RNase L and Double-Stranded RNA-Dependent Protein Kinase Exert Complementary Roles in Islet Cell Defense during Coxsackievirus Infection1
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Monica Hultcrantz | Robert Silverman | R. Silverman | N. Sarvetnick | M. Hultcrantz | M. Flodström-Tullberg | C. Fine | Malin Flodström-Tullberg | Alexandr Stotland | Amy Maday | Devin Tsai | Cody Fine | Bryan Williams | Nora Sarvetnick | B. Williams | A. Stotland | A. Maday | Devin Tsai | Amy Maday
[1] R. Waldherr,et al. [Myocarditis and insulitis following coxsackie virus infection]. , 1976, Zeitschrift fur Kardiologie.
[2] T. Onodera,et al. Isolation of a virus from the pancreas of a child with diabetic ketoacidosis. , 1979, The New England journal of medicine.
[3] A. Notkins,et al. PANCREATIC ISLET-CELL DAMAGE IN CHILDREN WITH FATAL VIRAL INFECTIONS , 1980, The Lancet.
[4] T. Onodera,et al. Virus-induced diabetes mellitus. XVIII. Inhibition by a nondiabetogenic variant of encephalomyocarditis virus , 1980, The Journal of experimental medicine.
[5] P. Hartig,et al. Diversity within a human isolate of coxsackie B4: Relationship to viral‐induced diabetes , 1983, Journal of medical virology.
[6] Silverman,et al. rRNA cleavage as an index of ppp(A2'p)nA activity in interferon-treated encephalomyocarditis virus-infected cells , 1983, Journal of virology.
[7] A. Torres,et al. Coxsackie virus: a review. , 1984, Boletin de la Asociacion Medica de Puerto Rico.
[8] I. Gresser,et al. Role of sex and early interferon production in the susceptibility of mice to encephalomyocarditis virus. , 1985, The Journal of general virology.
[9] D. R. Gamble,et al. Coxsackie B4 virus induces short‐term changes in the metabolic functions of mouse pancreatic islets in vitro , 1986, Cell biochemistry and function.
[10] M. Vigneron,et al. Constitutive expression of (2′–5′) oligo A synthetase confers resistance to picornavirus infection , 1987, Nature.
[11] T. M. Szopa,et al. In Vivo Infection of Mice with Coxsackie B4 Virus Induces Long‐term Functional Changes in Pancreatic Islets with Minimal Alteration in Blood Glucose , 1989, Diabetic medicine : a journal of the British Diabetic Association.
[12] T. Urano,et al. Outbreak of herpangina associated with coxsackievirus B3 infection , 1989, The Pediatric infectious disease journal.
[13] J. Petersen,et al. Interferon stimulates the expression of 2',5'-oligoadenylate synthetase and MHC class I antigens in insulin-producing cells. , 1991, Journal of interferon research.
[14] H. Festenstein,et al. Coxsackievirus B4 infection of the mouse pancreas: the role of natural killer cells in the control of virus replication and resistance to infection. , 1992, The Journal of general virology.
[15] G. Frisk,et al. Impaired Ca2+ response to glucose in mouse beta-cells infected with coxsackie B or Echo virus. , 1994, Virus research.
[16] A. Aguzzi,et al. Deficient signaling in mice devoid of double‐stranded RNA‐dependent protein kinase. , 1995, The EMBO journal.
[17] T. Dyrberg,et al. Differential Responsiveness to Interferon-α in β-Cells and Non-β-Cells , 1996, Diabetes.
[18] M. Dalakas,et al. A Study of the Interferon Antiviral Mechanism: Apoptosis Activation by the 2–5A System , 1997, The Journal of experimental medicine.
[19] R. Silverman,et al. Interferon action and apoptosis are defective in mice devoid of 2′,5′‐oligoadenylate‐dependent RNase L , 1997, The EMBO journal.
[20] D. Eizirik,et al. Interferon-γ-Induced Interferon Regulatory Factor-1 (IRF-1) Expression in Rodent and Human Islet Cells Precedes Nitric Oxide Production. , 1997, Endocrinology.
[21] Troy Krahl,et al. Diabetes induced by Coxsackie virus: Initiation by bystander damage and not molecular mimicry , 1998, Nature Medicine.
[22] G. Kochs,et al. Mx proteins: mediators of innate resistance to RNA viruses. , 1998, Revue scientifique et technique.
[23] Mark R. Player,et al. The 2–5 A system: Modulation of viral and cellular processes through acceleration of RNA degradation , 1998, Pharmacology & Therapeutics.
[24] R. Silverman,et al. Impact of RNase L overexpression on viral and cellular growth and death. , 1998, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[25] D. Eizirik,et al. Exposure of human islets to cytokines can result in disproportionately elevated proinsulin release. , 1999, The Journal of clinical investigation.
[26] M. Knip,et al. Environmental factors in the pathogenesis of type 1 diabetes mellitus. , 2009, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.
[27] B. Williams,et al. Interferon action in triply deficient mice reveals the existence of alternative antiviral pathways. , 1999, Virology.
