Molecular immunologic correlates of spontaneous latency in a rabbit model of pulmonary tuberculosis

[1]  G. Kaplan,et al.  Spontaneous latency in a rabbit model of pulmonary tuberculosis. , 2012, The American journal of pathology.

[2]  V. Deretic,et al.  Autophagy protects against active tuberculosis by suppressing bacterial burden and inflammation , 2012, Proceedings of the National Academy of Sciences.

[3]  J. Ernst.  The immunological life cycle of tuberculosis , 2012, Nature Reviews Immunology.

[4]  V. Dixit,et al.  Signaling in innate immunity and inflammation. , 2012, Cold Spring Harbor perspectives in biology.

[5]  John C. Marioni,et al.  Deciphering the genetic architecture of variation in the immune response to Mycobacterium tuberculosis infection , 2012, Proceedings of the National Academy of Sciences.

[6]  A. Choi,et al.  Autophagy: a core cellular process with emerging links to pulmonary disease. , 2011, American journal of respiratory and critical care medicine.

[7]  G. Kaplan,et al.  Chronic pulmonary cavitary tuberculosis in rabbits: a failed host immune response , 2011, Open Biology.

[8]  Zhikai Xu,et al.  A review of murine models of latent tuberculosis infection , 2011, Scandinavian journal of infectious diseases.

[9]  B. Mishra,et al.  Cathelicidin is involved in the intracellular killing of mycobacteria in macrophages , 2011, Cellular microbiology.

[10]  S. Sridhar,et al.  Redefining latent tuberculosis. , 2011, Future microbiology.

[11]  G. Kaplan,et al.  Phosphodiesterase-4 Inhibition Alters Gene Expression and Improves Isoniazid – Mediated Clearance of Mycobacterium tuberculosis in Rabbit Lungs , 2011, PLoS pathogens.

[12]  G. Kaplan,et al.  Phosphodiesterase-4 inhibition combined with isoniazid treatment of rabbits with pulmonary tuberculosis reduces macrophage activation and lung pathology. , 2011, The American journal of pathology.

[13]  J. Keane,et al.  Autophagy in the immune response to tuberculosis: clinical perspectives , 2011, Clinical and experimental immunology.

[14]  Xiaoying Fu,et al.  Identification of Mycobacterium tuberculosis-Specific Th1, Th17 and Th22 Cells Using the Expression of CD40L in Tuberculous Pleurisy , 2011, PloS one.

[15]  U. Schaible,et al.  Interferon Gamma Activated Macrophages Kill Mycobacteria by Nitric Oxide Induced Apoptosis , 2011, PloS one.

[16]  J. Ernst,et al.  Initiation and regulation of T-cell responses in tuberculosis , 2011, Mucosal Immunology.

[17]  Alimuddin Zumla,et al.  Immunological biomarkers of tuberculosis , 2011, Nature Reviews Immunology.

[18]  M. Bachmann,et al.  The Bidirectional Crosstalk between Human Dendritic Cells and Natural Killer Cells , 2011, Journal of Innate Immunity.

[19]  V. Deretic.  Autophagy in immunity and cell‐autonomous defense against intracellular microbes , 2011, Immunological reviews.

[20]  Stephen J. Huang,et al.  Genome-wide transcription profiling of human sepsis: a systematic review , 2010, Critical care.

[21]  T. Dick,et al.  A Color Atlas of Comparative Pathology of Pulmonary Tuberculosis , 2010 .

[22]  Irma E. Gonzalez-Curiel,et al.  Kinetics and cellular sources of cathelicidin during the course of experimental latent tuberculous infection and progressive pulmonary tuberculosis , 2010, Clinical and experimental immunology.

[23]  T. Ottenhoff,et al.  LL-37 Directs Macrophage Differentiation toward Macrophages with a Proinflammatory Signature , 2010, The Journal of Immunology.

[24]  V. Pascual,et al.  Assessing the human immune system through blood transcriptomics , 2010, BMC Biology.

[25]  C. Dye,et al.  The Population Dynamics and Control of Tuberculosis , 2010, Science.

[26]  F. Brombacher,et al.  CD28 and IL-4: two heavyweights controlling the balance between immunity and inflammation , 2010, Medical Microbiology and Immunology.

[27]  H. Virgin,et al.  Delivery of cytosolic components by autophagic adaptor protein p62 endows autophagosomes with unique antimicrobial properties. , 2010, Immunity.

[28]  W. Bishai,et al.  Penitentiary or penthouse condo: the tuberculous granuloma from the microbe's point of view , 2010, Cellular microbiology.

[29]  I. Orme,et al.  Immune response to Mycobacterium tuberculosis and identification of molecular markers of disease. , 2009, American journal of respiratory cell and molecular biology.

[30]  S. Khader,et al.  The role of cytokines in the initiation, expansion, and control of cellular immunity to tuberculosis , 2008, Immunological reviews.

[31]  E. Nardell,et al.  What animal models teach humans about tuberculosis. , 2008, American journal of respiratory cell and molecular biology.

[32]  S. Kaufmann,et al.  Handbook of Tuberculosis , 2008 .

