MicroRNA Expression in KRAS- and BRAF-mutated Colorectal Cancers.
暂无分享,去创建一个
R. Palmqvist | M. L. Wikberg | S. Edin | Robin Myte | I. Ljuslinder | Carl Zingmark | Anna Löfgren-Burström | Xingru Li | Ida V. Lundberg | C. Zingmark | Maria L. Wikberg
[1] Jingyuan Fang,et al. The Structure and Clinical Roles of MicroRNA in Colorectal Cancer , 2016, Gastroenterology research and practice.
[2] Shung-Haur Yang,et al. Mutation spectra of RAS gene family in colorectal cancer. , 2016, American journal of surgery.
[3] F. Sinicrope,et al. Molecular Biomarkers in the Personalized Treatment of Colorectal Cancer. , 2016, Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association.
[4] Y. Choi,et al. MicroRNA Expression Signatures Associated With BRAF-Mutated Versus KRAS-Mutated Colorectal Cancers , 2016, Medicine.
[5] R. Wolff,et al. Colorectal tumor molecular phenotype and miRNA: expression profiles and prognosis , 2016, Modern Pathology.
[6] G. Storm,et al. MicroRNA Targeting to Modulate Tumor Microenvironment , 2016, Front. Oncol..
[7] T. Hu,et al. Association of a let-7 KRAS rs712 polymorphism with the risk of breast cancer. , 2015, Genetics and molecular research : GMR.
[8] J. Chen,et al. Multiple Roles of MicroRNA-100 in Human Cancer and its Therapeutic Potential , 2015, Cellular Physiology and Biochemistry.
[9] R. Palmqvist,et al. The infiltration, and prognostic importance, of Th1 lymphocytes vary in molecular subgroups of colorectal cancer , 2015, The journal of pathology. Clinical research.
[10] Chuanhua Cao,et al. Diverse functions of miR-373 in cancer , 2015, Journal of Translational Medicine.
[11] Xiao-jun Liu,et al. MicroRNA-100 functions as a tumor suppressor by inhibiting Lgr5 expression in colon cancer cells. , 2015, Molecular medicine reports.
[12] Zhaoxia Wang,et al. Potential role of miR-100 in cancer diagnosis, prognosis, and therapy , 2015, Tumor Biology.
[13] A. Jemal,et al. Global cancer statistics, 2012 , 2015, CA: a cancer journal for clinicians.
[14] Tao Xi,et al. miR-31 promotes proliferation of colon cancer cells by targeting E2F2 , 2015, Biotechnology Letters.
[15] Hiromu Suzuki,et al. MicroRNA‐31 expression in relation to BRAF mutation, CpG island methylation and colorectal continuum in serrated lesions , 2014, International journal of cancer.
[16] F. Kisseljov. MicroRNAs and cancer , 2014, Molecular Biology.
[17] Hiromu Suzuki,et al. Association of microRNA-31 with BRAF mutation, colorectal cancer survival and serrated pathway. , 2014, Carcinogenesis.
[18] Shuang Wang,et al. Elevated MicroRNA-31 Expression Regulates Colorectal Cancer Progression by Repressing Its Target Gene SATB2 , 2013, PloS one.
[19] Reiko Nishihara,et al. Microsatellite instability and BRAF mutation testing in colorectal cancer prognostication. , 2013, Journal of the National Cancer Institute.
[20] R. Palmqvist,et al. The prognostic role of KRAS, BRAF, PIK3CA and PTEN in colorectal cancer , 2013, British Journal of Cancer.
[21] Aung Ko Win,et al. KRAS-mutation status in relation to colorectal cancer survival: the joint impact of correlated tumour markers , 2013, British Journal of Cancer.
[22] Albert C. Koong,et al. BRAF-mutated, Microsatellite-stable Adenocarcinoma of the Proximal Colon: An Aggressive Adenocarcinoma With Poor Survival, Mucinous Differentiation, and Adverse Morphologic Features , 2012, The American journal of surgical pathology.
[23] A. Duval,et al. MiRNA Genes Constitute New Targets for Microsatellite Instability in Colorectal Cancer , 2012, PloS one.
