Clonal origins of ETV6-RUNX1+ acute lymphoblastic leukemia: studies in monozygotic twins
暂无分享,去创建一个
N. Potter | M. Greaves | L. Kearney | D. Gonzalez | D. Alpár | L. Ermini | C. Bateman | A. Ford | T. Szczepański | F. V. Delft | C. Bateman | I. Titley | M. Mansur | N. Potter | D. Wren | F. V. Delft
[1] Leslie L Robison,et al. Acute lymphoblastic leukaemia , 2018, Radiopaedia.org.
[2] M. Greaves,et al. Evolutionary trajectories of hyperdiploid ALL in monozygotic twins , 2014, Leukemia.
[3] M. Stratton,et al. RAG-mediated recombination is the predominant driver of oncogenic rearrangement in ETV6-RUNX1 acute lymphoblastic leukemia , 2014, Nature Genetics.
[4] P. Campbell,et al. Single-cell mutational profiling and clonal phylogeny in cancer , 2013, Genome research.
[5] R. Houlston,et al. Developmental timing of mutations revealed by whole-genome sequencing of twins with acute lymphoblastic leukemia , 2013, Proceedings of the National Academy of Sciences.
[6] C. Lobe,et al. Initially disadvantaged, TEL-AML1 cells expand and initiate leukemia in response to irradiation and cooperating mutations , 2013, Leukemia.
[7] V. Carlton,et al. Massive evolution of the immunoglobulin heavy chain locus in children with B precursor acute lymphoblastic leukemia. , 2012, Blood.
[8] E. Schuuring,et al. EuroClonality/BIOMED-2 guidelines for interpretation and reporting of Ig/TCR clonality testing in suspected lymphoproliferations , 2012, Leukemia.
[9] A. Ashworth,et al. A Modified Method for Whole Exome Resequencing from Minimal Amounts of Starting DNA , 2012, PloS one.
[10] Louise S. Matheson,et al. Modeling the evolution of ETV6-RUNX1-induced B-cell precursor acute lymphoblastic leukemia in mice. , 2011, Blood.
[11] K. Anderson,et al. Genetic variegation of clonal architecture and propagating cells in leukaemia , 2011, Nature.
[12] M. Greaves,et al. Acquisition of genome-wide copy number alterations in monozygotic twins with acute lymphoblastic leukemia. , 2010, Blood.
[13] K. Anderson,et al. Clonal origins of relapse in ETV6-RUNX1 acute lymphoblastic leukemia. , 2009, Blood.
[14] S. Orkin,et al. TEL-AML1 corrupts hematopoietic stem cells to persist in the bone marrow and initiate leukemia. , 2009, Cell stem cell.
[15] M. Lieber,et al. Human Chromosomal Translocations at CpG Sites and a Theoretical Basis for Their Lineage and Stage Specificity , 2008, Cell.
[16] B. Schäfer,et al. Minimal residual disease-directed risk stratification using real-time quantitative PCR analysis of immunoglobulin and T-cell receptor gene rearrangements in the international multicenter trial AIEOP-BFM ALL 2000 for childhood acute lymphoblastic leukemia , 2008, Leukemia.
[17] T. Enver,et al. Initiating and Cancer-Propagating Cells in TEL-AML1-Associated Childhood Leukemia , 2008, Science.
[18] J. Cayuela,et al. Analysis of minimal residual disease by Ig/TCR gene rearrangements: guidelines for interpretation of real-time quantitative PCR data , 2007, Leukemia.
[19] M. Reinders,et al. Ig gene rearrangement steps are initiated in early human precursor B cell subsets and correlate with specific transcription factor expression , 2006, The Journal of Immunology.
[20] M. Greaves,et al. Defining the oncogenic function of the TEL/AML1 (ETV6/RUNX1) fusion protein in a mouse model , 2005, Oncogene.
[21] K. Anderson,et al. Distinct patterns of hematopoietic stem cell involvement in acute lymphoblastic leukemia , 2005, Nature Medicine.
