Fat mass and obesity-associated gene rs9939609 polymorphism is a potential biomarker of recurrent venous thromboembolism in male but not in female patients.
暂无分享,去创建一个
[1] J. Sundquist,et al. Identification of Genetic Aberrations in Thrombomodulin Gene in Patients With Recurrent Venous Thromboembolism , 2017, Clinical and applied thrombosis/hemostasis : official journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis.
[2] S. Sebert,et al. Fto-Deficiency Affects the Gene and MicroRNA Expression Involved in Brown Adipogenesis and Browning of White Adipose Tissue in Mice , 2016, International journal of molecular sciences.
[3] B. Dahlbäck,et al. Epidemiology of Familial Aggregation of Venous Thromboembolism , 2016, Seminars in Thrombosis and Hemostasis.
[4] Richard D Riley,et al. University of Birmingham A systematic review of prognostic models for recurrent venous thromboembolism (VTE) post treatment of first unprovoked VTE , 2016 .
[5] L. Orozco,et al. Gender-Dependent Association of FTO Polymorphisms with Body Mass Index in Mexicans , 2016, PloS one.
[6] J. Fahrni,et al. Assessing the risk of recurrent venous thromboembolism – a practical approach , 2015, Vascular health and risk management.
[7] J. Heit. Epidemiology of venous thromboembolism , 2015, Nature Reviews Cardiology.
[8] M. Benn,et al. Obesity as a causal risk factor for deep venous thrombosis: a Mendelian randomization study , 2015, Journal of internal medicine.
[9] S. Hannenhalli,et al. Conservation in first introns is positively associated with the number of exons within genes and the presence of regulatory epigenetic signals , 2014, BMC Genomics.
[10] J. Mi,et al. Age- and Sex-Dependent Association between FTO rs9939609 and Obesity-Related Traits in Chinese Children and Adolescents , 2014, PloS one.
[11] P. Mannucci,et al. Inherited risk factors for venous thromboembolism , 2014, Nature Reviews Cardiology.
[12] S. Cannegieter,et al. Venous thrombosis: understanding the paradoxes of recurrence , 2013, Journal of thrombosis and haemostasis : JTH.
[13] H. Kluin-Nelemans,et al. Increased risk of arterial thromboembolism after a prior episode of venous thromboembolism: results from the Prevention of REnal and Vascular ENd stage Disease (PREVEND) Study , 2012, British journal of haematology.
[14] A. Gottsäter,et al. Evaluation of recurrent venous thromboembolism in patients with Factor V Leiden mutation in heterozygous form. , 2012, Thrombosis research.
[15] Nirav R. Shah,et al. Measuring Adiposity in Patients: The Utility of Body Mass Index (BMI), Percent Body Fat, and Leptin , 2012, PloS one.
[16] M. Donati,et al. Thrombosis and obesity: cellular bases. , 2012, Thrombosis research.
[17] A. Ciampi,et al. Men had a higher risk of recurrent venous thromboembolism than women: a large population study. , 2012, Gender medicine.
[18] J. Sundquist,et al. Age- and Gender-Specific Familial Risks for Venous ThromboembolismClinical Perspective , 2011 .
[19] F. Rosendaal,et al. Risk assessment for recurrent venous thrombosis , 2010, The Lancet.
[20] A. Iorio,et al. Risk of recurrence after a first episode of symptomatic venous thromboembolism provoked by a transient risk factor: a systematic review. , 2010, Archives of internal medicine.
[21] Roger D. Cox,et al. Overexpression of Fto leads to increased food intake and results in obesity , 2010, Nature Genetics.
[22] J. Pitha,et al. A FTO variant and risk of acute coronary syndrome. , 2010, Clinica chimica acta; international journal of clinical chemistry.
[23] P. Wells,et al. Systematic Review: Case-Fatality Rates of Recurrent Venous Thromboembolism and Major Bleeding Events Among Patients Treated for Venous Thromboembolism , 2010, Annals of Internal Medicine.
[24] T. Baglin,et al. Unraveling the thrombophilia paradox: from hypercoagulability to the prothrombotic state , 2010, Journal of thrombosis and haemostasis : JTH.
[25] L. Lannfelt,et al. The common FTO variant rs9939609 is not associated with BMI in a longitudinal study on a cohort of Swedish men born 1920-1924 , 2009, BMC Medical Genetics.
[26] S. Yanovski,et al. The FTO gene rs9939609 obesity-risk allele and loss of control over eating. , 2009, The American journal of clinical nutrition.
[27] A. Gottsäter,et al. Prospective analysis of risk factors and distribution of venous thromboembolism in the population-based Malmö Thrombophilia Study (MATS). , 2009, Thrombosis research.
