Effect of Iodine Excess on Th1, Th2, Th17, and Treg Cell Subpopulations in the Thyroid of NOD.H-2h4 Mice
暂无分享,去创建一个
W. Teng | Z. Shan | Jia Xu | T. Gao | R. Shi | Xiao Yang | Xi-yu Zhou | Jin-qiao Qu
[1] Foad Katirai,et al. Th17 cell plays a role in the pathogenesis of Hashimoto's thyroiditis in patients. , 2013, Clinical immunology.
[2] Yushu Li,et al. Dynamic Changes of CD4+CD25+ Regulatory T Cells in NOD.H-2h4 Mice with Iodine-Induced Autoimmune Thyroiditis , 2011, Biological Trace Element Research.
[3] Jue Wei,et al. Interleukin-17 Contributes to the Pathogenesis of Autoimmune Hepatitis through Inducing Hepatic Interleukin-6 Expression , 2011, PloS one.
[4] F. Sánchez‐Madrid,et al. Increased circulating pro-inflammatory cytokines and Th17 lymphocytes in Hashimoto's thyroiditis. , 2010, The Journal of clinical endocrinology and metabolism.
[5] M. Talor,et al. Environmental triggers of autoimmune thyroiditis. , 2009, Journal of autoimmunity.
[6] K. Eguchi,et al. T helper type 17 immune response plays an indispensable role for development of iodine-induced autoimmune thyroiditis in nonobese diabetic-H2h4 mice. , 2009, Endocrinology.
[7] F. Shi,et al. CCL2 recruitment of IL‐6‐producing CD11b+ monocytes to the draining lymph nodes during the initiation of Th17‐dependent B cell‐mediated autoimmunity , 2008, European journal of immunology.
[8] A. Yoshimura,et al. Foxp3 Inhibits RORγt-mediated IL-17A mRNA Transcription through Direct Interaction with RORγt*♦ , 2008, Journal of Biological Chemistry.
[9] V. Kuchroo,et al. Induction and effector functions of TH17 cells , 2008, Nature.
[10] Chen Dong,et al. T helper 17 lineage differentiation is programmed by orphan nuclear receptors ROR alpha and ROR gamma. , 2008, Immunity.
[11] A. Paradowska,et al. The function of interleukin 17 in the pathogenesis of rheumatoid arthritis , 2007, Archivum Immunologiae et Therapiae Experimentalis.
[12] I. Horie,et al. CD4+CD25+ naturally occurring regulatory T cells and not lymphopenia play a role in the pathogenesis of iodide-induced autoimmune thyroiditis in NOD-H2h4 mice. , 2007, Journal of autoimmunity.
[13] D. Levy,et al. IL-6 programs TH-17 cell differentiation by promoting sequential engagement of the IL-21 and IL-23 pathways , 2007, Nature Immunology.
[14] R. Nussenblatt,et al. TH17 cells contribute to uveitis and scleritis and are expanded by IL-2 and inhibited by IL-27/STAT1 , 2007, Nature Medicine.
[15] L. Hennighausen,et al. Interleukin-2 signaling via STAT5 constrains T helper 17 cell generation. , 2007, Immunity.
[16] G. Erdogan,et al. High prevalence of thyroid dysfunction and autoimmune thyroiditis in adolescents after elimination of iodine deficiency in the Eastern Black Sea Region of Turkey. , 2006, Thyroid : official journal of the American Thyroid Association.
[17] D. G. Zisoulis,et al. T-bet is a critical determinant in the instability of the IL-17-secreting T-helper phenotype. , 2006, Blood.
[18] Yushu Li,et al. Effect of iodine intake on thyroid diseases in China. , 2006, The New England journal of medicine.
[19] H. Weiner,et al. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells , 2006, Nature.
[20] Ying Wang,et al. A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17 , 2005, Nature Immunology.
[21] N. Rose,et al. Kinetics of mononuclear cell infiltration and cytokine expression in iodine-induced thyroiditis in the NOD-H2h4 mouse. , 2003, Experimental and molecular pathology.
[22] G. Sharp,et al. Dual Roles for IFN-γ, But Not for IL-4, in Spontaneous Autoimmune Thyroiditis in NOD.H-2h4 Mice1 , 2002, The Journal of Immunology.
[23] H. Yagita,et al. Characteristics of inflammatory cells in spontaneous autoimmune thyroiditis of NOD.H-2h4 mice. , 2001, Journal of autoimmunity.
[24] G. Sharp,et al. Spontaneous autoimmune thyroiditis in NOD.H-2h4 mice. , 1999, Journal of autoimmunity.
[25] N. Rose,et al. Iodine-induced autoimmune thyroiditis in NOD-H-2h4 mice. , 1996, Clinical immunology and immunopathology.
[26] N. Bagchi,et al. Thyroid cell injury is an initial event in the induction of autoimmune thyroiditis by iodine in obese strain chickens. , 1995, Endocrinology.
[27] L. Braverman,et al. Werner and Ingbar's the Thyroid: A Fundamental and Clinical Text , 1991 .
[28] L. Braverman,et al. The effect of iodide ingestion on the development of spontaneous lymphocytic thyroiditis in the diabetes-prone BB/W rat. , 1986, Endocrinology.
[29] S. Werner. The Thyroid A Fundamental and Clinical Text , 1957 .
[30] F. Trimarchi,et al. Serum interleukin-23 (IL-23) is increased in Hashimoto's thyroiditis. , 2014, Endocrine journal.
[31] An Xin-rong. Correlation between Treg/Th17 cells imbalance and autoimmunity in Hashimoto thyroiditis , 2012 .
[32] M. Schott,et al. Induction of Autoimmune Thyroiditis by Depletion of CD4+CD25+ Regulatory T cells in Thyroiditis-Resistant IL-17, But Not Interferon-γ Receptor, Knockout Nonobese Diabetic-H2h4 Mice , 2012 .
[33] T. Jie. The change of Th17 lymphocytes and cytokines in autoimmune thyroid diseases , 2011 .
[34] M. Schott. T Helper Type 17 Immune Response Plays an Indispensable Role for Development of Iodine-Induced Autoimmune Thyroiditis in Nonobese Diabetic-H2h4 Mice , 2010 .