AGEs‐RAGE System Mediates Atrial Structural Remodeling in the Diabetic Rat
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Shuichi Kaneko | S. Kaneko | T. Aizawa | M. Takamura | T. Yamashita | L. Fu | A. Sekiguchi | Takeshi Yamashita | Takeshi Kato | Takeshi Kato | Akiko Sekiguchi | Takayuki Tsuneda | Kouichi Sagara | Masayuki Takamura | Tadanori Aizawa | Long-Tai Fu | T. Tsuneda | K. Sagara | Takayuki Tsuneda
[1] J. Finsterer,et al. Diabetic atrial fibrillation patients: mortality and risk for stroke or embolism during a 10‐year follow‐up , 2003, Diabetes/metabolism research and reviews.
[2] X. Chen,et al. RAGE and amyloid-β peptide neurotoxicity in Alzheimer's disease , 1996, Nature.
[3] F A Mathewson,et al. The natural history of atrial fibrillation: incidence, risk factors, and prognosis in the Manitoba Follow-Up Study. , 1995, The American journal of medicine.
[4] D. Reda,et al. Amiodarone versus sotalol for atrial fibrillation. , 2005, The New England journal of medicine.
[5] V. Hwa,et al. Connective tissue growth factor/IGF-binding protein-related protein-2 is a mediator in the induction of fibronectin by advanced glycosylation end-products in human dermal fibroblasts. , 2002, Endocrinology.
[6] M. Movahed,et al. Diabetes mellitus is a strong, independent risk for atrial fibrillation and flutter in addition to other cardiovascular disease. , 2005, International journal of cardiology.
[7] K. O. Elliston,et al. Cloning and expression of a cell surface receptor for advanced glycosylation end products of proteins. , 1992, The Journal of biological chemistry.
[8] H. Sokabe,et al. Does so-called streptozocin hypertension exist in rats? , 1987, Hypertension.
[9] J. Yee,et al. Regulation of connective tissue growth factor activity in cultured rat mesangial cells and its expression in experimental diabetic glomerulosclerosis. , 2000, Journal of the American Society of Nephrology : JASN.
[10] M. Katovich,et al. Effects of Streptozotocin-Induced Diabetes and Insulin Treatment on Blood Pressure in the Male Rat , 1995, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[11] L. Thal,et al. Blood Levels of Alpha-1-Antichymotrypsin and Risk Factors for Alzheimer’s Disease: Effects of Gender and Apolipoprotein E Genotype , 2000, Dementia and Geriatric Cognitive Disorders.
[12] B. Gersh,et al. Atrial fibrillation: a perspective: thinking inside and outside the box. , 2004, Circulation.
[13] A. Schmidt,et al. Suppression of accelerated diabetic atherosclerosis by the soluble receptor for advanced glycation endproducts , 1998, Nature Medicine.
[14] D. Severson,et al. Type I and II models of diabetes produce different modifications of K+ currents in rat heart: role of insulin , 1998, The Journal of physiology.
[15] P. Grant,et al. Effects of novel polymorphisms in the RAGE gene on transcriptional regulation and their association with diabetic retinopathy. , 2001, Diabetes.
[16] W. Hurley,et al. Isolation and characterization of two binding proteins for advanced glycosylation end products from bovine lung which are present on the endothelial cell surface. , 1992, The Journal of biological chemistry.
[17] Z. Makita,et al. Advanced Glycation End Product-induced Apoptosis and Overexpression of Vascular Endothelial Growth Factor and Monocyte Chemoattractant Protein-1 in Human-cultured Mesangial Cells* , 2002, The Journal of Biological Chemistry.
[18] T. Kislinger,et al. Blockade of RAGE–amphoterin signalling suppresses tumour growth and metastases , 2000, Nature.
[19] K. Preissner,et al. The Pattern Recognition Receptor (RAGE) Is a Counterreceptor for Leukocyte Integrins , 2003, The Journal of experimental medicine.
[20] E. Stier,et al. Receptors for advanced glycation end-products (AGE)—expression by endothelial cells in non-diabetic uraemic patients , 1996 .
[21] T. Igarashi,et al. Chronic hypertension induced by streptozotocin in rats , 1978, Naunyn-Schmiedeberg's Archives of Pharmacology.
[22] G. O. Carrier,et al. Influence of Short‐Term Experimental Diabetes on Blood Pressure and Heart Rate in Response to Norepinephrine and Angiotensin II in the Conscious Rat , 1983, Journal of cardiovascular pharmacology.
[23] M. Takigawa,et al. Inhibition of endogenous expression of connective tissue growth factor by its antisense oligonucleotide and antisense RNA suppresses proliferation and migration of vascular endothelial cells. , 1998, Journal of biochemistry.
[24] D. Levy,et al. Impact of atrial fibrillation on the risk of death: the Framingham Heart Study. , 1998, Circulation.
[25] R. Wada,et al. Effects of OPB‐9195, anti‐glycation agent, on experimental diabetic neuropathy , 2001, European journal of clinical investigation.
[26] Merlin C. Thomas,et al. A Breaker of Advanced Glycation End Products Attenuates Diabetes‐Induced Myocardial Structural Changes , 2003, Circulation research.
[27] S. Takasawa,et al. Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice. , 2001, The Journal of clinical investigation.
[28] D. Singer,et al. Prevalence of diagnosed atrial fibrillation in adults: national implications for rhythm management and stroke prevention: the AnTicoagulation and Risk Factors in Atrial Fibrillation (ATRIA) Study. , 2001, JAMA.
