Transected neurites, apoptotic neurons, and reduced inflammation in cortical multiple sclerosis lesions
暂无分享,去创建一个
B. Trapp | J. Peterson | S. Mörk | L. Bö | A. Chang | Ansi Chang | John W. Peterson | Lars Bö | Sverre Mörk | Bruce D. Trapp | Ansi Chang
[1] M. Graeber,et al. Axotomy of the rat facial nerve leads to increased CR3 complement receptor expression by activated microglial cells , 1988, Journal of neuroscience research.
[2] W. Streit,et al. Reactive microgliosis , 1999, Progress in Neurobiology.
[3] A J Thompson,et al. Progressive cerebral atrophy in multiple sclerosis. A serial MRI study. , 1996, Brain : a journal of neurology.
[4] C. Portera-Cailliau,et al. Occipital cortex ablation in adult rat causes retrograde neuronal death in the lateral geniculate nucleus that resembles apoptosis , 1998, Neuroscience.
[5] W. L. Benedict,et al. Multiple Sclerosis , 2007, Journal - Michigan State Medical Society.
[6] B. Trapp,et al. Amyloid Load and Neural Elements in Alzheimer's Disease and Nondemented Individuals with High Amyloid Plaque Density , 1996, Experimental Neurology.
[7] P M Matthews,et al. Axonal injury or loss in the internal capsule and motor impairment in multiple sclerosis. , 2000, Archives of neurology.
[8] F. Barkhof,et al. Cortical lesions in multiple sclerosis. , 1999, Brain : a journal of neurology.
[9] M. Palkovits,et al. Axonal changes in chronic demyelinated cervical spinal cord plaques. , 2000, Brain : a journal of neurology.
[10] C. Mackay,et al. T-cell function and migration. Two sides of the same coin. , 2000, The New England journal of medicine.
[11] D. Boire,et al. Transneuronal retrograde degeneration of retinal ganglion cells following cerebral hemispherectomy in cats , 1997, Brain Research.
[12] G. Landreth,et al. Inflammatory Mechanisms in Alzheimer's Disease: Inhibition of β-Amyloid-Stimulated Proinflammatory Responses and Neurotoxicity by PPARγ Agonists , 2000, The Journal of Neuroscience.
[13] D. Walker,et al. Microglia in degenerative neurological disease , 1993, Glia.
[14] H. Lassmann,et al. Axonal Pathology in Multiple Sclerosis. A Historical Note , 1999, Brain Pathology.
[15] S. Appel,et al. TISSUE CULTURE STUDIES OF DEMYELINATION * , 1965, Annals of the New York Academy of Sciences.
[16] B. Trapp,et al. Detection of MHC class II-antigens on macrophages and microglia, but not on astrocytes and endothelia in active multiple sclerosis lesions , 1994, Journal of Neuroimmunology.
[17] P M Matthews,et al. Axonal damage correlates with disability in patients with relapsing-remitting multiple sclerosis. Results of a longitudinal magnetic resonance spectroscopy study. , 1998, Brain : a journal of neurology.
[18] L. Scheinberg,et al. On the immunopathology of plaque development and repair in multiple sclerosis , 1988, Journal of Neuroimmunology.
[19] W. Mcdonald. RACHELLE FISHMAN-MATTHEW MOORE LECTURE: The Pathological and Clinical Dynamics of Multiple Sclerosis , 1994, Journal of neuropathology and experimental neurology.
[20] H. Lassmann,et al. Multiple sclerosis and chronic autoimmune encephalomyelitis: a comparative quantitative study of axonal injury in active, inactive, and remyelinated lesions. , 2000, The American journal of pathology.
[21] P. Matthews,et al. Imaging of axonal damage in multiple sclerosis: Spatial distribution of magnetic resonance imaging lesions , 1997, Annals of neurology.
[22] J. Breitner. The role of anti-inflammatory drugs in the prevention and treatment of Alzheimer's disease. , 1996, Annual review of medicine.
[23] F. Roncaroli,et al. Neuropathology of multiple sclerosis , 2012 .
[24] T. Springer. Traffic signals for lymphocyte recirculation and leukocyte emigration: The multistep paradigm , 1994, Cell.
[25] G. Baltuch,et al. Microglia as mediators of inflammatory and degenerative diseases. , 1999, Annual review of neuroscience.
[26] R. Rudick,et al. Use of the brain parenchymal fraction to measure whole brain atrophy in relapsing-remitting MS , 1999, Neurology.
[27] G. Landreth,et al. Inflammatory mechanisms in Alzheimer's disease: inhibition of beta-amyloid-stimulated proinflammatory responses and neurotoxicity by PPARgamma agonists. , 2000, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[28] M. Carson,et al. Integrating innate and adaptive immunity in the whole animal , 1999, Immunological reviews.
[29] B. Weinshenker,et al. Epidemiology of multiple sclerosis. , 1996, Neurologic clinics.
[30] R. Rudick,et al. Axonal transection in the lesions of multiple sclerosis. , 1998, The New England journal of medicine.
[31] J. Parisi,et al. Heterogeneity of multiple sclerosis lesions: Implications for the pathogenesis of demyelination , 2000, Annals of neurology.
[32] G. Barker,et al. 1H magnetic resonance spectroscopy of chronic cerebral white matter lesions and normal appearing white matter in multiple sclerosis , 1997, Journal of neurology, neurosurgery, and psychiatry.
[33] B. Brownell,et al. The distribution of plaques in the cerebrum in multiple sclerosis , 1962, Journal of neurology, neurosurgery, and psychiatry.
[34] B. Trapp,et al. NG2-Positive Oligodendrocyte Progenitor Cells in Adult Human Brain and Multiple Sclerosis Lesions , 2000, The Journal of Neuroscience.
[35] Nikos Evangelou,et al. Quantitative pathological evidence for axonal loss in normal appearing white matter in multiple sclerosis , 2000, Annals of neurology.
[36] V. Perry,et al. Axonal damage in acute multiple sclerosis lesions. , 1997, Brain : a journal of neurology.
[37] T. Tabira,et al. Multiple sclerosis cerebrospinal fluid produces myelin lesions in tadpole optic nerves. , 1976, The New England journal of medicine.
[38] R. Rudick,et al. Neurological disability correlates with spinal cord axonal loss and reduced N‐acetyl aspartate in chronic multiple sclerosis patients , 2000, Annals of neurology.