Organ-protective effects on the liver and kidney by minocycline in small piglets undergoing cardiopulonary bypass
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F. Mohr | S. Dhein | F. Bakhtiary | M. Kostelka | A. Sobiraj | A. Salameh | M. Grassl | M. Gerdom | M. Vollroth | K. Krämer | S. Salisch | Axel Sobiraj
[1] T. Dawson,et al. Parthanatos: mitochondrial‐linked mechanisms and therapeutic opportunities , 2014, British journal of pharmacology.
[2] I. Sevrioukova. Apoptosis-inducing factor: structure, function, and redox regulation. , 2011, Antioxidants & redox signaling.
[3] C. Krawczeski,et al. Incidence, risk factors, and outcomes of acute kidney injury after pediatric cardiac surgery: A prospective multicenter study* , 2011, Critical care medicine.
[4] A. Ündar,et al. Pediatric Cardiopulmonary Bypass , 2011, World journal for pediatric & congenital heart surgery.
[5] R. Nieuwland,et al. Complement activation on the surface of cell-derived microparticles during cardiac surgery with cardiopulmonary bypass - is retransfusion of pericardial blood harmful? , 2011, Perfusion.
[6] J. Karliner,et al. Minocycline Protects Cardiac Myocytes Against Simulated Ischemia–Reperfusion Injury by Inhibiting Poly(ADP-ribose) Polymerase-1 , 2010, Journal of cardiovascular pharmacology.
[7] Zhen Jin,et al. Protective effects of penehyclidine hydrochloride on liver injury in a rat cardiopulmonary bypass model , 2010, European journal of anaesthesiology.
[8] W. Stoney. Evolution of cardiopulmonary bypass. , 2009, Circulation.
[9] F. Mohr,et al. Ischemic and inflammatory lung impairment by extracorporeal circulation: effect of PARP-inhibition by INO1001. , 2008, Pharmacological research.
[10] D. Portilla,et al. Analytic Reviews: Cardiac Surgery as a Cause of Acute Kidney Injury: Pathogenesis and Potential Therapies , 2008 .
[11] C. Szabó,et al. Role of poly(ADP-ribose) polymerase 1 (PARP-1) in cardiovascular diseases: the therapeutic potential of PARP inhibitors. , 2007, Cardiovascular drug reviews.
[12] Yingbin Xiao,et al. Growth hormone prevents acute liver injury induced by cardiopulmonary bypass in a rat model. , 2007, The Journal of thoracic and cardiovascular surgery.
[13] P. Rhee,et al. The decreasing incidence of late posttraumatic acute respiratory distress syndrome: the potential role of lung protective ventilation and conservative transfusion practice. , 2007, The Journal of trauma.
[14] C. Becker,et al. Pathophysiology of contrast-induced nephropathy. , 2006, The American journal of cardiology.
[15] R. Swanson,et al. Minocycline inhibits poly(ADP-ribose) polymerase-1 at nanomolar concentrations. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[16] C. Yeh,et al. INHIBITION OF POLY(ADP-RIBOSE) POLYMERASE REDUCES CARDIOMYOCYTIC APOPTOSIS AFTER GLOBAL CARDIAC ARREST UNDER CARDIOPULMONARY BYPASS , 2006, Shock.
[17] C. Szabó. Cardioprotective effects of poly(ADP-ribose) polymerase inhibition. , 2005, Pharmacological research.
[18] S. Hagl,et al. Poly(ADP-ribose) polymerase inhibitor PJ-34 reduces mesenteric vascular injury induced by experimental cardiopulmonary bypass with cardiac arrest. , 2005, American journal of physiology. Heart and circulatory physiology.
[19] E. Baskın,et al. Acute Renal Failure and Mortality After Open-Heart Surgery in Infants , 2005, Renal failure.
[20] S. Hagl,et al. Poly-ADP-Ribose Polymerase Inhibition Reduces Mesenteric Injury after Cardiopulmonary Bypass* , 2004, The Thoracic and cardiovascular surgeon.
[21] A. Consolini,et al. Development of an HPLC method for determination of metabolic compounds in myocardial tissue. , 2004, Journal of pharmaceutical and biomedical analysis.
[22] S. Hagl,et al. INO-1001 A NOVEL POLY(ADP-RIBOSE) POLYMERASE (PARP) INHIBITOR IMPROVES CARDIAC AND PULMONARY FUNCTION AFTER CRYSTALLOID CARDIOPLEGIA AND EXTRACORPORAL CIRCULATION , 2004, Shock.
[23] A. Grayson,et al. Acute renal failure in coronary artery bypass surgery: independent effect of cardiopulmonary bypass. , 2004, The Annals of thoracic surgery.
[24] S. Ohri,et al. A prospective randomized study to evaluate the renoprotective action of beating heart coronary surgery in low risk patients. , 2002, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.
[25] K. Taylor,et al. Lung injury and acute respiratory distress syndrome after cardiopulmonary bypass. , 1999, The Annals of thoracic surgery.
[26] A. Plessis. Mechanisms of brain injury during infant cardiac surgery. , 1999 .
[27] M. Nicholson,et al. New model of renal warm ischaemia–reperfusion injury for comparative functional, morphological and pathophysiological studies , 1998, The British journal of surgery.
[28] S. Cuzzocrea,et al. Peroxynitrite‐induced thymocyte apoptosis: the role of caspases and poly (ADP‐ribose) synthetase (PARS) activation , 1998, Immunology.
[29] G. Holmes,et al. Developmental and neurologic status of children after heart surgery with hypothermic circulatory arrest or low-flow cardiopulmonary arrest , 1995 .
[30] E. Ingham. The effects of tetracyclines and erythromycin on complement activation in vitro. , 1990, Acta dermato-venereologica.
[31] S Westaby,et al. Complement and the damaging effects of cardiopulmonary bypass. , 1983, Thorax.
[32] B. Myers,et al. Acute renal failure following cardiac surgery. , 1979, The Journal of thoracic and cardiovascular surgery.
[33] Fang Yuan,et al. Minocycline protects against hepatic ischemia/reperfusion injury in a rat model. , 2015, Biomedical reports.
[34] F. Mohr,et al. Reno-protective effects of epigallocatechingallate in a small piglet model of extracorporeal circulation. , 2013, Pharmacological Research.
[35] G. Kroemer,et al. NATURAL COMPOUNDS AND THEIR ROLE IN APOPTOTIC CELL SIGNALING PATHWAYS AIF: Not Just an Apoptosis-Inducing Factor , 2009 .
[36] G. Angelini,et al. Splanchnic organ injury during coronary surgery with or without cardiopulmonary bypass: a randomized, controlled trial. , 2006, The Annals of thoracic surgery.
[37] E. Kobayashi,et al. Enhanced responsiveness of circulatory neutrophils after cardiopulmonary bypass: increased aggregability and superoxide producing capacity. , 2000, Artificial organs.
[38] A. du Plessis. Mechanisms of brain injury during infant cardiac surgery. , 1999, Seminars in pediatric neurology.
[39] G. Hill. The Inflammatory Response to Cardiopulmonary Bypass , 1996, International anesthesiology clinics.
[40] G. Nisticó,et al. Nitric oxide release during hypothermic versus normothermic cardiopulmonary bypass. , 1995, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.