Persistent binding of ligands to the aryl hydrocarbon receptor.
暂无分享,去创建一个
[1] G. Perdew,et al. The role of chaperone proteins in the aryl hydrocarbon receptor core complex. , 2002, Chemico-biological interactions.
[2] M. Denison,et al. Synthesis and estrogen receptor affinity of a 4-hydroxytamoxifen-labeled ligand for diagnostic imaging. , 2002, Bioorganic & medicinal chemistry.
[3] B. Moorthy. Persistent expression of 3-methylcholanthrene-inducible cytochromes P4501A in rat hepatic and extrahepatic tissues. , 2000, The Journal of pharmacology and experimental therapeutics.
[4] R. Pollenz. The aryl-hydrocarbon receptor, but not the aryl-hydrocarbon receptor nuclear translocator protein, is rapidly depleted in hepatic and nonhepatic culture cells exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin. , 1996, Molecular pharmacology.
[5] T. Ogle. Characterization of progesterone binding to nuclear receptors in rat placenta. , 1986, Journal of steroid biochemistry.
[6] M. Denison,et al. Activation of the aryl hydrocarbon receptor by structurally diverse exogenous and endogenous chemicals. , 2003, Annual review of pharmacology and toxicology.
[7] A Poland,et al. 2,3,7,8-tetrachlorodibenzo-p-dioxin and related halogenated aromatic hydrocarbons: examination of the mechanism of toxicity. , 1982, Annual review of pharmacology and toxicology.
[8] C. Bradfield,et al. Ah receptor signaling pathways. , 1996, Annual review of cell and developmental biology.
[9] A. Goldstein,et al. Principles of Drug Action. The Basis of Pharmacology , 1970 .
[10] M. Maines. Current protocols in toxicology , 1999 .
[11] T. Gasiewicz,et al. Transformation of the aryl hydrocarbon receptor to a DNA-binding form is accompanied by release of the 90 kDa heat-shock protein and increased affinity for 2,3,7,8-tetrachlorodibenzo-p-dioxin. , 1993, The Biochemical journal.
[12] S. Safe,et al. Absence of positive co-operativity in the binding of 2,3,7,8-tetrachlorodibenzo-p-dioxin to its cytosolic receptor protein. , 1987, The Biochemical journal.
[13] W. Meylan,et al. Atom/fragment contribution method for estimating octanol-water partition coefficients. , 1995, Journal of pharmaceutical sciences.
[14] M. Ingelman-Sundberg,et al. Different structural requirements of the ligand binding domain of the aryl hydrocarbon receptor for high- and low-affinity ligand binding and receptor activation. , 2004, Molecular pharmacology.
[15] Y Fujii-Kuriyama,et al. Dioxin binding activities of polymorphic forms of mouse and human arylhydrocarbon receptors. , 1994, The Journal of biological chemistry.
[16] S. Safe,et al. Synthesis and aryl hydrocarbon receptor binding properties of radiolabeled polychlorinated dibenzofuran congeners. , 1987, Archives of biochemistry and biophysics.
[17] R. D. Di Giulio,et al. Effects of the polycyclic aromatic hydrocarbon heterocycles, carbazole and dibenzothiophene, on in vivo and in vitro cypia activity and polycyclic aromatic hydrocarbon‐derived embryonic deformities , 2005, Environmental toxicology and chemistry.
[18] Allan B. Okey,et al. Ah receptor for 2,3,7,8-tetrachlorodibenzo-p-dioxin: Comparative studies in mammalian and nonmammalian species , 1986 .
[19] Michael A. Gallo,et al. Biological Basis for Risk Assessment of Dioxins and Related Compounds , 1991 .
[20] E. J. Dougherty,et al. Redefining the Role of the Endogenous XAP2 and C-terminal hsp70-interacting Protein on the Endogenous Ah Receptors Expressed in Mouse and Rat Cell Lines* , 2005, Journal of Biological Chemistry.
[21] M. Denison,et al. Species-specific binding of transformed Ah receptor to a dioxin responsive transcriptional enhancer. , 1992, European journal of pharmacology.
