo Kv4.3 gene transfer. , 2000, The Journal of clinical investigation.
[68] M. Sanguinetti,et al. Spectrum of HERG K+-channel dysfunction in an inherited cardiac arrhythmia. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[69] 宁北芳,et al. 疟原虫var基因转换速率变化导致抗原变异[英]/Paul H, Robert P, Christodoulou Z, et al//Proc Natl Acad Sci U S A , 2005 .
[70] R. Dolmetsch. Excitation-Transcription Coupling: Signaling by Ion Channels to the Nucleus , 2003, Science's STKE.
[71] Zhao Zhang,et al. Functional Roles of Ca(v)1.3 (alpha(1D)) calcium channel in sinoatrial nodes: insight gained using gene-targeted null mutant mice. , 2002, Circulation research.
[72] M. Biel,et al. Cellular expression and functional characterization of four hyperpolarization-activated pacemaker channels in cardiac and neuronal tissues. , 2001, European journal of biochemistry.
[73] P. Boyden,et al. Electrical remodeling in ischemia and infarction. , 1999, Cardiovascular research.
[74] Yoshihiro Kubo,et al. Primary structure and functional expression of a rat G-protein-coupled muscarinic potassium channel , 1993, Nature.
[75] H. Hamada,et al. Contribution of KChIP2 to the developmental increase in transient outward current of rat cardiomyocytes. , 2003, Journal of molecular and cellular cardiology.
[76] E. Keller. Developmental Robustness , 2002, Annals of the New York Academy of Sciences.
[77] J. Ross,et al. A Defect in the Kv Channel-Interacting Protein 2 (KChIP2) Gene Leads to a Complete Loss of I to and Confers Susceptibility to Ventricular Tachycardia , 2001, Cell.
[78] J. Hume,et al. Ionic basis of the different action potential configurations of single guinea‐pig atrial and ventricular myocytes. , 1985, The Journal of physiology.
[79] A. Grant,et al. Abnormal cardiac Na(+) channel properties and QT heart rate adaptation in neonatal ankyrin(B) knockout mice. , 2000, Circulation research.
[80] G. Rubin,et al. The Role of the Genome Project in Determining Gene Function: Insights from Model Organisms , 1996, Cell.
[81] Electrical remodeling in hearts from a calcium-dependent mouse model of hypertrophy and failure: complex nature of K+ current changes and action potential duration. , 2003, Journal of the American College of Cardiology.
[82] J. Seidman,et al. Transcription factor haploinsufficiency: when half a loaf is not enough. , 2002, The Journal of clinical investigation.
[83] U Ravens,et al. Molecular Basis of Downregulation of G-Protein–Coupled Inward Rectifying K+ Current (IK,ACh) in Chronic Human Atrial Fibrillation: Decrease in GIRK4 mRNA Correlates With Reduced IK,ACh and Muscarinic Receptor–Mediated Shortening of Action Potentials , 2001, Circulation.
[84] Milena B. Furtado,et al. Homeodomain factor Nkx2-5 in heart development and disease. , 2002, Cold Spring Harbor symposia on quantitative biology.
[85] G. Crabtree,et al. Cell signaling can direct either binary or graded transcriptional responses , 2001, The EMBO journal.
[86] H. Duff,et al. Upregulation of the rat cardiac sodium channel by in vivo treatment with a class I antiarrhythmic drug. , 1991, The Journal of clinical investigation.
[87] N. Klugbauer,et al. Expression of T‐ and L‐type calcium channel mRNA in murine sinoatrial node , 2000, FEBS letters.
[88] W. Giles,et al. Cardiac Ion Channel Expression and Contractile Function in Mice with Deletion of Thyroid Hormone Receptor α or β1 , 2001 .
[89] I. Komuro,et al. Roles of cardiac transcription factors in cardiac hypertrophy. , 2003, Circulation research.
[90] D. Linden,et al. Induction of cerebellar long-term depression in culture requires postsynaptic action of Sodium Ions , 1993, Neuron.
[91] D. Mckinnon,et al. Distribution and prevalence of hyperpolarization-activated cation channel (HCN) mRNA expression in cardiac tissues. , 1999, Circulation research.
[92] R. Carey,et al. Newly recognized components of the renin-angiotensin system: potential roles in cardiovascular and renal regulation. , 2003, Endocrine reviews.
