Stability of BAT26 in Lynch syndrome colorectal tumours

[1]  Caroline Brennetot,et al.  Mononucleotide repeats BAT‐26 and BAT‐25 accurately detect MSI‐H tumors and predict tumor content: Implications for population screening , 2005, International journal of cancer.

[2]  G. Taylor,et al.  Genomic deletions in MSH2 or MLH1 are a frequent cause of hereditary non‐polyposis colorectal cancer: Identification of novel and recurrent deletions by MLPA , 2003, Human mutation.

[3]  H. Nakagawa,et al.  Identification and characterization of genomic rearrangements of MSH2 and MLH1 in Lynch syndrome (HNPCC) by novel techniques , 2003, Human mutation.

[4]  Daniel J Sargent,et al.  Immunohistochemistry versus microsatellite instability testing in phenotyping colorectal tumors. , 2002, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.

[5]  B. Leggett,et al.  Features of colorectal cancers with high-level microsatellite instability occurring in familial and sporadic settings: parallel pathways of tumorigenesis. , 2001, The American journal of pathology.

[6]  C. Adebamowo,et al.  Polymorphic variation at the BAT-25 and BAT-26 loci in individuals of African origin. Implications for microsatellite instability testing. , 1999, The American journal of pathology.

[7]  A. Chapelle.  Testing tumors for microsatellite instability , 1999, European Journal of Human Genetics.

[8]  P. Møller,et al.  MSH2 genomic deletions are a frequent cause of HNPCC , 1998, Nature Genetics.

[9]  S Srivastava,et al.  A National Cancer Institute Workshop on Microsatellite Instability for cancer detection and familial predisposition: development of international criteria for the determination of microsatellite instability in colorectal cancer. , 1998, Cancer research.

[10]  K. Do,et al.  Morphology of sporadic colorectal cancer with DNA replication errors , 1998, Gut.

[11]  J. Rüschoff,et al.  Diagnostic microsatellite instability: definition and correlation with mismatch repair protein expression. , 1997, Cancer research.

[12]  G. Thomas,et al.  BAT-26, an indicator of the replication error phenotype in colorectal cancers and cell lines. , 1997, Cancer research.

[13]  Chiara Pastrello,et al.  Stability of BAT26 in tumours of hereditary nonpolyposis colorectal cancer patients with MSH2 intragenic deletion , 2006, European Journal of Human Genetics.