Bleomycin-Induced Lung Injury Activation and Fibroproliferation in IRAK-M Promotes Alternative Macrophage

[1]  Yan-ting Zhu,et al.  Platelet-derived growth factor mediates interleukin-13-induced collagen I production in mouse airway fibroblasts , 2014, Journal of Biosciences.

[2]  R. Flavell,et al.  Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy. , 2014, Journal of the American Society of Nephrology : JASN.

[3]  S. Gharib,et al.  MMP28 promotes macrophage polarization toward M2 cells and augments pulmonary fibrosis , 2014, Journal of leukocyte biology.

[4]  D. Voehringer,et al.  IL-25 and type 2 innate lymphoid cells induce pulmonary fibrosis , 2013, Proceedings of the National Academy of Sciences.

[5]  A. Prasse,et al.  Lung Collagens Perpetuate Pulmonary Fibrosis via CD204 and M2 Macrophage Activation , 2013, PloS one.

[6]  M. Olszewski,et al.  Implicating Exudate Macrophages and Ly-6Chigh Monocytes in CCR2-Dependent Lung Fibrosis following Gene-Targeted Alveolar Injury , 2013, The Journal of Immunology.

[7]  R. Locksley,et al.  Innate lymphoid type 2 cells sustain visceral adipose tissue eosinophils and alternatively activated macrophages , 2013, The Journal of experimental medicine.

[8]  T. Albertson,et al.  A review of current and novel therapies for idiopathic pulmonary fibrosis. , 2013, Journal of thoracic disease.

[9]  N. Frangogiannis,et al.  Endogenous IRAK-M Attenuates Postinfarction Remodeling Through Effects on Macrophages and Fibroblasts , 2012, Arteriosclerosis, thrombosis, and vascular biology.

[10]  Huy A. Nguyen,et al.  Pulmonary surfactant protein A and surfactant lipids upregulate IRAK-M, a negative regulator of TLR-mediated inflammation in human macrophages. , 2012, American journal of physiology. Lung cellular and molecular physiology.

[11]  S. Toda,et al.  Periostin promotes chronic allergic inflammation in response to Th2 cytokines. , 2012, The Journal of clinical investigation.

[12]  T. Standiford,et al.  TLR Signaling Prevents Hyperoxia-Induced Lung Injury by Protecting the Alveolar Epithelium from Oxidant-Mediated Death , 2012, The Journal of Immunology.

[13]  Ivana V. Yang,et al.  The Peripheral Blood Transcriptome Identifies the Presence and Extent of Disease in Idiopathic Pulmonary Fibrosis , 2012, PloS one.

[14]  R. Bowler,et al.  IL-13 dampens human airway epithelial innate immunity through induction of IL-1 receptor-associated kinase M. , 2012, The Journal of allergy and clinical immunology.

[15]  X. Hua,et al.  MyD88 provides a protective role in long-term radiation-induced lung injury , 2012, International journal of radiation biology.

[16]  Yunfeng Zhou,et al.  The development of classically and alternatively activated macrophages has different effects on the varied stages of radiation-induced pulmonary injury in mice. , 2011, Journal of radiation research.

[17]  S. Forbes,et al.  Ly6Chi monocytes direct alternatively activated profibrotic macrophage regulation of lung fibrosis. , 2011, American journal of respiratory and critical care medicine.

[18]  Yan Lu,et al.  Class A scavenger receptor attenuates myocardial infarction-induced cardiomyocyte necrosis through suppressing M1 macrophage subset polarization , 2011, Basic Research in Cardiology.

[19]  R. Kuick,et al.  TGF-β-induced IRAK-M Expression in Tumor Associated Macrophages Regulates Lung Tumor Growth , 2010, Oncogene.

[20]  Y. Fukuda,et al.  Statin attenuates experimental anti-glomerular basement membrane glomerulonephritis together with the augmentation of alternatively activated macrophages. , 2010, The American journal of pathology.

[21]  K. Tabeta,et al.  Interleukin-1 receptor-associated kinase-M in gingival epithelial cells attenuates the inflammatory response elicited by Porphyromonas gingivalis. , 2010, Journal of periodontal research.

[22]  Zhuo-wei Hu,et al.  Toll like receptor 2 mediates bleomycin-induced acute lung injury, inflammation and fibrosis in mice. , 2010, Yao xue xue bao = Acta pharmaceutica Sinica.

