Staphylococcus aureus requires less virulence to establish an infection in diabetic hosts.
暂无分享,去创建一个
A. Mellmann | G. Peters | C. Sunderkötter | B. Löffler | L. Tuchscherr | É. Korpos | L. Sorokin | S. Deinhardt-Emmer | H. van de Vyver | Clais R. Findeisen | Salome Kherkheulidze | Anke Siegmund | Olaf Bach | Martin Rindert | Stefanie Deinhardt-Emmer
[1] M. Spraul,et al. Diabetic Foot Syndrome. , 2020, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.
[2] T. Foster,et al. The remarkably multifunctional fibronectin binding proteins of Staphylococcus aureus , 2016, European Journal of Clinical Microbiology and Infectious Diseases.
[3] A. Mellmann,et al. Real-Time Genome Sequencing of Resistant Bacteria Provides Precision Infection Control in an Institutional Setting , 2016, Journal of Clinical Microbiology.
[4] C. Dunyach-Rémy,et al. Staphylococcus aureus Toxins and Diabetic Foot Ulcers: Role in Pathogenesis and Interest in Diagnosis , 2016, Toxins.
[5] B. Furman,et al. Streptozotocin‐Induced Diabetic Models in Mice and Rats , 2015, Current protocols in pharmacology.
[6] M. Fraunholz,et al. Sigma Factor SigB Is Crucial to Mediate Staphylococcus aureus Adaptation during Chronic Infections , 2015, PLoS pathogens.
[7] V. Fowler,et al. Amino acid alterations in fibronectin binding protein A (FnBPA) and bacterial genotype are associated with cardiac device related infection in Staphylococcus aureus bacteraemia. , 2015, The Journal of infection.
[8] Benjamin A Lipsky,et al. Ceftaroline fosamil for treatment of diabetic foot infections: the CAPTURE study experience , 2015, Diabetes/metabolism research and reviews.
[9] G. Peters,et al. Staphylococcus aureus isolates from chronic osteomyelitis are characterized by high host cell invasion and intracellular adaptation, but still induce inflammation. , 2014, International journal of medical microbiology : IJMM.
[10] Benjamin A Lipsky,et al. Do Diabetic Foot Infections With Methicillin-Resistant Staphylococcus aureus Differ From Those With Other Pathogens? , 2014, The international journal of lower extremity wounds.
[11] S. Fauziyah,et al. Antibiotic therapy for diabetic foot infections in a tertiary care hospital in Jakarta, Indonesia. , 2014, Diabetes & metabolic syndrome.
[12] Suleyman Yazici,et al. The relationship between complete blood count parameters and Fontaine’s Stages in patients with peripheral arterial disease , 2014, Vascular.
[13] Benjamin A Lipsky,et al. Diabetic Foot Ulcer Microbiome: One Small Step for Molecular Microbiology . . . One Giant Leap for Understanding Diabetic Foot Ulcers? , 2013, Diabetes.
[14] R. Ridley,et al. Differential adhesion and invasion by Staphylococcus aureus of epithelial cells derived from different anatomical sites. , 2012, Journal of medical microbiology.
[15] M. Fraunholz,et al. Intracellular staphylococcus aureus: Live-in and let die , 2012, Front. Cell. Inf. Microbio..
[16] Sunny Park,et al. Reduced Neutrophil Apoptosis in Diabetic Mice during Staphylococcal Infection Leads to Prolonged Tnfα Production and Reduced Neutrophil Clearance , 2011, PloS one.
[17] R. Proctor,et al. Staphylococcus aureus phenotype switching: an effective bacterial strategy to escape host immune response and establish a chronic infection , 2011, EMBO molecular medicine.
[18] Benjamin A Lipsky,et al. Treating Osteomyelitis: Antibiotics and Surgery , 2011, Plastic and reconstructive surgery.
[19] D. Viemann,et al. Staphylococcal strains vary greatly in their ability to induce an inflammatory response in endothelial cells. , 2010, The Journal of infectious diseases.
[20] F. Lowy. Staphylococcus aureus infections. , 2009, The New England journal of medicine.
[21] George Y. Liu. Molecular Pathogenesis of Staphylococcus aureus Infection , 2009, Pediatric Research.
[22] J. S. van de Gevel,et al. Contribution of (sub)domains of Staphylococcus aureus fibronectin-binding protein to the proinflammatory and procoagulant response of human vascular endothelial cells , 2009, Thrombosis and Haemostasis.
[23] Sunny Park,et al. Defects in Innate Immunity Predispose C57BL/6J-Leprdb/Leprdb Mice to Infection by Staphylococcus aureus , 2008, Infection and Immunity.