[33]  M. Torres,et al.  Expression of Cathelicidin LL-37 during Mycobacterium tuberculosis Infection in Human Alveolar Macrophages, Monocytes, Neutrophils, and Epithelial Cells , 2007, Infection and Immunity.

[34]  J. Altman,et al.  Memory CD8+ T Cells Require CD28 Costimulation1 , 2007, The Journal of Immunology.

[35]  J. Keane,et al.  T helper 2 cytokines inhibit autophagic control of intracellular Mycobacterium tuberculosis. , 2007, Immunity.

[36]  Chul Hee Choi,et al.  Intracellular signalling cascades regulating innate immune responses to Mycobacteria: branching out from Toll‐like receptors , 2007, Cellular microbiology.

[37]  W. Britton,et al.  Life and death in the granuloma: immunopathology of tuberculosis , 2007, Immunology and cell biology.

[38]  E. Julián,et al.  Determinant role for Toll-like receptor signalling in acute mycobacterial infection in the respiratory tract. , 2006, Microbes and infection.

[39]  Alimuddin Zumla,et al.  Myobacterium tuberculosis Induces Selective Up-Regulation of TLRs in the Mononuclear Leukocytes of Patients with Active Pulmonary Tuberculosis1 , 2006, The Journal of Immunology.

[40]  S. Kaufmann,et al.  New insights into the function of granulomas in human tuberculosis , 2006, The Journal of pathology.

[41]  W. Shafer,et al.  Antimicrobial peptides and endotoxin inhibit cytokine and nitric oxide release but amplify respiratory burst response in human and murine macrophages , 2005, Cellular microbiology.

[42]  M. Reed,et al.  Virulence of selected Mycobacterium tuberculosis clinical isolates in the rabbit model of meningitis is dependent on phenolic glycolipid produced by the bacilli. , 2005, The Journal of infectious diseases.

[43]  V. Deretic,et al.  Autophagy Is a Defense Mechanism Inhibiting BCG and Mycobacterium tuberculosis Survival in Infected Macrophages , 2004, Cell.

[44]  Eric Vivier,et al.  Natural Killer Cell Signaling Pathways , 2004, Science.

[45]  B. Ryffel,et al.  Toll-like receptor pathways in the immune responses to mycobacteria. , 2004, Microbes and infection.

[46]  P. Ghazal,et al.  Molecular signatures for diagnosis of infection: application of microarray technology , 2004, Journal of applied microbiology.

[47]  W. Bishai,et al.  Different Strains of Mycobacterium tuberculosis Cause Various Spectrums of Disease in the Rabbit Model of Tuberculosis , 2003, Infection and Immunity.

[48]  D. Hoft,et al.  Differential Effects of Control and Antigen-Specific T Cells on Intracellular Mycobacterial Growth , 2003, Infection and Immunity.

[49]  R. Appelberg,et al.  CD8- and CD95/95L-dependent mechanisms of resistance in mice with chronic pulmonary tuberculosis. , 2001, American journal of respiratory cell and molecular biology.

[50]  C. Nathan,et al.  Reactive oxygen and nitrogen intermediates in the relationship between mammalian hosts and microbial pathogens. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[51]  R. González-Amaro,et al.  Co‐stimulatory signals increase the reactivity of γδ T cells towards mycobacterial antigens , 2000 .

[52]  P. Haslett,et al.  Mycobacterium tuberculosis CDC1551 induces a more vigorous host response in vivo and in vitro, but is not more virulent than other clinical isolates. , 1999, Journal of immunology.

[53]  J. Mudgett,et al.  Identification of nitric oxide synthase as a protective locus against tuberculosis. , 1997, Proceedings of the National Academy of Sciences of the United States of America.

[54]  K. Tanaka,et al.  Effects of nitric oxide synthase inhibitors on murine infection with Mycobacterium tuberculosis , 1995, Infection and immunity.

[55]  J. Flynn,et al.  An essential role for interferon gamma in resistance to Mycobacterium tuberculosis infection , 1993, The Journal of experimental medicine.

[56]  B. Bloom,et al.  Killing of virulent Mycobacterium tuberculosis by reactive nitrogen intermediates produced by activated murine macrophages , 1992, The Journal of experimental medicine.

[57]  THE WORLD HEALTH ORGANIZATION , 1954 .

[58]  M. Sandor,et al.  Monocyte-derived inflammatory dendritic cells in the granuloma during mycobacterial infection. , 2012, Advances in experimental medicine and biology.

[59]  S. Jennings,et al.  Memory CD8 T Cells Require CD28 Costimulation , 2007 .

[60]  B. Ryffel,et al.  Toll-like receptor 2-deficient mice succumb to Mycobacterium tuberculosis infection. , 2004, The American journal of pathology.

[61]  R. González-Amaro,et al.  Co-stimulatory signals increase the reactivity of gammadelta T cells towards mycobacterial antigens. , 2000, Clinical and experimental immunology.

[62]  Global Tuberculosis Programme.  Global tuberculosis control : WHO report , 1997 .

[63]  I. Orme,et al.  Materials and Methods Briefdefinitive Report Disseminated Tuberculosis in Interferon 7 Gene-disrupted Mice , 2022 .