[28] L. A. Stephens,et al. Tumor Necrosis Factor-α-Activated Cell Death Pathways in NIT-1 Insulinoma Cells and Primary Pancreatic β Cells. , 1999, Endocrinology.
[29] M. Dallman,et al. Impaired Antiviral Response and Alpha/Beta Interferon Induction in Mice Lacking Beta Interferon , 2000, Journal of Virology.
[30] N. Sarvetnick,et al. Pancreatic expression of interferon-γ protects mice from lethal coxsackievirus B3 infection and subsequent myocarditis , 2000, Nature Medicine.
[31] L. Bouwens,et al. Mechanisms of coxsackievirus-induced damage to human pancreatic beta-cells. , 2000, The Journal of clinical endocrinology and metabolism.
[32] C. Bogdan. The function of type I interferons in antimicrobial immunity. , 2000, Current opinion in immunology.
[33] Xiao-Ling Li,et al. RNase-L-dependent Destabilization of Interferon-induced mRNAs , 2000, The Journal of Biological Chemistry.
[34] F. Pattou,et al. Persistent Infection of Human Pancreatic Islets by Coxsackievirus B Is Associated with Alpha Interferon Synthesis in β Cells , 2000, Journal of Virology.
[35] K. Klingel,et al. Cardioselective Infection With Coxsackievirus B3 Requires Intact Type I Interferon Signaling: Implications for Mortality and Early Viral Replication , 2001, Circulation.
[36] N. Sarvetnick,et al. A critical role for inducible nitric oxide synthase in host survival following coxsackievirus B4 infection. , 2001, Virology.
[37] R. Silverman,et al. Increased severity of HSV-1 keratitis and mortality in mice lacking the 2-5A-dependent RNase L gene. , 2001, Investigative ophthalmology & visual science.
[38] G. Barber. Host defense, viruses and apoptosis , 2001, Cell Death and Differentiation.
[39] J. Corbett,et al. Double-stranded RNA-dependent protein kinase is not required for double-stranded RNA-induced nitric oxide synthase expression or nuclear factor-kappaB activation by islets. , 2001, Diabetes.
[40] D. Scheuner,et al. Mechanisms of β-cell death in response to double-stranded (ds) RNA and interferon-γ dsrna-dependent protein kinase apoptosis and nitric oxide-dependent necrosis , 2001 .
[41] C. Samuel,et al. Antiviral Actions of Interferons , 2001, Clinical Microbiology Reviews.
[42] J. Galama,et al. Functional impairment and killing of human beta cells by enteroviruses: the capacity is shared by a wide range of serotypes, but the extent is a characteristic of individual virus strains , 2002, Diabetologia.
[43] David A. Chesler,et al. The role of IFN-γ in immune responses to viral infections of the central nervous system , 2002 .
[44] R. Lockey,et al. 2′-5′ Oligoadenylate Synthetase Plays a Critical Role in Interferon-γ Inhibition of Respiratory Syncytial Virus Infection of Human Epithelial Cells* , 2002, The Journal of Biological Chemistry.
[45] N. Sarvetnick,et al. Target cell defense prevents the development of diabetes after viral infection , 2002, Nature Immunology.
[46] E. Jaeckel,et al. Viruses and Diabetes , 2002, Annals of the New York Academy of Sciences.
[47] W. Merrick,et al. Viral Stress-inducible Protein p56 Inhibits Translation by Blocking the Interaction of eIF3 with the Ternary Complex eIF2·GTP·Met-tRNAi* , 2003, Journal of Biological Chemistry.
[48] M. Weitzman,et al. The suppressor of cytokine signaling-1 (SOCS1) is a novel therapeutic target for enterovirus-induced cardiac injury. , 2003, The Journal of clinical investigation.
[49] B. Williams,et al. The murine double-stranded RNA-dependent protein kinase PKR and the murine 2',5'-oligoadenylate synthetase-dependent RNase L are required for IFN-beta-mediated resistance against herpes simplex virus type 1 in primary trigeminal ganglion culture. , 2003, Virology.
[50] G. Barber,et al. The dsRNA binding protein family: critical roles, diverse cellular functions , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[51] N. Sarvetnick,et al. Diabetogenic potential of human pathogens uncovered in experimentally permissive beta-cells. , 2003, Diabetes.
[52] B. Williams,et al. ISG20, a New Interferon-induced RNase Specific for Single-stranded RNA, Defines an Alternative Antiviral Pathway against RNA Genomic Viruses* , 2003, The Journal of Biological Chemistry.
[53] K. Klingel,et al. Enterovirus infection in human pancreatic islet cells, islet tropism in vivo and receptor involvement in cultured islet beta cells , 2004, Diabetologia.
[54] M. Flodström-Tullberg. Viral infections: their elusive role in regulating susceptibility to autoimmune disease. , 2003, Microbes and infection.
[55] B. Williams,et al. Apoptosis and interferons: Role of interferon-stimulated genes as mediators of apoptosis , 2003, Apoptosis.
[56] A. Notkins. Virus-induced diabetes mellitus , 2005, Archives of Virology.