Proximal Giant Neurofilamentous Axonopathy in Mice Genetically Engineered to Resist Calpain and Caspase Cleavage of α-II Spectrin
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M. Iordanov | D. Kretzschmar | M. Lecomte | J. Wentzell | A. L. Ramos | E. Couchi | R. Kassa | D. Tshala-Katumbay | G. Nicolas | V. Monterroso
[1] P. Wong,et al. Upregulation of Dpysl2 and Spna2 gene expression in the rat brain after ischemic stroke , 2009, Neurochemistry International.
[2] D. Tshala-Katumbay,et al. Probing mechanisms of axonopathy. Part II: Protein targets of 2,5-hexanedione, the neurotoxic metabolite of the aliphatic solvent n-hexane. , 2009, Toxicological sciences : an official journal of the Society of Toxicology.
[3] D. Tshala-Katumbay,et al. Probing mechanisms of axonopathy. Part I: Protein targets of 1,2-diacetylbenzene, the neurotoxic metabolite of aromatic solvent 1,2-diethylbenzene. , 2008, Toxicological sciences : an official journal of the Society of Toxicology.
[4] M. Rasband,et al. Spectrin and Ankyrin-Based Cytoskeletons at Polarized Domains in Myelinated Axons , 2008, Experimental biology and medicine.
[5] R. Hayes,et al. Calpain- and caspase-mediated alphaII-spectrin and tau proteolysis in rat cerebrocortical neuronal cultures after ecstasy or methamphetamine exposure. , 2007, The international journal of neuropsychopharmacology.
[6] Y. Colin,et al. A Mutant αII-spectrin Designed to Resist Calpain and Caspase Cleavage Questions the Functional Importance of This Process in Vivo* , 2007, Journal of Biological Chemistry.
[7] T. Steitz,et al. Structure of the Calmodulin αII-Spectrin Complex Provides Insight into the Regulation of Cell Plasticity* , 2006, Journal of Biological Chemistry.
[8] F. Tortella,et al. Comparing calpain- and caspase-3-mediated degradation patterns in traumatic brain injury by differential proteome analysis. , 2006, The Biochemical journal.
[9] T. Boyer,et al. Identification of a short form of ubiquitin-specific protease 3 that is a repressor of rat glutathione S-transferase gene expression. , 2006, The Biochemical journal.
[10] A. Lajtha,et al. Protein content of various regions of rat brain and adult and aging human brain , 1992, AGE.
[11] R. Hayes,et al. Concurrent calpain and caspase-3 mediated proteolysis of alpha II-spectrin and tau in rat brain after methamphetamine exposure: a similar profile to traumatic brain injury. , 2005, Life sciences.
[12] A. DeCaprio,et al. Protein adduct formation as a molecular mechanism in neurotoxicity. , 2005, Toxicological sciences : an official journal of the Society of Toxicology.
[13] A. Sikorski,et al. Spectrin and calpain: a ‘target’ and a ‘sniper’ in the pathology of neuronal cells , 2005, Cellular and Molecular Life Sciences CMLS.
[14] D. Tshala-Katumbay,et al. A new murine model of giant proximal axonopathy , 2005, Acta Neuropathologica.
[15] R. Neumar,et al. Proteins released from degenerating neurons are surrogate markers for acute brain damage , 2004, Neurobiology of Disease.
[16] X. Puente,et al. Protease degradomics: mass spectrometry discovery of protease substrates and the CLIP-CHIP, a dedicated DNA microarray of all human proteases and inhibitors , 2004, Biological chemistry.
[17] R. L. Hayes,et al. Maitotoxin Induces Calpain But Not Caspase-3 Activation and Necrotic Cell Death in Primary Septo-Hippocampal Cultures , 1999, Neurochemical Research.
[18] S. Hirai,et al. Axonal swellings in the corticospinal tracts in amyotrophic lateral sclerosis , 2004, Acta Neuropathologica.
[19] R. Neumar,et al. Cross-talk between Calpain and Caspase Proteolytic Systems During Neuronal Apoptosis* , 2003, The Journal of Biological Chemistry.
[20] Ammar Al-Chalabi,et al. Neurofilaments and neurological disease. , 2003, BioEssays : news and reviews in molecular, cellular and developmental biology.
[21] J. Geddes,et al. Evaluation of conditions for calpain inhibition in the rat spinal cord: effective postinjury inhibition with intraspinal MDL28170 microinjection. , 2003, Journal of neurotrauma.
[22] P. Spencer,et al. Aromatic as well as aliphatic hydrocarbon solvent axonopathy. , 2002, International journal of hygiene and environmental health.
[23] M. S. Kim,et al. 1,2-diacetylbenzene, the neurotoxic metabolite of a chromogenic aromatic solvent, induces proximal axonopathy. , 2001, Toxicology and applied pharmacology.
[24] Jeremy J. Flint,et al. Accumulation of non‐erythroid αII‐spectrin and calpain‐cleaved αII‐spectrin breakdown products in cerebrospinal fluid after traumatic brain injury in rats , 2001 .
[25] J. Sangerman,et al. Spectrin (βSpIIΣ1) is an essential component of synaptic transmission , 2000, Brain Research.
[26] Kevin K. W Wang,et al. Calpain and caspase: can you tell the difference? , 2000, Trends in Neurosciences.
[27] J. Sangerman,et al. Spectrin (betaSpIIsigma1) is an essential component of synaptic transmission. , 2000, Brain research.
[28] R. Gilbertsen,et al. Non-erythroid alpha-spectrin breakdown by calpain and interleukin 1 beta-converting-enzyme-like protease(s) in apoptotic cells: contributory roles of both protease families in neuronal apoptosis. , 1996, The Biochemical journal.
[29] D. Anthony,et al. Pyrrole oxidation and protein cross-linking as necessary steps in the development of gamma-diketone neuropathy. , 1988, Chemical research in toxicology.
[30] A. DeCaprio,et al. Mechanism of formation and quantitation of imines, pyrroles, and stable nonpyrrole adducts in 2,5-hexanedione-treated protein. , 1987, Molecular pharmacology.
[31] Michel Baudry,et al. Brain spectrin, calpain and long-term changes in synaptic efficacy , 1987, Brain Research Bulletin.
[32] D. Ap. n-Hexane neurotoxicity: a mechanism involving pyrrole adduct formation in axonal cytoskeletal protein. , 1987 .
[33] A. Hirano,et al. Fine Structural Observations of Neurofilamentous Changes in Amyotrophic Lateral Sclerosis , 1984, Journal of neuropathology and experimental neurology.
[34] S. Carpenter,et al. Neurofibrillary axonal swellings and amyotrophic lateral sclerosis , 1984, Journal of the Neurological Sciences.