[23]  M. Chignard,et al.  Combined Tlr2 and Tlr4 deficiency increases radiation-induced pulmonary fibrosis in mice. , 2010, International journal of radiation oncology, biology, physics.

[24]  Steven L. Kunkel,et al.  Critical Role of IL-1 Receptor-Associated Kinase-M in Regulating Chemokine-Dependent Deleterious Inflammation in Murine Influenza Pneumonia , 2009, The Journal of Immunology.

[25]  S. Uh,et al.  Interleukin-13 and Its Receptors in Idiopathic Interstitial Pneumonia: Clinical Implications for Lung Function , 2009, Journal of Korean medical science.

[26]  C. Hogaboam,et al.  Murine models of pulmonary fibrosis. , 2008, American journal of physiology. Lung cellular and molecular physiology.

[27]  M. Ballinger,et al.  COMPARISON OF CONDITIONING REGIMENS FOR ALVEOLAR MACROPHAGE RECONSTITUTION AND INNATE IMMUNE FUNCTION POST BONE MARROW TRANSPLANT , 2008, Experimental lung research.

[28]  W. Seeger,et al.  Functional role and species-specific contribution of arginases in pulmonary fibrosis. , 2008, American journal of physiology. Lung cellular and molecular physiology.

[29]  K. Isse,et al.  Endotoxin tolerance in human intrahepatic biliary epithelial cells is induced by upregulation of IRAK‐M , 2006, Liver international : official journal of the International Association for the Study of the Liver.

[30]  T. Standiford,et al.  Sepsis-induced suppression of lung innate immunity is mediated by IRAK-M. , 2006, The Journal of clinical investigation.

[31]  P. Noble,et al.  The role of Toll-like receptors in non-infectious lung injury , 2006, Cell Research.

[32]  M. Rojas,et al.  Activation of alveolar macrophages via the alternative pathway in herpesvirus-induced lung fibrosis. , 2006, American journal of respiratory cell and molecular biology.

[33]  G. Prestwich,et al.  Regulation of lung injury and repair by Toll-like receptors and hyaluronan , 2005, Nature Medicine.

[34]  T. Moore,et al.  Protection from Fluorescein Isothiocyanate-Induced Fibrosis in IL-13-Deficient, but Not IL-4-Deficient, Mice Results from Impaired Collagen Synthesis by Fibroblasts1 , 2004, The Journal of Immunology.

[35]  R. Puri,et al.  Therapeutic Attenuation of Pulmonary Fibrosis Via Targeting of IL-4- and IL-13-Responsive Cells 1 , 2003, The Journal of Immunology.

[36]  T. Standiford,et al.  Hyperoxia Mediates Acute Lung Injury and Increased Lethality in Murine Legionella Pneumonia: The Role of Apoptosis1 , 2003, The Journal of Immunology.

[37]  T. Moore,et al.  Protection from Pulmonary Fibrosis in the Absence of CCR2 Signaling1 , 2001, The Journal of Immunology.

[38]  R. Homer,et al.  Interleukin-13 Induces Tissue Fibrosis by Selectively Stimulating and Activating Transforming Growth Factor β1 , 2001, The Journal of experimental medicine.

[39]  D. Cohen,et al.  T cell independence of bleomycin‐induced pulmonary fibrosis , 1999, Journal of leukocyte biology.

[40]  L. Armstrong,et al.  Production of interleukin 13 by alveolar macrophages from normal and fibrotic lung. , 1998, American journal of respiratory cell and molecular biology.

[41]  E. Bröcker,et al.  Interleukin‐13 selectively induces monocyte chemoattractant protein‐1 synthesis and secretion by human endothelial cells. Involvement of IL‐4Rα and Stat6 phosphorylation , 1997, Immunology.

[42]  A. Brody,et al.  Isolation and primary culture of murine alveolar type II cells. , 1996, American journal of respiratory cell and molecular biology.

[43]  Z. Guo,et al.  Effects of alveolar macrophage conditioned media from interstitial lung disease patients on the procollagen mRNA expression in human lung fibroblasts. , 1995, Chinese medical sciences journal = Chung-kuo i hsueh k'o hsueh tsa chih.

[44]  M. Kaghad,et al.  Interleukin-13 is a new human lymphokine regulating inflammatory and immune responses. , 1993, Nature.