Genetic influences contributing to LDL particle size in familial combined hyperlipidaemia
暂无分享,去创建一个
L. Peltonen | R. Cantor | M. Laakso | J. Viikari | M. Taskinen | P. Pajukanta | J. Pihlajamäki | P. Kovanen | I. Nuotio | S. Lahdenperä | J. Vakkilainen | K. Ylitalo | S. Lahdenperä
[1] J. Viikari,et al. LDL particle size in familial combined hyperlipidemia: effects of serum lipids, lipoprotein-modifying enzymes, and lipid transfer proteins. , 2002, Journal of lipid research.
[2] J. Hendriks,et al. Diagnosis of Familial Combined Hyperlipidemia Based on Lipid Phenotype Expression in 32 Families: Results of a 5-Year Follow-Up Study , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[3] R. Cantor,et al. Serum C3 but Not Plasma Acylation-Stimulating Protein Is Elevated in Finnish Patients With Familial Combined Hyperlipidemia , 2001, Arteriosclerosis, thrombosis, and vascular biology.
[4] A. Motulsky,et al. Pleiotropic genetic effects on LDL size, plasma triglyceride, and HDL cholesterol in families. , 1999, Arteriosclerosis, thrombosis, and vascular biology.
[5] A. J. Lusis,et al. Novel genes for familial combined hyperlipidemia. , 1999, Current opinion in lipidology.
[6] G. Jarvik,et al. Complex segregation analyses: uses and limitations. , 1998, American journal of human genetics.
[7] R. Krauss,et al. Families with familial combined hyperlipidemia and families enriched for coronary artery disease share genetic determinants for the atherogenic lipoprotein phenotype. , 1998, American journal of human genetics.
[8] L. Kiemeney,et al. A common genetic mechanism determines plasma apolipoprotein B levels and dense LDL subfraction distribution in familial combined hyperlipidemia. , 1998, American journal of human genetics.
[9] J. Graaf,et al. Defects of lipoprotein metabolism in familial combined hyperlipidaemia. , 1998, Current opinion in lipidology.
[10] Viikari,et al. Glucose intolerance in familial combined hyperlipidaemia , 1998 .
[11] C. Packard,et al. Lipoprotein heterogeneity and apolipoprotein B metabolism. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[12] T. Lehtimäki,et al. Phenotype expression in familial combined hyperlipidemia. , 1997, Atherosclerosis.
[13] G. Dagenais,et al. Small, dense low-density lipoprotein particles as a predictor of the risk of ischemic heart disease in men. Prospective results from the Québec Cardiovascular Study. , 1997, Circulation.
[14] L. Kiemeney,et al. Inherited susceptibility determines the distribution of dense low-density lipoprotein subfraction profiles in familial combined hyperlipidemia. , 1996, American journal of human genetics.
[15] C. Packard,et al. Relations between plasma lipids and postheparin plasma lipases and VLDL and LDL subfraction patterns in normolipemic men and women. , 1995, Arteriosclerosis, thrombosis, and vascular biology.
[16] J. Hokanson,et al. Characterization of low‐density lipoprotein subclasses: methodologie approaches and clinical relevance , 1994, Current opinion in lipidology.
[17] E. Wijsman,et al. Genetic predictors of FCHL in four large pedigrees. Influence of ApoB level major locus predicted genotype and LDL subclass phenotype. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.
[18] J. Viikari,et al. Age and gender specific serum lipid and apolipoprotein fractiles of Finnish children and young adults. The Cardiovascular Risk in Young Finns Study , 1994, Acta paediatrica.
[19] M. Farrall,et al. Complex segregation analysis provides evidence for a major gene acting on serum triglyceride levels in 55 British families with familial combined hyperlipidemia. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.
[20] E. Vartiainen,et al. Twenty-year trends in coronary risk factors in north Karelia and in other areas of Finland. , 1994, International journal of epidemiology.
[21] A. de la Chapelle. Disease gene mapping in isolated human populations: the example of Finland. , 1993, Journal of medical genetics.
[22] R. Krauss,et al. Insulin resistance and hyperinsulinemia in individuals with small, dense low density lipoprotein particles. , 1993, The Journal of clinical investigation.
[23] J. Hokanson,et al. Plasma triglyceride and LDL heterogeneity in familial combined hyperlipidemia. , 1993, Arteriosclerosis and thrombosis : a journal of vascular biology.
[24] J. Hokanson,et al. Complex segregation analysis of LDL peak particle diameter , 1993, Genetic epidemiology.
[25] M. King,et al. Atherogenic lipoprotein phenotype. A proposed genetic marker for coronary heart disease risk. , 1990, Circulation.
[26] R. Krauss,et al. Inheritance of low density lipoprotein subclass patterns in familial combined hyperlipidemia. , 1990, Arteriosclerosis.
[27] K. Hirschhorn. Inheritance of low-density lipoprotein subclass patterns: results of complex segregation analysis. , 1989, American journal of human genetics.
[28] P. Wilson,et al. Effect of Gender, Age, and Lipid Status on Low Density Lipoprotein Su bf raction Distribution: Results from the Framingham Offspring Study , 1987, Arteriosclerosis.
[29] G. Bonney,et al. On the statistical determination of major gene mechanisms in continuous human traits: regressive models. , 1984, American journal of medical genetics.
[30] R. Krauss,et al. Identification of multiple subclasses of plasma low density lipoproteins in normal humans. , 1982, Journal of lipid research.
[31] R. Deitch. Commentary from Westminster , 1981, The Lancet.
[32] H. Akaike. A new look at the statistical model identification , 1974 .
[33] A. Motulsky,et al. Hyperlipidemia in coronary heart disease. II. Genetic analysis of lipid levels in 176 families and delineation of a new inherited disorder, combined hyperlipidemia. , 1973, The Journal of clinical investigation.
[34] A. Aro,et al. Family study of serum lipids and lipoproteins in coronary heart-disease. , 1973, Lancet.