DNA lesions and repair.

[1]  H. Kampinga,et al.  Selective inhibition of repair of active genes by hyperthermia is due to inhibition of global and transcription coupled repair pathways. , 1995, Carcinogenesis.

[2]  M. Weinfeld,et al.  The action of Escherichia coli endonuclease III on multiply damaged sites in DNA. , 1995, Journal of molecular biology.

[3]  S. Thode,et al.  Mechanisms of overlap formation in nonhomologous DNA end joining , 1994, Molecular and cellular biology.

[4]  P. Munz,et al.  A modified single-strand annealing model best explains the joining of DNA double-strand breaks mammalian cells and cell extracts. , 1995, Nucleic acids research.

[5]  A. Riggs,et al.  Binding of transcription factors creates hot spots for UV photoproducts in vivo , 1992, Molecular and cellular biology.

[6]  P. Pfeiffer,et al.  Joining of nonhomologous DNA double strand breaks in vitro. , 1988, Nucleic acids research.

[7]  L. Mullenders,et al.  Transcription‐coupled repair removes both cyclobutane pyrimidine dimers and 6‐4 photoproducts with equal efficiency and in a sequential way from transcribed DNA in xeroderma pigmentosum group C fibroblasts. , 1995, The EMBO journal.

[8]  J. Thacker,et al.  The rejoining of double-strand breaks in DNA by human cell extracts. , 1990, Nucleic acids research.

[9]  W. Thilly,et al.  Resolution and conservation of mismatches in DNA end joining. , 1994, Mutagenesis.

[10]  C. Young,et al.  Characterization of DNA end joining in a mammalian cell nuclear extract: junction formation is accompanied by nucleotide loss, which is limited and uniform but not site specific , 1994, Molecular and cellular biology.

[11]  Jack W. Szostak,et al.  The double-strand-break repair model for recombination , 1983, Cell.

[12]  D. Weaver,et al.  What to do at an end: DNA double-strand-break repair. , 1995, Trends in genetics : TIG.

[13]  M. Meuth,et al.  DNA sequence determination of γ-radiation-induced mutations of the hamster aprt locus , 1989 .

[14]  D. Roth,et al.  Mechanisms of nonhomologous recombination in mammalian cells , 1985, Molecular and cellular biology.

[15]  K. Prise,et al.  Evidence for induction of DNA double-strand breaks at paired radical sites. , 1993, Radiation research.

[16]  D. Roth,et al.  Nonhomologous recombination in mammalian cells: role for short sequence homologies in the joining reaction , 1986, Molecular and cellular biology.

[17]  J. Ward,et al.  The difference that linear energy transfer makes to precursors of DNA strand breaks. , 1996, Radiation research.

[18]  P. Cooper,et al.  Preferential repair of ionizing radiation-induced damage in the transcribed strand of an active human gene is defective in Cockayne syndrome. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[19]  J. Thacker,et al.  A mechanism for deletion formation in DNA by human cell extracts: the involvement of short sequence repeats. , 1992, Nucleic acids research.

[20]  W. Goedecke,et al.  Nonhomologous DNA end joining of synthetic hairpin substrates in Xenopus laevis egg extracts. , 1994, Nucleic acids research.

[21]  J. Ward,et al.  DNA damage produced by ionizing radiation in mammalian cells: identities, mechanisms of formation, and reparability. , 1988, Progress in nucleic acid research and molecular biology.

[22]  M. Löbrich,et al.  DNA double-strand breaks induced by high-energy neon and iron ions in human fibroblasts. II. Probing individual notI fragments by hybridization. , 1994, Radiation research.

[23]  J. Ward,et al.  Mammalian cells are not killed by DNA single-strand breaks caused by hydroxyl radicals from hydrogen peroxide. , 1985, Radiation research.

[24]  P. Jeggo,et al.  DNA double-strand break repair and V(D)J recombination: involvement of DNA-PK. , 1995, Trends in biochemical sciences.

[25]  J. Ward Radiation mutagenesis: the initial DNA lesions responsible. , 1995, Radiation research.

[26]  M. Clemens,et al.  Homologous and illegitimate recombination in developing Xenopus oocytes and eggs , 1993, Molecular and cellular biology.

[27]  W. Morgan,et al.  Spectrum of mutations produced by specific types of restriction enzyme-induced double-strand breaks. , 1992, Mutagenesis.

[28]  G. Iliakis,et al.  Effects of hyperthermia on chromatin condensation and nucleoli disintegration as visualized by induction of premature chromosome condensation in interphase mammalian cells. , 1989, Cancer research.

[29]  E. Feldmann,et al.  A novel nuclease activity from Xenopus laevis releases short oligomers from 5′‐ends of double‐and single‐stranded DNA , 1996, Genes to cells : devoted to molecular & cellular mechanisms.

[30]  S. Thode,et al.  A novel pathway of DNA end-to-end joining , 1990, Cell.

[31]  R. J. Preston,et al.  Commentary to Thacker: A Consideration of the Mechanisms of Induction of Mutations in Mammalian Cells by Low Doses and Dose Rates of Ionizing Radiation , 1992 .

[32]  J A Aguilera,et al.  Variation of single-strand break yield with scavenger concentration for plasmid DNA irradiated in aqueous solution. , 1993, Radiation research.

[33]  A. Natarajan,et al.  Distribution of X-ray-induced G2 chromatid damage among Chinese hamster chromosomes: influence of chromatin conformation. , 1994, Mutation research.

[34]  P. Smith n-Butyrate alters chromatin accessibility to DNA repair enzymes. , 1986, Carcinogenesis.

[35]  J. Thacker,et al.  Multiple components are involved in the efficient joining of double stranded DNA breaks in human cell extracts. , 1992, Nucleic acids research.

[36]  M. Smerdon,et al.  Enhanced DNA repair synthesis in hyperacetylated nucleosomes. , 1989, The Journal of biological chemistry.

[37]  J. Ward,et al.  DNA repair by thiols in air shows two radicals make a double-strand break. , 1995, Radiation research.

[38]  B. Vojnovic,et al.  Measurement of DNA damage by electrons with energies between 25 and 4000 eV. , 1993, International journal of radiation biology.

[39]  A. F. Fuciarelli,et al.  Radiation damage in DNA : structure/function relationships at early times , 1996 .