Increased TREM-1 expression in inflamed skin has no functional impact on the pathogenesis of cutaneous disorders.
暂无分享，去创建一个
[1] H. Ait-Oufella,et al. Trem‐1 is not crucial in psoriasiform imiquimod‐induced skin inflammation in mice , 2016, Experimental dermatology.
[2] H. Koga,et al. Discovering potential drug-targets for personalized treatment of autoimmune disorders - what we learn from epidermolysis bullosa acquisita , 2016, Expert opinion on therapeutic targets.
[3] Stefan F. Martin. Immunological mechanisms in allergic contact dermatitis , 2015, Current opinion in allergy and clinical immunology.
[4] T. van der Poll,et al. Triggering receptor expressed on myeloid cells‐1 (TREM‐1) improves host defence in pneumococcal pneumonia , 2014, The Journal of pathology.
[5] A. Oxenius,et al. TREM-1 Deficiency Can Attenuate Disease Severity without Affecting Pathogen Clearance , 2014, PLoS pathogens.
[6] N. J. Eungdamrong,et al. TREM-1 as a Potential Therapeutic Target in Psoriasis , 2013, The Journal of investigative dermatology.
[7] L. Joosten,et al. TREM‐1: intracellular signaling pathways and interaction with pattern recognition receptors , 2013, Journal of leukocyte biology.
[8] W. Seaman,et al. Characterization of TREM‐3, an activating receptor on mouse macrophages: definition of a family of single Ig domain receptors on mouse chromosome 17 , 2002, European journal of immunology.
[9] M. Colonna,et al. TREM-1 amplifies inflammation and is a crucial mediator of septic shock , 2001, Nature.