[29] Z. Makita,et al. Suppression of transforming growth factor beta and vascular endothelial growth factor in diabetic nephropathy in rats by a novel advanced glycation end product inhibitor, OPB-9195 , 1999, Diabetologia.
[30] S. Lévy,et al. Characterization of different subsets of atrial fibrillation in general practice in France: the ALFA study. The College of French Cardiologists. , 1999, Circulation.
[31] L. Råstam,et al. Atrial fibrillation and its association with type 2 diabetes and hypertension in a Swedish community , 2004, Diabetes, obesity & metabolism.
[32] S. Kaneko,et al. What are Arrhythmogenic Substrates in Diabetic Rat Atria? , 2006, Journal of cardiovascular electrophysiology.
[33] J. Chen,et al. The Receptor for Advanced Glycation End Products (RAGE) Is a Cellular Binding Site for Amphoterin , 1995, The Journal of Biological Chemistry.
[34] P A Wolf,et al. Prevalence, incidence, prognosis, and predisposing conditions for atrial fibrillation: population-based estimates. , 1998, The American journal of cardiology.
[35] F. Martin,et al. Suppression Subtractive Hybridization Identifies High Glucose Levels as a Stimulus for Expression of Connective Tissue Growth Factor and Other Genes in Human Mesangial Cells* , 1999, The Journal of Biological Chemistry.
[36] C. Soto,et al. Receptor-dependent cell stress and amyloid accumulation in systemic amyloidosis , 2000, Nature Network Boston.
[37] M. Domanski,et al. Relationships Between Sinus Rhythm, Treatment, and Survival in the Atrial Fibrillation Follow-Up Investigation of Rhythm Management (AFFIRM) Study , 2004, Circulation.
[38] G. Lip,et al. Diabetes mellitus and atrial fibrillation: perspectives on epidemiological and pathophysiological links. , 2005, International journal of cardiology.
[39] J. Yamamoto. Blood pressure and metabolic effects of streptozotocin in Wistar-Kyoto and spontaneously hypertensive rats. , 1988, Clinical and experimental hypertension. Part A, Theory and practice.
[40] M. Cooper,et al. Prevention of Accelerated Atherosclerosis by Angiotensin-Converting Enzyme Inhibition in Diabetic Apolipoprotein E–Deficient Mice , 2002, Circulation.
[41] P. Wolf,et al. Characteristics and prognosis of lone atrial fibrillation. 30-year follow-up in the Framingham Study. , 1985, JAMA.
[42] J. Seward,et al. The changing epidemiology and natural history of nonvalvular atrial fibrillation: clinical implications. , 2004, Transactions of the American Clinical and Climatological Association.
[43] M. Cooper,et al. Renal connective tissue growth factor induction in experimental diabetes is prevented by aminoguanidine. , 2002, Endocrinology.
[44] Gary R. Grotendorst. Connective tissue growth factor: a mediator of TGF-β action on fibroblasts , 1997 .
[45] P. Korantzopoulos,et al. The Association of Metabolic Syndrome with Atrial Fibrillation: An Emerging Epidemiological and Pathophysiological Hypothesis , 2005, Cardiology.
[46] A. Schmidt,et al. RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease. , 1996, Nature.
[47] A. Schmidt,et al. Activation of receptor for advanced glycation end products: a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis. , 1999, Circulation research.
[48] V. D’Agati,et al. RAGE drives the development of glomerulosclerosis and implicates podocyte activation in the pathogenesis of diabetic nephropathy. , 2003, The American journal of pathology.
[49] L. Lau,et al. Fisp12/Mouse Connective Tissue Growth Factor Mediates Endothelial Cell Adhesion and Migration through Integrin αvβ3, Promotes Endothelial Cell Survival, and Induces Angiogenesis In Vivo , 1999, Molecular and Cellular Biology.
[50] J. Magyar,et al. Action potentials and potassium currents in rat ventricular muscle during experimental diabetes. , 1992, Journal of molecular and cellular cardiology.
[51] V. D’Agati,et al. Receptor for Advanced Glycation End Products Mediates Inflammation and Enhanced Expression of Tissue Factor in Vasculature of Diabetic Apolipoprotein E–Null Mice , 2001, Arteriosclerosis, thrombosis, and vascular biology.
[52] Gary R. Grotendorst,et al. Stimulation of fibroblast cell growth, matrix production, and granulation tissue formation by connective tissue growth factor. , 1996, The Journal of investigative dermatology.
[53] E. Stier,et al. Receptors for advance glycation end-products (AGE) - expression by endothelial cells in non-diabetic uraemic patients. , 1996, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[54] P. Schattner,et al. An Update on the Etiology and Epidemiology of Diabetes Mellitus , 2006, Annals of the New York Academy of Sciences.
[55] T. Tomita,et al. Streptozotocin Diabetic Rats Are Hypertensive Despite Reduced Hypothalamic Responsiveness , 1982, Hypertension.
[56] Z. Makita,et al. Progression of Nephropathy in Spontaneous Diabetic Rats Is Prevented by OPB-9195, a Novel Inhibitor of Advanced Glycation , 1997, Diabetes.
[57] M. Neurath,et al. RAGE Mediates a Novel Proinflammatory Axis A Central Cell Surface Receptor for S100/Calgranulin Polypeptides , 1999, Cell.