[24] S. Lenzen,et al. The mechanisms of alloxan- and streptozotocin-induced diabetes , 2008, Diabetologia.
[25] A. Kennedy,et al. Identification of novel cytolytic peptides as key virulence determinants for community-associated MRSA , 2007, Nature Medicine.
[26] Jean C. Lee,et al. The pathogenesis of Staphylococcus aureus infection in the diabetic NOD mouse. , 2005, Diabetes.
[27] U. Fischer,et al. Multiple virulence factors are required for Staphylococcus aureus‐induced apoptosis in endothelial cells , 2005, Cellular microbiology.
[28] C. Wolz,et al. sae is essential for expression of the staphylococcal adhesins Eap and Emp. , 2005, Microbiology.
[29] G. Peters,et al. Truncation of Fibronectin-Binding Proteins in Staphylococcus aureus Strain Newman Leads to Deficient Adherence and Host Cell Invasion Due to Loss of the Cell Wall Anchor Function , 2004, Infection and Immunity.
[30] F. Waldvogel,et al. Osteomyelitis , 2004, The Lancet.
[31] G. Peters,et al. Reduced adherence and host cell invasion by methicillin-resistant Staphylococcus aureus expressing the surface protein Pls. , 2004, The Journal of infectious diseases.
[32] K. Schulze-Osthoff,et al. Staphylococcus aureusα‐toxin induces apoptosis in peripheral blood mononuclear cells: role of endogenous tumour necrosis factor‐α and the mitochondrial death pathway , 2003, Cellular microbiology.
[33] B. Menzies,et al. The role of fibronectin binding proteins in the pathogenesis of Staphylococcus aureus infections , 2003, Current opinion in infectious diseases.
[34] M. Clare-Salzer,et al. Activation of CD1d-restricted T cells protects NOD mice from developing diabetes by regulating dendritic cell subsets , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[35] Chyung-Ru Wang,et al. Cd1-Restricted Nk T Cells Protect Nonobese Diabetic Mice from Developing Diabetes , 2001, The Journal of experimental medicine.
[36] M. Pfaffl,et al. A new mathematical model for relative quantification in real-time RT-PCR. , 2001, Nucleic acids research.
[37] J. Bluestone,et al. B7/CD28 costimulation is essential for the homeostasis of the CD4+CD25+ immunoregulatory T cells that control autoimmune diabetes. , 2000, Immunity.
[38] Bhanu Sinha,et al. Fibronectin‐binding protein acts as Staphylococcus aureus invasin via fibronectin bridging to integrin α5β1 , 1999, Cellular microbiology.
[39] M. Smeltzer,et al. Mechanisms of Staphylococcus aureus invasion of cultured osteoblasts. , 1999, Microbial pathogenesis.
[40] T. Foster,et al. Surface protein adhesins of Staphylococcus aureus. , 1998, Trends in microbiology.
[41] H. Gaskins,et al. Defects in the differentiation and function of antigen presenting cells in NOD/Lt mice. , 1993, Journal of immunology.
[42] I Nicoletti,et al. A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry. , 1991, Journal of immunological methods.
[43] J. Bach,et al. Anti‐suppressor effect of cyclophosphamide on the development of spontaneous diabetes in nod mice , 1988, European journal of immunology.
[44] J. Satoh,et al. Immunologic Aspects of the Nonobese Diabetic (NOD) Mouse: Abnormalities of Cellular Immunity , 1983, Diabetes.
[45] D. Armstrong,et al. 2012 infectious diseases society of america clinical practice guideline for the diagnosis and treatment of diabetic foot infections. , 2013, Journal of the American Podiatric Medical Association.
[46] J. Roth,et al. Subcutaneous infection with S. aureus in mice reveals association of resistance with influx of neutrophils and Th2 response. , 2011, The Journal of investigative dermatology.
[47] M. Bischoff,et al. Role of (cid:2) B in the Expression of Staphylococcus aureus Cell Wall Adhesins ClfA and FnbA and Contribution to Infectivity in a Rat Model of Experimental Endocarditis , 2005 .
[48] Mark S. Anderson,et al. The NOD mouse: a model of immune dysregulation. , 2005, Annual review of immunology.
[49] J. Bluestone,et al. Impairment of NK cell function by NKG2D modulation in NOD mice. , 2003, Immunity.
[50] S Bhakdi,et al. Alpha-toxin of Staphylococcus aureus. , 1991, Microbiological reviews.
[51] F. Cerra,et al. Stimulation of repair in chronic, nonhealing, cutaneous ulcers using platelet-derived wound healing formula. , 1990, Surgery, gynecology & obstetrics.