Cannabinoid receptor 1 and acute resistance exercise – In vivo and in vitro studies in human skeletal muscle

[1]  T. Hornberger,et al.  The Role of Diacylglycerol Kinase ζ and Phosphatidic Acid in the Mechanical Activation of Mammalian Target of Rapamycin (mTOR) Signaling and Skeletal Muscle Hypertrophy* , 2013, The Journal of Biological Chemistry.

[2]  D. Vertommen,et al.  Mammalian target of rapamycin-independent S6K1 and 4E-BP1 phosphorylation during contraction in rat skeletal muscle. , 2013, Cellular signalling.

[3]  P. Wiklund,et al.  Exercise, the endocannabinoid system and metabolic health , 2013 .

[4]  M. Thiel,et al.  Effects of exercise stress on the endocannabinoid system in humans under field conditions , 2012, European Journal of Applied Physiology.

[5]  D. Sabatini,et al.  A unifying model for mTORC1-mediated regulation of mRNA translation , 2012, Nature.

[6]  M. Gorospe,et al.  Cannabinoids Induce Pancreatic β-Cell Death by Directly Inhibiting Insulin Receptor Activation , 2012, Science Signaling.

[7]  R. Kreider,et al.  Effects of Resistance Exercise Intensity on Extracellular Signal-Regulated Kinase 1/2 Mitogen-Activated Protein Kinase Activation in Men , 2012, Journal of strength and conditioning research.

[8]  K. Smith,et al.  Muscle protein synthesis in response to nutrition and exercise , 2012, The Journal of physiology.

[9]  K. Mackie,et al.  Cannabinoid 1 Receptor Promotes Cardiac Dysfunction, Oxidative Stress, Inflammation, and Fibrosis in Diabetic Cardiomyopathy , 2012, Diabetes.

[10]  R. Krauss,et al.  Acute Overactive Endocannabinoid Signaling Induces Glucose Intolerance, Hepatic Steatosis, and Novel Cannabinoid Receptor 1 Responsive Genes , 2011, PloS one.

[11]  C. Proud,et al.  mTORC1 signaling: what we still don't know. , 2011, Journal of molecular cell biology.

[12]  K. Esser,et al.  Early activation of mTORC1 signalling in response to mechanical overload is independent of phosphoinositide 3‐kinase/Akt signalling , 2011, The Journal of physiology.

[13]  Philippe Pierre,et al.  Novel insights into the regulation of skeletal muscle protein synthesis as revealed by a new nonradioactive in vivo technique , 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[14]  R. Rozenfeld.  Type I Cannabinoid Receptor Trafficking: All Roads Lead to Lysosome , 2011, Traffic.

[15]  D. Sabatini,et al.  mTOR: from growth signal integration to cancer, diabetes and ageing , 2010, Nature Reviews Molecular Cell Biology.

[16]  S. Jacob,et al.  Effects of in vitro antagonism of endocannabinoid‐1 receptors on the glucose transport system in normal and insulin‐resistant rat skeletal muscle , 2010, Diabetes, obesity & metabolism.

[17]  K. Baar,et al.  A Limited Role for PI(3,4,5)P3 Regulation in Controlling Skeletal Muscle Mass in Response to Resistance Exercise , 2010, PloS one.

[18]  K. Mackie,et al.  Regulation of MAP Kinase–Directed Mitogenic and Protein Kinase B–Mediated Signaling by Cannabinoid Receptor Type 1 in Skeletal Muscle Cells , 2009, Diabetes.

[19]  N. Ueda,et al.  Biology of endocannabinoid synthesis system. , 2009, Prostaglandins & other lipid mediators.

[20]  R. Maldonado,et al.  Cannabinoid modulation of hippocampal long-term memory is mediated by mTOR signaling , 2009, Nature Neuroscience.

[21]  T. Hornberger,et al.  The role of phosphoinositide 3‐kinase and phosphatidic acid in the regulation of mammalian target of rapamycin following eccentric contractions , 2009, The Journal of physiology.

[22]  A. Mero,et al.  HIGHLIGHTED TOPIC Regulation of Protein Metabolism in Exercise and Recovery Resistance exercise with whey protein ingestion affects mTOR signaling pathway and myostatin in men , 2009 .

[23]  M. Drummond,et al.  Nutritional and contractile regulation of human skeletal muscle protein synthesis and mTORC1 signaling. , 2009, Journal of applied physiology.

[24]  N. Tennagels,et al.  Cannabinoid type 1 receptors in human skeletal muscle cells participate in the negative crosstalk between fat and muscle , 2009, Diabetologia.

[25]  Philippe Pierre,et al.  SUnSET, a nonradioactive method to monitor protein synthesis , 2009, Nature Methods.

[26]  Sang Gyun Kim,et al.  Rapamycin differentially inhibits S6Ks and 4E-BP1 to mediate cell-type-specific repression of mRNA translation , 2008, Proceedings of the National Academy of Sciences.

[27]  Masahiko Watanabe,et al.  Peripheral, but Not Central, CB1 Antagonism Provides Food Intake–Independent Metabolic Benefits in Diet-Induced Obese Rats , 2008, Diabetes.

[28]  S. Davis,et al.  Endocannabinoid system overactivity and the metabolic syndrome: Prospects for treatment , 2008, Current diabetes reports.

[29]  G. Wittert,et al.  Effects of cannabinoid receptors on skeletal muscle oxidative pathways , 2007, Molecular and Cellular Endocrinology.

[30]  Daoyan Liu,et al.  Exercise reduces adipose tissue via cannabinoid receptor type 1 which is regulated by peroxisome proliferator-activated receptor-delta. , 2007, Biochemical and biophysical research communications.

[31]  K. Gadde.  Effect of rimonabant on weight and cardiometabolic risk factors. , 2006, JAMA.

[32]  S. Chien,et al.  The role of phospholipase D and phosphatidic acid in the mechanical activation of mTOR signaling in skeletal muscle. , 2006, Proceedings of the National Academy of Sciences of the United States of America.

[33]  F Xavier Pi-Sunyer,et al.  Effect of rimonabant, a cannabinoid-1 receptor blocker, on weight and cardiometabolic risk factors in overweight or obese patients: RIO-North America: a randomized controlled trial. , 2006, JAMA.

[34]  Steven P. Gygi,et al.  mTOR and S6K1 Mediate Assembly of the Translation Preinitiation Complex through Dynamic Protein Interchange and Ordered Phosphorylation Events , 2005, Cell.

[35]  J. Després,et al.  Effects of rimonabant on metabolic risk factors in overweight patients with dyslipidemia. , 2005, The New England journal of medicine.

[36]  E. Sausville,et al.  Biochemical correlates of mTOR inhibition by the rapamycin ester CCI-779 and tumor growth inhibition. , 2001, Clinical cancer research : an official journal of the American Association for Cancer Research.

[37]  R. Hampson,et al.  Chronic ▵9‐Tetrahydrocannabinol Treatment Produces a Time‐Dependent Loss of Cannabinoid Receptors and Cannabinoid Receptor‐Activated G Proteins in Rat Brain , 1999, Journal of neurochemistry.

[38]  N. Volkow,et al.  Binding of the non-classical cannabinoid CP 55,940, and the diarylpyrazole AM251 to rodent brain cannabinoid receptors. , 1997, Life sciences.

[39]  G. Butler-Browne,et al.  Clones of human satellite cells can express in vitro both fast and slow myosin heavy chains. , 1994, Developmental biology.

[40]  F. Bermúdez-Silva,et al.  Expression of the cannabinoid system in muscle: effects of a high-fat diet and CB1 receptor blockade. , 2011, The Biochemical journal.