Glutamate-induced long-term potentiation of the frequency of miniature synaptic currents in cultured hippocampal neurons
暂无分享,去创建一个
[1] G. Lynch,et al. Intracellular injections of EGTA block induction of hippocampal long-term potentiation , 1983, Nature.
[2] O. Krishtal,et al. NMDA receptor agonists selectively block N-type calcium channels in hippocampal neurons , 1991, Nature.
[3] C. Stevens,et al. NMDA and non-NMDA receptors are co-localized at individual excitatory synapses in cultured rat hippocampus , 1989, Nature.
[4] R S Zucker,et al. Postsynaptic calcium is sufficient for potentiation of hippocampal synaptic transmission. , 1988, Science.
[5] T. Bliss,et al. Long‐lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant path , 1973, The Journal of physiology.
[6] P. Greengard,et al. Effects of synapsin I and calcium/calmodulin-dependent protein kinase II on spontaneous neurotransmitter release in the squid giant synapse. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[7] R. Tsien,et al. Presynaptic enhancement shown by whole-cell recordings of long-term potentiation in hippocampal slices , 1990, Nature.
[8] D. Madison,et al. A requirement for the intercellular messenger nitric oxide in long-term potentiation. , 1991, Science.
[9] J. Connor,et al. Dendritic spines as individual neuronal compartments for synaptic Ca2+ responses , 1991, Nature.
[10] R. Malinow. Transmission between pairs of hippocampal slice neurons: quantal levels, oscillations, and LTP. , 1991, Science.
[11] B. McNaughton,et al. Long‐term enhancement of CA1 synaptic transmission is due to increased quantal size, not quantal content , 1991, Hippocampus.
[12] F. Edwards. LTP is a long term problem , 1991, Nature.
[13] S. Silberberg,et al. Activation of protein kinase C augments evoked transmitter release , 1987, Nature.
[14] R. Nicoll,et al. Postsynaptic contribution to long-term potentiation revealed by the analysis of miniature synaptic currents , 1992, Nature.
[15] M. Jackson,et al. Presynaptic enhancement of synaptic transmission in hippocampal cell cultures by phorbol esters , 1990, Brain Research.
[16] R. Nicoll,et al. An essential role for postsynaptic calmodulin and protein kinase activity in long-term potentiation , 1989, Nature.
[17] B. Katz,et al. Spontaneous subthreshold activity at motor nerve endings , 1952, The Journal of physiology.
[18] E. Kandel,et al. Learning-related synaptic plasticity: LTP and LTD , 1991, Current Opinion in Neurobiology.
[19] P. Andersen,et al. Protein kinase C injection into hippocampal pyramidal cells elicits features of long term potentiation , 1987, Nature.
[20] Solomon H. Snyder,et al. Nitric oxide, a novel neuronal messenger , 1992, Neuron.
[21] T. Bliss,et al. Long-term potentiation of the perforant path in vivo is associated with increased glutamate release , 1982, Nature.
[22] Is maintenance of LTP presynaptic? , 1991, Nature.
[23] J. Clements. Quantal synaptic transmission? , 1991, Nature.
[24] A. Mauro,et al. Effects of Black Widow Spider Venom on the Frog Neuromuscular Junction: Effects on End-plate Potential, Miniature End-plate Potential and Nerve Terminal Spike , 1970, Nature.
[25] D. Choi. Calcium-mediated neurotoxicity: relationship to specific channel types and role in ischemic damage , 1988, Trends in Neurosciences.
[26] D. Faber,et al. Quantal analysis and synaptic efficacy in the CNS , 1991, Trends in Neurosciences.
[27] G. Collingridge,et al. Excitatory amino acids in synaptic transmission in the Schaffer collateral‐commissural pathway of the rat hippocampus. , 1983, The Journal of physiology.
[28] M. Segal. Synaptic transmission between cultured rat hippocampal neurons is enhanced by activation of protein kinase-C , 1989, Neuroscience Letters.
[29] A. G. Gusev,et al. Analysis of ESP fluctuations indicates increased presynaptic release during long-term potentiation in area of hippocampal slices. , 1991 .
[30] C. Stevens,et al. Presynaptic mechanism for long-term potentiation in the hippocampus , 1990, Nature.
[31] Robert C. Malenka,et al. Postsynaptic factors control the duration of synaptic enhancement in area CA1 of the hippocampus , 1991, Neuron.
[32] G. Lynch,et al. Reevaluating the constraints on hypotheses regarding LTP expression , 1991, Hippocampus.
[33] B Sakmann,et al. Quantal analysis of inhibitory synaptic transmission in the dentate gyrus of rat hippocampal slices: a patch‐clamp study. , 1990, The Journal of physiology.
[34] G. Edelman,et al. The NO hypothesis: possible effects of a short-lived, rapidly diffusible signal in the development and function of the nervous system. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[35] S. Deadwyler,et al. Long-term potentiation : from biophysics to behavior , 1988 .
[36] R. Tsien,et al. Inhibition of postsynaptic PKC or CaMKII blocks induction but not expression of LTP. , 1989, Science.
[37] C. Jahr,et al. Quisqualate receptor-mediated depression of calcium currents in hippocampal neurons , 1990, Neuron.
[38] Graham L. Collingridge,et al. Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiation , 1989, Nature.
[39] E. W. Kairiss,et al. Long-term synaptic potentiation. , 1988, Science.
[40] K. Stratford,et al. Quantal analysis of excitatory synaptic action and depression in hippocampal slices , 1991, Nature.
[41] J. Clements,et al. Presynaptic glutamate receptors depress excitatory monosynaptic transmission between mouse hippocampal neurones. , 1990, The Journal of physiology.
[42] S. Redman. Quantal analysis of synaptic potentials in neurons of the central nervous system. , 1990, Physiological reviews.
[43] B. Katz,et al. Quantal components of the end‐plate potential , 1954, The Journal of physiology.
[44] D. Amaral,et al. Neurons, numbers and the hippocampal network. , 1990, Progress in brain research.
[45] A. Lev-Tov,et al. A study of tetanic and post‐tetanic potentiation of miniature end‐plate potentials at the frog neuromuscular junction. , 1980, The Journal of physiology.
[46] E. Kandel,et al. Facilitatory and inhibitory transmitters modulate spontaneous transmitter release at cultured Aplysia sensorimotor synapses. , 1990, The Journal of physiology.
[47] J. Hubbard,et al. An examination of the effects of osmotic pressure changes upon transmitter release from mammalian motor nerve terminals , 1968, The Journal of physiology.
[48] M. Mayer,et al. The physiology of excitatory amino acids in the vertebrate central nervous system , 1987, Progress in Neurobiology.
[49] Thomas H. Brown,et al. Spontaneous miniature synaptic potentials in hippocampal neurons , 1979, Brain Research.
[50] L. Nowak,et al. Electrophysiological studies of NMDA receptors , 1987, Trends in Neurosciences.
[51] W Singer,et al. Excitatory amino acid receptors and synaptic plasticity. , 1990, Trends in pharmacological sciences.
[52] E. Kandel,et al. Tests of the roles of two diffusible substances in long-term potentiation: evidence for nitric oxide as a possible early retrograde messenger. , 1991, Proceedings of the National Academy of Sciences of the United States of America.