[28] A. Folsom,et al. C-reactive protein and venous thromboembolism , 2009, Thrombosis and Haemostasis.
[29] M. Prins,et al. A lower risk of recurrent venous thrombosis in women compared with men is explained by sex-specific risk factors at time of first venous thrombosis in thrombophilic families. , 2009, Blood.
[30] D. Allison,et al. Mortality, Health Outcomes, and Body Mass Index in the Overweight Range: A Science Advisory From the American Heart Association , 2009, Circulation.
[31] Jens C. Brüning,et al. Inactivation of the Fto gene protects from obesity , 2009, Nature.
[32] M. Schulze,et al. Association of the FTO rs9939609 Single Nucleotide Polymorphism With C‐reactive Protein Levels , 2009, Obesity.
[33] Tom R. Gaunt,et al. American Journal of Epidemiology Practice of Epidemiology Hardy-weinberg Equilibrium Testing of Biological Ascertainment for Mendelian Randomization Studies , 2022 .
[34] J. Heit,et al. The epidemiology of venous thromboembolism in the community. , 2008, Arteriosclerosis, thrombosis, and vascular biology.
[35] J. Douketis,et al. The Risk for Fatal Pulmonary Embolism after Discontinuing Anticoagulant Therapy for Venous Thromboembolism , 2007, Annals of Internal Medicine.
[36] Cecilia Becattini,et al. Cardiovascular Risk Factors and Venous Thromboembolism: A Meta-Analysis , 2008, Circulation.
[37] M. Jarvelin,et al. A Common Variant in the FTO Gene Is Associated with Body Mass Index and Predisposes to Childhood and Adult Obesity , 2007, Science.
[38] D. Hunter. Gene–environment interactions in human diseases , 2005, Nature Reviews Genetics.
[39] T. Baglin,et al. High risk of recurrent venous thromboembolism in men , 2004, Journal of thrombosis and haemostasis : JTH.
[40] E. Minar,et al. The risk of recurrent venous thromboembolism in men and women. , 2004, The New England journal of medicine.
[41] M. de Andrade,et al. Familial segregation of venous thromboembolism , 2004, Journal of thrombosis and haemostasis : JTH.
[42] J. Vaupel,et al. Major Genetic Susceptibility for Venous Thromboembolism in Men: A Study of Danish Twins , 2003, Epidemiology.
[43] D. Allison,et al. Twins and virtual twins: bases of relative body weight revisited , 2002, International Journal of Obesity.
[44] J. Stephens,et al. Haplotype Variation and Linkage Disequilibrium in 313 Human Genes , 2001, Science.
[45] M Gent,et al. A comparison of three months of anticoagulation with extended anticoagulation for a first episode of idiopathic venous thromboembolism. , 1999, The New England journal of medicine.
[46] B. Dahlbäck,et al. A New Direct, Fast and Quantitative Enzyme-linked Ligandsorbent Assay for Measurement of Free Protein S Antigen , 1998, Thrombosis and Haemostasis.
[47] M. Neale,et al. Genetic and Environmental Factors in Relative Body Weight and Human Adiposity , 1997, Behavior genetics.
[48] B. Dahlbäck,et al. Prevalence of factor V gene mutation amongst myocardial infarction patients and healthy controls is higher in Sweden than in other countries , 1996, Journal of internal medicine.
[49] D. Cooper,et al. Population differences in the frequency of the factor V Leiden variant among people with clinically symptomatic protein C deficiency. , 1995, Journal of medical genetics.
[50] R. Francis,et al. Rapid amidolytic assay of protein C in whole plasma using an activator from the venom of Agkistrodon contortrix. , 1987, American journal of clinical pathology.
[51] O. Odegård,et al. Heparin cofactor activity measured with an amidolytic method. , 1975, Thrombosis research.
[52] J. Sundquist,et al. A sibling based design to quantify genetic and shared environmental effects of venous thromboembolism in Sweden. , 2017, Thrombosis research.
[53] R. Loos,et al. The bigger picture of FTO—the first GWAS-identified obesity gene , 2014, Nature Reviews Endocrinology.
[54] M. Patlas,et al. The clinical importance of visceral adiposity: a critical review of methods for visceral adipose tissue analysis. , 2012, The British journal of radiology.
[55] Christian Gieger,et al. Six new loci associated with body mass index highlight a neuronal influence on body weight regulation , 2009, Nature Genetics.
[56] W. Ageno,et al. Abdominal obesity and the risk of recurrent deep vein thrombosis. , 2007, Thrombosis research.
[57] E. Minar,et al. The Risk of Early Recurrent Venous Thromboembolism after Oral Anticoagulant Therapy in Patients with the G20210A Transition in the Prothrombin Gene , 1999, Thrombosis and Haemostasis.