[93] G. Lembo,et al. Molecular interplay between mechanical and humoral signalling in cardiac hypertrophy. , 2003, Trends in molecular medicine.
[94] Na+ signals at central synapses. , 2002, The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry.
[95] H. Fozzard. Cardiac sodium and calcium channels: a history of excitatory currents. , 2002, Cardiovascular research.
[96] R. Fields,et al. Gene regulation by patterned electrical activity during neural and skeletal muscle development , 1999, Current Opinion in Neurobiology.
[97] P. Molenaar,et al. The human heart endothelin system: ET-1 synthesis, storage, release and effect. , 2000, Trends in pharmacological sciences.
[98] J. Nerbonne,et al. Functional knockout of the transient outward current, long-QT syndrome, and cardiac remodeling in mice expressing a dominant-negative Kv4 alpha subunit. , 1998, Circulation research.
[99] R. S. Williams,et al. Electrical Stimulation of Neonatal Cardiac Myocytes Activates the NFAT3 and GATA4 Pathways and Up-regulates the Adenylosuccinate Synthetase 1 Gene* , 2000, The Journal of Biological Chemistry.
[100] Jamie I Vandenberg,et al. Slowed conduction and ventricular tachycardia after targeted disruption of the cardiac sodium channel gene Scn5a , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[101] Jeanne M Nerbonne,et al. Regional upregulation of Kv2.1-encoded current, IK,slow2, in Kv1DN mice is abolished by crossbreeding with Kv2DN mice. , 2003, American journal of physiology. Heart and circulatory physiology.
[102] Is there an A-type K+ current in guinea pig ventricular myocytes? , 2003, American journal of physiology. Heart and circulatory physiology.
[103] Calum A MacRae,et al. Risk stratification in the long-QT syndrome. , 2003, The New England journal of medicine.
[104] E. Davidson. Genomic Regulatory Systems: Development and Evolution , 2005 .
[105] Y. Shimoni,et al. Role of PKC in autocrine regulation of rat ventricular K+ currents by angiotensin and endothelin. , 2003, American journal of physiology. Heart and circulatory physiology.
[106] D. Mckinnon,et al. Quantitative analysis of potassium channel mRNA expression in atrial and ventricular muscle of rats. , 1994, Circulation research.
[107] Y. Dong,et al. Systematic functional analysis of the Caenorhabditis elegans genome using RNAi , 2003, Nature.
[108] Igor Stagljar,et al. Analysis of membrane protein interactions using yeast-based technologies. , 2002, Trends in biochemical sciences.
[109] G. Tomaselli,et al. Electrophysiological remodeling in hypertrophy and heart failure. , 1999, Cardiovascular research.
[110] A. Gramolini,et al. Ankyrin-B mutation causes type 4 long-QT cardiac arrhythmia and sudden cardiac death , 2003, Nature.
[111] R. Palmiter,et al. Neuropeptide Y Is an Essential In Vivo Developmental Regulator of Cardiac ICa,L , 2003, Circulation research.
[112] Roberto Malinow,et al. AMPA receptor trafficking and long-term potentiation. , 2003, Philosophical transactions of the Royal Society of London. Series B, Biological sciences.
[113] E. Turner,et al. Direct autoregulation and gene dosage compensation by POU-domain transcription factor Brn3a , 2003, Development.
[114] M. Sanguinetti,et al. Molecular and Cellular Mechanisms of Cardiac Arrhythmias , 2001, Cell.
[115] H Honjo,et al. The sinoatrial node, a heterogeneous pacemaker structure. , 2000, Cardiovascular research.
[116] A. Coulombe,et al. Angiotensin II Signaling Pathways Mediate Expression of Cardiac T-Type Calcium Channels , 2003, Circulation research.
[117] Lin Chen,et al. Transcriptional regulation by calcium, calcineurin, and NFAT. , 2003, Genes & development.
[118] D. Srivastava,et al. Molecular mechanisms of ventricular hypoplasia. , 2002, Cold Spring Harbor symposia on quantitative biology.
[119] L. Boscá,et al. Regional distribution of hyperpolarization‐activated current (If) and hyperpolarization‐activated cyclic nucleotide‐gated channel mRNA expression in ventricular cells from control and hypertrophied rat hearts , 2003, The Journal of physiology.
[120] M. Allessie,et al. Atrial fibrillation begets atrial fibrillation. A study in awake chronically instrumented goats. , 1995, Circulation.