[22] R. Pollenz,et al. Ligand-dependent and -independent degradation of the human aryl hydrocarbon receptor (hAHR) in cell culture models. , 2006, Chemico-biological interactions.
[23] L. Lennard,et al. Principles of Drug Action. The Basis of Pharmacology , 1991 .
[24] S. Safe,et al. Polychlorinated biphenyls (PCBs), dibenzo-p-dioxins (PCDDs), dibenzofurans (PCDFs), and related compounds: environmental and mechanistic considerations which support the development of toxic equivalency factors (TEFs). , 1990, Critical reviews in toxicology.
[25] M. Denison. Heterogeneity of rat hepatic Ah receptor: identification of two receptor forms which differ in their biochemical properties. , 1992, Journal of Biochemical Toxicology.
[26] M. Denison,et al. The hepatic Ah receptor for 2,3,7,8-tetrachlorodibenzo-p-dioxin: species differences in subunit dissociation. , 1990, Archives of biochemistry and biophysics.
[27] B M Weichman,et al. Estradiol-binding kinetics of the activated and nonactivated estrogen receptor. , 1977, The Journal of biological chemistry.
[28] M. Denison,et al. Structure and function of the Ah receptor for 2,3,7,8-tetrachlorodibenzo-p-dioxin. Species difference in molecular properties of the receptors from mouse and rat hepatic cytosols. , 1986, The Journal of biological chemistry.
[29] B. Katzenellenbogen,et al. Structural requirements for high affinity ligand binding by estrogen receptors: a comparative analysis of truncated and full length estrogen receptors expressed in bacteria, yeast, and mammalian cells. , 1992, Molecular endocrinology.
[30] M. M. Bradford. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.
[31] A. Puga,et al. Molecular biology of the toxic response , 1999 .
[32] E. Glover,et al. Comparison of 2,3,7,8-tetrachlorodibenzo-p-dioxin, a potent inducer of aryl hydrocarbon hydroxylase, with 3-methylcholanthrene. , 1974, Molecular pharmacology.
[33] E. Glover,et al. 2,3,7,8,-Tetrachlorodibenzo-p-dioxin: segregation of toxocity with the Ah locus. , 1980, Molecular pharmacology.
[34] T. Pineau,et al. Ah receptor-dependent CYP1A induction by two carotenoids, canthaxanthin and beta-apo-8'-carotenal, with no affinity for the TCDD binding site. , 1997, Biochemical pharmacology.
[35] Alessandro Pandini,et al. Ligand binding and activation of the Ah receptor. , 2002, Chemico-biological interactions.
[36] C. Bradfield,et al. Kinetic and equilibrium studies of Ah receptor-ligand binding: use of [125I]2-iodo-7,8-dibromodibenzo-p-dioxin. , 1988, Molecular pharmacology.
[37] B. Weichman,et al. The role of ligand-binding as a determinant of the structure and activation of the estrogen receptor. , 1979, Advances in experimental medicine and biology.
[38] N. Ledirac,et al. Cytotoxic effects and induction of cytochromes P450 1A1/2 by insecticides, in hepatic or epidermal cells: binding capability to the Ah receptor. , 1998, Toxicology letters.
[39] McArdle,et al. 2 , 3 , 7 , 8-Tetrachlorodibenzo-p-dioxin : Segregation of Toxicity with the Ah Locus , 2022 .
[40] J. Landers,et al. Kinetic models for association of 2,3,7,8-tetrachlorodibenzo-p-dioxin with the Ah receptor. , 1988, Archives of biochemistry and biophysics.
[41] N. Bunce,et al. Additive binding of polychlorinated biphenyls and 2,3,7,8-tetrachlorodibenzo-p-dioxin to the murine hepatic Ah receptor. , 1994, Toxicology and applied pharmacology.
[42] N. Ledirac,et al. Carbaryl induces CYP1A1 gene expression in HepG2 and HaCaT cells but is not a ligand of the human hepatic Ah receptor. , 1997, Toxicology and applied pharmacology.
