HDAC4 degradation by combined TRAIL and valproic acid treatment induces apoptotic cell death of TRAIL-resistant head and neck cancer cells
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Yong-Sung Kim | Ho-Ryun Won | Y. Kim | Chul-Ho Kim | Bok-Soon Lee | Haeng-Jun Kim | D. Kim
[1] Yue Meng,et al. Gefitinib enhances human colon cancer cells to TRAIL-induced apoptosis of via autophagy- and JNK-mediated death receptors upregulation , 2016, Apoptosis.
[2] B. Prabhakar,et al. Targeting the metabolic pathway of human colon cancer overcomes resistance to TRAIL-induced apoptosis , 2016, Cell Death Discovery.
[3] Hui-Yun Wang,et al. Overexpressed HDAC4 is associated with poor survival and promotes tumor progression in esophageal carcinoma , 2016, Aging.
[4] L. Cappabianca,et al. NGF sensitizes TrkA SH-SY5Y neuroblastoma cells to TRAIL-induced apoptosis , 2016, Cell Death Discovery.
[5] Y. S. Shin,et al. Anti-cancer Effect of Luminacin, a Marine Microbial Extract, in Head and Neck Squamous Cell Carcinoma Progression via Autophagic Cell Death , 2015, Cancer research and treatment : official journal of Korean Cancer Association.
[6] D. P. Mishra,et al. Trailing TRAIL Resistance: Novel Targets for TRAIL Sensitization in Cancer Cells , 2015, Front. Oncol..
[7] Weili Zhao,et al. c-FLIP is involved in tumor progression of peripheral T-cell lymphoma and targeted by histone deacetylase inhibitors , 2014, Journal of Hematology & Oncology.
[8] H. Walczak,et al. Getting TRAIL back on track for cancer therapy , 2014, Cell Death and Differentiation.
[9] E. Seto,et al. Erasers of histone acetylation: the histone deacetylase enzymes. , 2014, Cold Spring Harbor perspectives in biology.
[10] K. Ramana,et al. Aldose reductase inhibition enhances TRAIL-induced human colon cancer cell apoptosis through AKT/FOXO3a-dependent upregulation of death receptors. , 2013, Free radical biology & medicine.
[11] Y. S. Shin,et al. AY4, an agonistic anti-death receptor 4 MAB, induces apoptotic cell death in anaplastic thyroid cancer cells via downregulation of Bcl-xL with reactive oxygen species generation. , 2013, Endocrine-related cancer.
[12] Y. S. Shin,et al. An agonistic antibody to human death receptor 4 induces apoptotic cell death in head and neck cancer cells through mitochondrial ROS generation. , 2012, Cancer letters.
[13] Louise Fairall,et al. A Death Effector Domain Chain DISC Model Reveals a Crucial Role for Caspase-8 Chain Assembly in Mediating Apoptotic Cell Death , 2012, Molecular cell.
[14] Ruud H. Brakenhoff,et al. The molecular biology of head and neck cancer , 2011, Nature Reviews Cancer.
[15] D. Dimitrov,et al. Identification and characterization of a novel agonistic anti-DR4 human monoclonal antibody , 2010, mAbs.
[16] Junho Chung,et al. Histone deacetylase inhibitors synergistically potentiate death receptor 4-mediated apoptotic cell death of human T-cell acute lymphoblastic leukemia cells , 2010, Apoptosis.
[17] T. Bayer,et al. Histone Deacetylase Inhibitor Valproic Acid Inhibits Cancer Cell Proliferation via Down-regulation of the Alzheimer Amyloid Precursor Protein* , 2010, The Journal of Biological Chemistry.
[18] D. Saur,et al. HDAC2 attenuates TRAIL-induced apoptosis of pancreatic cancer cells , 2010, Molecular Cancer.
[19] Yong-Sung Kim,et al. A novel agonistic antibody to human death receptor 4 induces apoptotic cell death in various tumor cells without cytotoxicity in hepatocytes , 2009, Molecular Cancer Therapeutics.
[20] W. Westra,et al. Molecular pathology of head and neck cancer: implications for diagnosis, prognosis, and treatment. , 2009, Annual review of pathology.
[21] Minoru Yoshida,et al. 14-3-3 regulates the nuclear import of class IIa histone deacetylases. , 2008, Biochemical and biophysical research communications.
[22] M. Karamouzis,et al. Head and neck cancer , 2008, The Lancet.
[23] Michele Pallaoro,et al. HDACs, histone deacetylation and gene transcription: from molecular biology to cancer therapeutics , 2007, Cell Research.
[24] E. Sausville,et al. Interactive effects of HDAC inhibitors and TRAIL on apoptosis are associated with changes in mitochondrial functions and expressions of cell cycle regulatory genes in multiple myeloma. , 2005, Neoplasia.
[25] K. Harish. Neck dissections: radical to conservative , 2005, World journal of surgical oncology.
[26] Wafik S El-Deiry,et al. TRAIL and apoptosis induction by TNF-family death receptors , 2003, Oncogene.
[27] S. Shankar,et al. Synergistic interactions of chemotherapeutic drugs and tumor necrosis factor-related apoptosis-inducing ligand/Apo-2 ligand on apoptosis and on regression of breast carcinoma in vivo. , 2003, Cancer research.
[28] A. Rustgi,et al. Paclitaxel Induces Prolonged Activation of the Ras/MEK/ERK Pathway Independently of Activating the Programmed Cell Death Machinery* , 2001, The Journal of Biological Chemistry.
[29] A. Rustgi,et al. Paclitaxel induces prolonged activation of the Ras/MEK/ERK pathway independently of activating the programmed cell death machinery. , 2001, The Journal of biological chemistry.
[30] Barry I. Posner,et al. Regulation of Histone Deacetylase 4 by Binding of 14-3-3 Proteins , 2000, Molecular and Cellular Biology.
[31] P. Marks,et al. Histone deacetylase inhibitors: inducers of differentiation or apoptosis of transformed cells. , 2000, Journal of the National Cancer Institute.
[32] Y. Goltsev,et al. Tumor necrosis factor receptor and Fas signaling mechanisms. , 1999, Annual review of immunology.
[33] E. Martz. Radical or conservative? , 1999, Delaware medical journal.
[34] J. Tschopp,et al. TRAIL receptors 1 (DR4) and 2 (DR5) signal FADD-dependent apoptosis and activate NF-kappaB. , 1997, Immunity.