[22] S. Orkin,et al. Developmental stage–selective effect of somatically mutated leukemogenic transcription factor GATA1 , 2005, Nature Genetics.
[23] R. Pieters,et al. Leukemic stem cells in childhood high-risk ALL/t(9;22) and t(4;11) are present in primitive lymphoid-restricted CD34+CD19- cells. , 2005, Cancer research.
[24] M. Dettling,et al. Prenatal origin of separate evolution of leukemia in identical twins , 2004, Leukemia.
[25] B. Nadel,et al. Unraveling the Consecutive Recombination Events in the Human IGK Locus1 , 2004, The Journal of Immunology.
[26] M. Greaves,et al. Modeling first-hit functions of the t(12;21) TEL-AML1 translocation in mice. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[27] M Hummel,et al. Design and standardization of PCR primers and protocols for detection of clonal immunoglobulin and T-cell receptor gene recombinations in suspect lymphoproliferations: Report of the BIOMED-2 Concerted Action BMH4-CT98-3936 , 2003, Leukemia.
[28] M. Greaves,et al. Leukemia in twins: lessons in natural history. , 2003, Blood.
[29] J. Dongen,et al. Age-related patterns of immunoglobulin and T-cell receptor gene rearrangements in precursor-B-ALL: implications for detection of minimal residual disease , 2003, Leukemia.
[30] M. Greaves,et al. Origins of chromosome translocations in childhood leukaemia , 2003, Nature Reviews Cancer.
[31] Fritz Melchers,et al. A genomic view of lymphocyte development. , 2003, Current opinion in immunology.
[32] J. Harbott,et al. Immature CD34+CD19- progenitor/stem cells in TEL/AML1-positive acute lymphoblastic leukemia are genetically and functionally normal. , 2002, Blood.
[33] M. Greaves,et al. Chromosome translocations and covert leukemic clones are generated during normal fetal development , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[34] M. Greaves,et al. Prenatal origin of acute lymphoblastic leukaemia in children , 1999, The Lancet.
[35] M. Greaves,et al. Structure and possible mechanisms of TEL-AML1 gene fusions in childhood acute lymphoblastic leukemia. , 1999, Cancer research.
[36] T. Szczepański,et al. Cross-lineage T cell receptor gene rearrangements occur in more than ninety percent of childhood precursor-B acute lymphoblastic leukemias: alternative PCR targets for detection of minimal residual disease , 1999, Leukemia.
[37] M. Greaves,et al. Fetal origins of the TEL-AML1 fusion gene in identical twins with leukemia. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[38] E. Macintyre,et al. High frequency of t(12;21) in childhood B-lineage acute lymphoblastic leukemia. , 1995, Blood.
[39] M. Greaves,et al. Rapid intraclonal switch of lineage dominance in congenital leukaemia with a MLL gene rearrangement. , 1995, Leukemia.
[40] T. Lister,et al. Relation of “lymphoid” phenotype and response to chemotherapy incorporating vincristine‐prednisolone in the acute phase of Ph1 positive leukemia , 1979, Cancer.
[41] F. L. Stanton,et al. Identical Twins , 1927, Edinburgh Medical Journal.
[42] A. Hall,et al. Genomic analysis of different clonal evolution in a twin pair with t(12;21) positive acute lymphoblastic leukemia sharing the same prenatal clone , 2008, Leukemia.
[43] M. Schrappe,et al. High incidence and unique features of antigen receptor gene rearrangements in TEL–AML1-positive leukemias , 2004, Leukemia.
[44] J. Dongen,et al. GENE REARRANGEMENTS ARE FREQUENT IN PRECURSOR-B-ACUTE LYMPHOBLASTIC LEUKEMIA BUT RARE IN NORMAL LYMPHOID CELLS , 2003 .
[45] M. Tristem. Molecular Evolution — A Phylogenetic Approach. , 2000, Heredity.