[43] J. Bartholomew,et al. Regulation of CYP1A1 by Indolo[3,2-b]carbazole in Murine Hepatoma Cells (*) , 1995, The Journal of Biological Chemistry.
[44] R. Pollenz,et al. Ligand-dependent and independent modulation of aryl hydrocarbon receptor localization, degradation, and gene regulation. , 2002, Molecular pharmacology.
[45] R. A. Butler,et al. An aryl hydrocarbon receptor (AHR) homologue from the soft-shell clam, Mya arenaria: evidence that invertebrate AHR homologues lack 2,3,7,8-tetrachlorodibenzo-p-dioxin and β-naphthoflavone binding , 2001 .
[46] R. Pollenz. The mechanism of AH receptor protein down-regulation (degradation) and its impact on AH receptor-mediated gene regulation. , 2002, Chemico-biological interactions.
[47] Alessandro Pandini,et al. Structural and functional characterization of the aryl hydrocarbon receptor ligand binding domain by homology modeling and mutational analysis. , 2007, Biochemistry.
[48] F. Fontaine,et al. Cytochrome 1A1 induction by primaquine in human hepatocytes and HepG2 cells: absence of binding to the aryl hydrocarbon receptor. , 1999, Biochemical pharmacology.
[49] A. Berghard,et al. The stability of dioxin-receptor ligands influences cytochrome P450IA1 expression in human keratinocytes. , 1992, Carcinogenesis.
[50] H. Bigelow. Woods Hole Oceanographic Institution , 1930, Nature.
[51] M. Denison,et al. Analysis of the Aryl Hydrocarbon Receptor (AhR) Signal Transduction Pathway , 2002, Current protocols in toxicology.
[52] A Tramontano,et al. A model for recognition of polychlorinated dibenzo-p-dioxins by the aryl hydrocarbon receptor. , 2002, European journal of biochemistry.
[53] O. Hankinson. The aryl hydrocarbon receptor complex. , 1995, Annual review of pharmacology and toxicology.
[54] D Mackay,et al. A novel method for measuring membrane-water partition coefficients of hydrophobic organic chemicals: comparison with 1-octanol-water partitioning. , 1988, Journal of Pharmacy and Science.
[55] R. D. Di Giulio,et al. Synergistic Embryotoxicity of Polycyclic Aromatic Hydrocarbon Aryl Hydrocarbon Receptor Agonists with Cytochrome P4501A Inhibitors in Fundulus heteroclitus , 2004, Environmental health perspectives.
[56] A. Rannug,et al. Rapid and transient induction of CYP1A1 gene expression in human cells by the tryptophan photoproduct 6-formylindolo[3,2-b]carbazole. , 1998, Chemico-biological interactions.
[57] G. Perdew,et al. The transactivation domain of the Ah receptor is a key determinant of cellular localization and ligand-independent nucleocytoplasmic shuttling properties. , 2005, Biochemistry.
[58] D. Phelan,et al. Carbaryl, a carbamate insecticide, is a ligand for the hepatic Ah (dioxin) receptor. , 1998, Toxicology and applied pharmacology.
[59] A. Boctor,et al. Analysis of binding of [3H]Estradiol to the cytosol fraction of rat pancreas: comparison with sites in the cytosol of uterus. , 1983, Endocrinology.
[60] T. Gasiewicz,et al. Characterization of the in vitro stability of the rat hepatic receptor for 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). , 1987, Archives of biochemistry and biophysics.
[61] P. Harper,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin versus 3-methylcholanthrene: comparative studies of Ah receptor binding, transformation, and induction of CYP1A1. , 1994, The Journal of biological chemistry.
[62] J. Whitlock,et al. Induction of cytochrome P4501A1. , 1999, Annual review of pharmacology and toxicology.
[63] P. Galtier,et al. Evidence for the ligand-independent activation of the AH receptor. , 1995, Biochemical and biophysical research communications.
[64] G. Perdew,et al. Use of 2-azido-3-[125I]iodo-7,8-dibromodibenzo-p-dioxin as a probe to determine the relative ligand affinity of human versus mouse aryl hydrocarbon receptor in cultured cells. , 2004, Molecular pharmacology.