VPS35-deficiency results in an impaired AMPA receptor trafficking and decreased dendritic spine maturation
暂无分享,去创建一个
L. Mei | B. Tang | W. Xiong | Xiang-Dong Sun | Fu-lei Tang | Yun Tian | Lei Wen
[1] L. Mei,et al. VPS35 Deficiency or Mutation Causes Dopaminergic Neuronal Loss by Impairing Mitochondrial Fusion and Function. , 2015, Cell reports.
[2] L. Mei,et al. VPS35 in Dopamine Neurons Is Required for Endosome-to-Golgi Retrieval of Lamp2a, a Receptor of Chaperone-Mediated Autophagy That Is Critical for α-Synuclein Degradation and Prevention of Pathogenesis of Parkinson's Disease , 2015, The Journal of Neuroscience.
[3] M. Farrer,et al. Retromer-dependent neurotransmitter receptor trafficking to synapses is altered by the Parkinson's disease VPS35 mutation p.D620N. , 2015, Human molecular genetics.
[4] P. Cullen,et al. Retromer Binding to FAM21 and the WASH Complex Is Perturbed by the Parkinson Disease-Linked VPS35(D620N) Mutation , 2014, Current Biology.
[5] D. Rubinsztein,et al. Mutation in VPS35 associated with Parkinson’s disease impairs WASH complex association and inhibits autophagy , 2014, Nature Communications.
[6] R. Nicoll,et al. Retromer Mediates a Discrete Route of Local Membrane Delivery to Dendrites , 2014, Neuron.
[7] A. Gautreau,et al. Retromer-mediated endosomal protein sorting: all WASHed up! , 2013, Trends in cell biology.
[8] L. Mei,et al. Vps35 loss promotes hyperresorptive osteoclastogenesis and osteoporosis via sustained RANKL signaling , 2013, The Journal of cell biology.
[9] K. Marder,et al. RAB7L1 Interacts with LRRK2 to Modify Intraneuronal Protein Sorting and Parkinson’s Disease Risk , 2013, Neuron.
[10] M. Seaman. The retromer complex – endosomal protein recycling and beyond , 2012, Journal of Cell Science.
[11] L. Mei,et al. VPS35 regulates developing mouse hippocampal neuronal morphogenesis by promoting retrograde trafficking of BACE1 , 2012, Biology Open.
[12] L. Mei,et al. Distinct Roles of Muscle and Motoneuron LRP 4 in Neuromuscular Junction Formation , 2012 .
[13] E. Derivery,et al. Actin Polymerization Controls the Organization of WASH Domains at the Surface of Endosomes , 2012, PloS one.
[14] M. Seaman,et al. Recruitment of the endosomal WASH complex is mediated by the extended 'tail' of Fam21 binding to the retromer protein Vps35. , 2012, The Biochemical journal.
[15] L. Mei,et al. VPS35 haploinsufficiency increases Alzheimer’s disease neuropathology , 2011, The Journal of cell biology.
[16] Marc N. Offman,et al. A mutation in VPS35, encoding a subunit of the retromer complex, causes late-onset Parkinson disease. , 2011, American journal of human genetics.
[17] M. Farrer,et al. VPS35 mutations in Parkinson disease. , 2011, American journal of human genetics.
[18] C. Hoogenraad,et al. Dendritic Spine Plasticity: New Regulatory Roles of Dynamic Microtubules , 2010, The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry.
[19] Benjamin E. L. Lauffer,et al. Sequence-Dependent Sorting of Recycling Proteins by Actin-Stabilized Endosomal Microdomains , 2010, Cell.
[20] T. Svitkina,et al. Regulation of the Postsynaptic Cytoskeleton: Roles in Development, Plasticity, and Disorders , 2010, The Journal of Neuroscience.
[21] L. Raymond,et al. Early synaptic pathophysiology in neurodegeneration: insights from Huntington's disease , 2010, Trends in Neurosciences.
[22] P. Verkade,et al. SNX-BAR proteins in phosphoinositide-mediated, tubular-based endosomal sorting. , 2010, Seminars in cell & developmental biology.
[23] D. Walsh,et al. Alzheimer's disease: synaptic dysfunction and Aβ , 2009, Molecular Neurodegeneration.
[24] D. Billadeau,et al. A FAM21-containing WASH complex regulates retromer-dependent sorting. , 2009, Developmental cell.
[25] Ling Lin,et al. Axon guidance and synaptic maintenance: preclinical markers for neurodegenerative disease and therapeutics , 2009, Trends in Neurosciences.
[26] L. Honig,et al. Retromer deficiency observed in Alzheimer's disease causes hippocampal dysfunction, neurodegeneration, and Aβ accumulation , 2008, Proceedings of the National Academy of Sciences.
[27] T. Soderling,et al. Regulatory mechanisms of AMPA receptors in synaptic plasticity , 2007, Nature Reviews Neuroscience.
[28] Yu-Qiang Ding,et al. Myosin X regulates netrin receptors and functions in axonal path-finding , 2007, Nature Cell Biology.
[29] Kristen M. Harris,et al. Plasticity-Induced Growth of Dendritic Spines by Exocytic Trafficking from Recycling Endosomes , 2006, Neuron.
[30] L. Honig,et al. Model‐guided microarray implicates the retromer complex in Alzheimer's disease , 2005, Annals of neurology.
[31] M. Seaman,et al. Recycle your receptors with retromer. , 2005, Trends in cell biology.
[32] Yu-Qiang Ding,et al. Phosphatidylinositol transfer protein-alpha in netrin-1-induced PLC signalling and neurite outgrowth. , 2005, Nature cell biology.
[33] G. Ming,et al. Focal adhesion kinase in netrin-1 signaling , 2004, Nature Neuroscience.
[34] Mikyoung Park,et al. Recycling Endosomes Supply AMPA Receptors for LTP , 2004, Science.
[35] R. Nicoll,et al. AMPA Receptor Trafficking at Excitatory Synapses , 2003, Neuron.
[36] Richard L. Huganir,et al. Postsynaptic organisation and regulation of excitatory synapses , 2000, Nature Reviews Neuroscience.
[37] M. Ehlers,et al. Reinsertion or Degradation of AMPA Receptors Determined by Activity-Dependent Endocytic Sorting , 2000, Neuron.
[38] K. Davis,et al. Correlation between elevated levels of amyloid beta-peptide in the brain and cognitive decline. , 2000, JAMA.
[39] S. Nakanishi. Molecular diversity of glutamate receptors and implications for brain function. , 1992, Science.