[24] O. Yokosuka,et al. Epigenetic silencing of microRNA-373 plays an important role in regulating cell proliferation in colon cancer. , 2011, Oncology reports.
[25] E. Van Cutsem,et al. Cetuximab plus irinotecan, fluorouracil, and leucovorin as first-line treatment for metastatic colorectal cancer: updated analysis of overall survival according to tumor KRAS and BRAF mutation status. , 2011, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[26] R. Palmqvist,et al. Colorectal cancer prognosis depends on T-cell infiltration and molecular characteristics of the tumor , 2011, Modern Pathology.
[27] R. Wolff,et al. MicroRNAs and colon and rectal cancer: Differential expression by tumor location and subtype , 2011, Genes, chromosomes & cancer.
[28] A. Krasinskas. EGFR Signaling in Colorectal Carcinoma , 2011, Pathology research international.
[29] R. Palmqvist,et al. The Role of the CpG Island Methylator Phenotype in Colorectal Cancer Prognosis Depends on Microsatellite Instability Screening Status , 2010, Clinical Cancer Research.
[30] J. Neumann,et al. Frequency and type of KRAS mutations in routine diagnostic analysis of metastatic colorectal cancer. , 2009, Pathology, research and practice.
[31] J. Barrett,et al. KRAS and BRAF mutations in advanced colorectal cancer are associated with poor prognosis but do not preclude benefit from oxaliplatin or irinotecan: results from the MRC FOCUS trial. , 2009, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[32] Jian-ming Li,et al. Down‐regulated expression of SATB2 is associated with metastasis and poor prognosis in colorectal cancer , 2009, The Journal of pathology.
[33] C. Croce,et al. Pathogenetic and clinical relevance of microRNAs in colorectal cancer. , 2009, Cancer genomics & proteomics.
[34] V. Heinemann,et al. Clinical relevance of EGFR- and KRAS-status in colorectal cancer patients treated with monoclonal antibodies directed against the EGFR. , 2009, Cancer treatment reviews.
[35] M. Loda,et al. CpG island methylator phenotype-low (CIMP-low) in colorectal cancer: possible associations with male sex and KRAS mutations. , 2006, The Journal of molecular diagnostics : JMD.
[36] P. Laird,et al. CpG island methylator phenotype underlies sporadic microsatellite instability and is tightly associated with BRAF mutation in colorectal cancer , 2006, Nature Genetics.
[37] S. Kakar,et al. BRAF mutation, CpG island methylator phenotype and microsatellite instability occur more frequently and concordantly in mucinous than non‐mucinous colorectal cancer , 2006, International journal of cancer.
[38] Y. Akao,et al. let-7 microRNA functions as a potential growth suppressor in human colon cancer cells. , 2006, Biological & pharmaceutical bulletin.
[39] F. Slack,et al. RAS Is Regulated by the let-7 MicroRNA Family , 2005, Cell.
[40] V. Ambros. The functions of animal microRNAs , 2004, Nature.
[41] Armando Santoro,et al. Cetuximab monotherapy and cetuximab plus irinotecan in irinotecan-refractory metastatic colorectal cancer. , 2004, The New England journal of medicine.
[42] Y. Yatabe,et al. Reduced Expression of the let-7 MicroRNAs in Human Lung Cancers in Association with Shortened Postoperative Survival , 2004, Cancer Research.
[43] R. Palmqvist,et al. SOX 2 Expression Is Regulated by BRAF and Contributes to Poor Patient Prognosis in Colorectal Cancer , 2017 .
[44] M. Kretz,et al. Non-coding RNAs: Classification, Biology and Functioning. , 2016, Advances in experimental medicine and biology.
[45] A. Adjei,et al. The Ras/Raf/MAPK pathway. , 2006, Journal of thoracic oncology : official publication of the International Association for the Study of Lung Cancer.
[46] A. Jemal,et al. Global cancer statistics , 2011, CA: a cancer journal for clinicians.
[47] B. Vogelstein,et al. Prevalence of ras gene mutations in human colorectal cancers , 1987, Nature.