The effect of surgical lesions to frontal cortex and substantia nigra on amphetamine responses in rats.

[1]  U. Ungerstedt.  Adipsia and aphagia after 6-hydroxydopamine induced degeneration of the nigro-striatal dopamine system. , 1971, Acta physiologica Scandinavica. Supplementum.

[2]  S. Iversen,et al.  Effects of substantia nigra lesions on the locomotor and stereotypy responses to amphetamine. , 1971, Nature: New biology.

[3]  S. Iversen,et al.  Enhanced amphetamine responses after frontal cortex lesions in the rat. , 1971, European journal of pharmacology.

[4]  L. Iversen,et al.  Regional effects of 6-hydroxydopamine on catecholamine containing neurones in rat brain and spinal cord. , 1970, Brain research.

[5]  J. Stolk,et al.  Antagonism of D-amphetamine by alpha-methyl-L-tyrosine: behavioral evidence for the participation of catecholamine stores and synthesis in the amphetamine stimulant response. , 1970, Neuropharmacology.

[6]  N. Uretsky,et al.  EFFECTS OF 6‐HYDROXYDOPAMINE ON CATECHOLAMINE CONTAINING NEURONES IN THE RAT BRAIN , 1970, Journal of neurochemistry.

[7]  U. Ungerstedt,et al.  Direct chemical stimulation of dopaminergic mechanisms in the neostriatum of the rat. , 1969, Brain research.

[8]  W. Isaac,et al.  Frontal lesions and illumination effects upon the activity of the albino rat , 1969 .

[9]  K. E. Moore,et al.  Norepinephrine: Release from Brain by d-Amphetamine in vivo , 1969, Science.

[10]  R. Faull,et al.  Changes in dopamine levels in the corpus striatum following lesions in the substantia nigra. , 1969, Experimental neurology.

[11]  B. Campbell,et al.  Potentiation of behavioral arousal after cortical damage and subsequent recovery. , 1969, Experimental neurology.

[12]  G. Mckenzie,et al.  The effect of dihydroxyphenylalanine, pheniprazine and dextroamphetamine on the in vivo release of dopamine from the caudate nucleus. , 1968, The Journal of pharmacology and experimental therapeutics.

[13]  H. Pakkenberg,et al.  Aminergic Systems in Basal Ganglia Associated with Stereotyped Hyperactive Behavior and Catalepsy , 1968 .

[14]  G. Bouvier,et al.  Effect of brain lesions on striatal monoamines in the cat. , 1967, Archives of neurology.

[15]  M. Kamback.  The effect of prefrontal lesions and food deprivation on response for stimulus change. , 1967, Experimental neurology.

[16]  N. Andén,et al.  A quantitative study on the nigro-neostriatal dopamine neuron system in the rat. , 1966, Acta physiologica Scandinavica.

[17]  H. C. Nielson,et al.  Effect of frontal pole ablation on biogenic amine levels in the brain. , 1966, Experimental neurology.

[18]  S. Tenen,et al.  Antiamphetamine effects following inhibition of tyrosine hydroxylase. , 1966, The Journal of pharmacology and experimental therapeutics.

[19]  Charles G. Gross,et al.  CAUDATE, CORTICAL, HIPPOCAMPAL AND DORSAL THALAMIC LESIONS IN RATS: ALTERNATION AND HEBB-WILLIAMS MAZE PERFORMANCE , 1965 .

[20]  W. Cowan,et al.  THE ORGANIZATION OF CORTICO-STRIATE CONNEXIONSIN THE RABBIT. , 1963, Brain : a journal of neurology.

[21]  Webster Ke.  Cortico-striate interrelations in the albino rat. , 1961 .

[22]  J. Rossum.  Mode of action of psychomotor stimulant drugs. , 1970 .

[23]  P. Bradley.  Synaptic transmission in the central nervous system and its relevance for drug action. , 1968, International review of neurobiology.

[24]  K. Fuxe,et al.  EVIDENCE FOR THE EXISTENCE OF MONOAMINE-CONTAINING NEURONS IN THE CENTRAL NERVOUS SYSTEM. I. DEMONSTRATION OF MONOAMINES IN THE CELL BODIES OF BRAIN STEM NEURONS. , 1964, Acta physiologica Scandinavica. Supplementum.

[25]  W. Krieg.  Accurate placement of minute lesions in the brain of the albino rat. , 1946, Quarterly bulletin. Northwestern University (Evanston, Ill.). Medical School.